鼠毒

Bone marrow transplantation + habu snake venom + GM-CSF group: recipients received GM-CSF intraperitoneal injection for seven days continuously after habu snake venom injection, and observed the green fluorescence in kidney on above-mentioned times.

骨髓移植+竹叶青蛇毒+GM-CSF组：骨髓移植后的受体鼠在竹叶青蛇毒注射后连续腹腔注射GM-CSF一周，在上述各时间点观察肾脏组织中的绿色荧光。

Bone marrow transplantation + habu snake venom + Ligustrazine + GM-CSF group: recipients received Ligustrazine and GM-CSF intraperitoneal injection for seven days continuously after habu snake venom injection, and observed the green fluorescence in kidney on above-mentioned times.

骨髓移植+竹叶青蛇毒+GM-CSF+川芎嗪组：骨髓移植后的受体鼠在竹叶青蛇毒注射后连续腹腔注射GM-CSF及川芎嗪一周，在上述各时间点观察肾脏组织中的绿色荧光。

B one marrow transplantation + habu snake venom + Ligustrazine group: recipients received Ligustrazine intraperitoneal injection for seven days continuously after habu snake venom injection, and observed the green fluorescence in kidney on above-mentioned times.

骨髓移植+竹叶青蛇毒+川芎嗪组：骨髓移植后的受体鼠在竹叶青蛇毒注射后连续腹腔注射川芎嗪一周，在上述各时间点观察肾脏组织中的绿色荧光。

Objective To study the killing efficiency of one million MLD/ml Type-C Botulinum lyophilized preparation to Ochotona curzoniae .

目的 试验100万毒价/mlC型肉毒梭菌毒素冻干剂对高原鼠兔的杀灭效果。

Inbred strains of mice and outbred rats received an intraplantar injection of honeybee venom and the between-group and within-group correlations were calculated for spontaneous nociceptive measures, thermal and mechanical hypersensitivity, and edema and temperature.

将一个剂量的蜜蜂毒(0.05mg/0.025ml)注入12个近交系（129P3/J、A/J、AKR/J、BALB/cJ、C3H/HeJ、C57BL/6J、C57BL/l0J、C58/J、CBA/J、DBA/2J、RⅢs/J和SM/J）小鼠或6个剂量的蜜蜂毒(0.001、0.005、0.01、0.05、0.1、0.2mg/0.05ml)注入远交系(Sprague-Dawley)大鼠的一侧足底皮下，分别检测自发伤害性反应、热和机械性痛敏，以及炎症的水肿和局部皮温，然后对组间和组内数据进行相关性分析。

It is concluded that latencies of P wave and N wave of CSEP prolong obviously with the development of sepsis.

脓毒血症过程中随病情的发展大鼠CSEP正、负波潜伏期有逐渐延长的趋势，为脓毒血症的基础与临床研究提供了依据。

C of the mixtures to SD rat were 285,165 and 180, respectively.

对大鼠的共毒系数分别为285、165和180，同样存在增毒效应或协同作用。

To investigate the effect of ketamine on hepatic mitochondria in septic mice. Cecal ligation and puncture were used to make a sepsis model.

目的 探讨氯胺酮对脓毒症大鼠肝线粒体能量代谢的影响以及氯胺酮对脓毒症肝损伤的保护机制。

The latedischarges decreased from 9.29 ± 0.97 to 6.71 ± 0.68 with the A-fiberconditioning stimulus increasing from 1 to 5 (n〓8, P〓0. 05).(7) The intervalbetween the conditioning stimulus and test stimulus (C-T interval) wasincreasing, the inhibition tended to plateau off. At shorter time intervalsthe inhibition became more effective. When C-T interval was limited in 50ms,the inhibitory effects was the strongest, here, the late discharges reducedfrom 12.57±1.21to 2.29±0.42 (n=11, P＜0. 01).(8) Behavior research showedthat the rat model of snake venom exhibited neuropathic pain with heathyperalgesia, cold and mechanical allodynia, which corresponding to the acuteelectrophysiological findings.

此时轻刷WDR神经元的感受野不能引起其活动改变，但伤害性齿镊夹捏仍可引起WDR神经元放电增多；〓5〓晚成分放电的潜伏期缩短，即宁静期的时程变短，由给蛇毒前的118.83〓3.67ms降至50.72〓1.36ms〓n〓32，P〓0.01〓；〓6〓在正常动物，如果预先给予只激活A纤维的弱条件电刺激〓mA，100μs〓可抑制随后的伤害性检验刺激所诱发的WDR神经元的晚成分放电，当条件刺激个数从1增加至5时，每次伤害性检验刺激所诱发的晚成分放电数从9.29〓0.97个降至6.71〓0.68个〓n〓8，P〓0.05〓；〓7〓固定条件刺激数为1个，当条件刺激与检验刺激之间的间隔增大时，A纤维条件刺激对WDR神经元晚成分放电的抑制作用逐渐减弱，当条件刺激与检验刺激之间的间隔在50 ms以内时，抑制效应最为显著，此时，晚成分放电数由正常时的12.57〓1.21个降至2.29〓0.42个〓n〓11，P〓0.01〓；〓8〓与急性研究中的WDR神经元电活动的变化结果相匹配，利用蛇毒制备的大鼠模型在行为学上表现为热痛觉过敏、冷觉的痛性感觉异常及机械痛觉过敏等慢性痛症状。

At the same time, most WDR neurons failedto respond to the light brush applied to the receptive fields, but they couldbe intensively excited by the noxious pinch.(5) The latency of the latedischarges was shortened from 118.83 ± 3.67ms to 50.72 ± 1.36ms (n〓32, P〓0. 01).(6) Preceding graded number of A〓fiber conditioning inputs (〓mA, 100 μs) delayed the C-activity evoked by the following nociceptive teststimulus activating both A- and C-fiber applied to the sciatic nerve. The latedischarges decreased from 9.29 ± 0.97 to 6.71 ± 0.68 with the A-fiberconditioning stimulus increasing from 1 to 5 (n〓8, P〓0. 05).(7) The intervalbetween the conditioning stimulus and test stimulus (C-T interval) wasincreasing, the inhibition tended to plateau off. At shorter time intervalsthe inhibition became more effective. When C-T interval was limited in 50ms,the inhibitory effects was the strongest, here, the late discharges reducedfrom 12.57±1.21to 2.29±0.42 (n=11, P＜0. 01).(8) Behavior research showedthat the rat model of snake venom exhibited neuropathic pain with heathyperalgesia, cold and mechanical allodynia, which corresponding to the acuteelectrophysiological findings.

此时轻刷WDR神经元的感受野不能引起其活动改变，但伤害性齿镊夹捏仍可引起WDR神经元放电增多；〓5〓晚成分放电的潜伏期缩短，即宁静期的时程变短，由给蛇毒前的118.83〓3.67ms降至50.72〓1.36ms〓n〓32，P〓0.01〓；〓6〓在正常动物，如果预先给予只激活A纤维的弱条件电刺激〓mA，100μs〓可抑制随后的伤害性检验刺激所诱发的WDR神经元的晚成分放电，当条件刺激个数从1增加至5时，每次伤害性检验刺激所诱发的晚成分放电数从9.29〓0.97个降至6.71〓0.68个〓n〓8，P〓0.05〓；〓7〓固定条件刺激数为1个，当条件刺激与检验刺激之间的间隔增大时，A纤维条件刺激对WDR神经元晚成分放电的抑制作用逐渐减弱，当条件刺激与检验刺激之间的间隔在50 ms以内时，抑制效应最为显著，此时，晚成分放电数由正常时的12.57〓1.21个降至2.29〓0.42个〓n〓11，P〓0.01〓；〓8〓与急性研究中的WDR神经元电活动的变化结果相匹配，利用蛇毒制备的大鼠模型在行为学上表现为热痛觉过敏、冷觉的痛性感觉异常及机械痛觉过敏等慢性痛症状。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。