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Therefore, menu items with disodium inosinate and/or disodium guanylate also contain MSG.

因此,菜单项与钠盐的inosinate和/或钠盐的鸟苷也含有味精。

They also contain two ingredients that advertise the presence of MSG: disodium inosinate, and disodium guanylate.

他们还包含两个成分宣传在场的讯息:钠盐inosinate ,钠盐的鸟苷

Methods: The human KDR promoter (-225 bp~+127 bp) was cloned by PCR. Subsequently, a recombinant adenoviral plasmid carrying KDR promoter for the HSV-tk gene was constructed with the AdEasy system. As a control, the plasmid pAdCMV-tk carrying CMV promoter for the HSV-tk gene was also constructed. 293 packaging cells were transfected by both newly-constructed plasmids and the infectious viruses AdKDR-tk and AdCMV-tk were generated. Then the KDR-producing cells and the KDR nonproducing cells (HepG2) were infected with AdKDR-tkand AdCMV-tk, followed by ganciclovir administration.

应用PCR克隆出人KDR基因启动子序列-225bp~+127bp,以AdEasy system为载体,构建携带受KDR启动子或CMV启动子调控tk基因表达的2种重组腺病毒质粒pAdKDR-tk和pAdCMV-tk,在293细胞中包装、扩增后,体外感染表达KDR的脐静脉血管内皮细胞系HUVEC和不表达KDR的肝癌细胞系HepG2,并给予不同浓度的丙氧鸟苷处理,5d后收集存活细胞并计数。

If GVHD occurred, GCV would be administered in order to kill TK〓T lymphocytes selectedly and to control GVHD, while preserving therapeutic effects of T lymphocytes.

将TK基因体外转染供者T细胞成为TK〓T细胞,然后和CD34〓细胞混合移植,一旦发生GVHD,则给予特异性药物丙氧鸟苷治疗,选择性杀死引起GVHD的TK〓T细胞,使GVHD得到控制,同时又可保留T细胞的治疗作用。

Once again, the chili has hidden MSG: autolyzed yeast extract, spices, artificial flavors, natural flavorings, disodium inosinate and disodium guanylate (MSG give-aways).

再次,辣椒已隐藏的讯息: autolyzed酵母提取物,香料,人工香料,天然调味料,钠盐的inosinate和钠盐的鸟苷

This disease is important to recognize because there is an effective drug treatment, acyclovir.

认识这种是很重要的,因为针对它有一种有效的治疗药物,无环鸟苷

He was treated empirically with acyclovir, and multiple blood and CSF cultures were negative.

他在接受经验性的无环鸟苷治疗后,多次血培养和脑脊液培养是阴性。

Objective To investigate in Chinese population the relationship between GTP cyclohydrolase 1 gene mutation and phenotypes of dopa-responsive dystonia.

目的 分析国人多巴敏感性肌张力障碍患者三磷酸鸟苷环羟化酶1(GCH-1)基因突变与表现型的关系。

Mechanical strains also regulated the protein and mRNA expression of several differentiation markers, as well as the activation of extracellular signal-regulated kinases, p38 MAP kinase and protein kinase B in a frequency-dependent manner. Furthermore, the inhibition of p38 pathway could block the strain-frequency induced the phenotype modulation of VSMCs, neither ERKs nor Akt. Frequency of mechanical strain, not conditioned medium, regulated the phenotype of VSMCs in a frequency-dependent manner. Rho-GDI alpha was suppressed by the mechanical strain at 1Hz.

采用免疫细胞化学法检测VSMCs形态和排列的变化;RT-PCR和Western blotting检测表型标志分子α-肌动蛋白、肌球蛋白重链(SM1/2)、肌动蛋白相关蛋白SM22α和调宁蛋白(h1-calponin)的mRNA和蛋白水平的变化;抑制剂或RNA干扰阻断可能的信号调节分子的活性或表达,包括p38、细胞外信号调节激酶1/(2extracellular signal-regulated kinases, ERK1/2)、蛋白激酶B和Rho-鸟苷酸解离抑制因子(Rho-guanine nucleotide dissociation inhibitor, Rho-GDI alpha),研究了不同频率张应变对VSMCs表型转化的影响及其调节机制。

The growth of tumor was tested and pathological analysis was performed after ganciclovir and retroviruses treatment induced by doxycycline.

观察体外强力霉素诱导,丙氧鸟苷和逆转录病毒作用后移植瘤生长的变化及组织病理改变,分析其对乳腺癌裸鼠移植瘤的调控性治疗作用。

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