髓鞘

Methods From January 2002 to May 2006,39 children with demyelinating disases were scanned through cerebra by MRI,and MRI shows were analyzed.

2002年1月～2006年5月应用MRI对39例有脱髓鞘病变的患儿进行颅脑扫描，分析其MRI表现。

In the present report we describe a young man with CTX who was on high dose steroids for a misdiagnosed chronic inflammatory demyelinating polyneuropathy and had normal level of serum cholestanol.

本文报道1例青年男性CTX患者，该患者因被误诊为慢性炎症性脱髓鞘性多神经病而接受大剂量的激素治疗，并且患者的血清胆固烷醇水平正常。

There were chondroitic acid proteoglycan in the center of the injured nerve,thegelatinase maybe degradated them and promoted the regenerative process.

神经的修复过程宏观上看是髓鞘的退变与再生过程，微观上是众多的细胞因子综合调控的结果。

There were infiltration of inflammatory cells inside nerve tissue and perivascular cuffings in HE staining. In trichrome staining, the axon with inhomogeneous chromatosis was swollen and beaded-discontinued; The myelin was porous and partly disaggregated.

HE染色发现神经组织内炎细胞浸润，血管&袖套&样病灶形成；三色染色可见轴突肿胀，呈串珠状，且不连续，着色不均匀；髓鞘结构层次紊乱，疏松，崩解。

Methods Cerebral tissue specimens were obtained from 15 patients with HSLE and the histological observations under optical and electron microscopes were carried out by HE, Bielschowsky's, and chromotrope 2R-brilliant green staining.

收集15例HSLE患者的脑组织标本，通过电镜、HE染色、髓鞘染色及神经纤维染色来观察其组织学特点，并进行病理分析。

Objective to evaluate the functional recovery of acute spinal cord injuried rats treated with exogenous wnt-3a signal protein administration and to explore its mechanism.methods moderate spinal cord contusion injury was made in 40 adult sprague dawley rats at t10.twenty rats served as contusion controls(group 1).twenty rats were treated with wnt-3a for three days after injury (group 2).the functional recovery of the rats was observed through basso,beattie,bresnahan open field locomotor score.rats were killed at 14 or 28 days after injury,then spinal cords were removed for histopathological examinations,and the expression of the bromodeoxyuridine plus neural cell markers was stained with immunohistochemical method.results rats of two groups receiving a contusive injury recovered substantial function within 1 week.by 28 days,rats in groups 2 scored 7.0 points better on the bbb scores than rats in group 1 group 2=16.94,after 28 days vs.

目的 研究外源性wnt-3a信号蛋白对脊髓损伤的修复作用，并探讨其作用机制。方法取40只成年雌性sd大鼠在t10节段制备适度脊髓挫伤模型。随机从中取20只为损伤对照组(group 1)，另外20只为损伤治疗组(group 2)。脊髓损伤3天后用wnt-3a蛋白治疗。这些大鼠的功能恢复通过basso、beattie、bresnahan开放视野运动评分来观察。这些大鼠分批在损伤后14天或28天被处死，取出损伤节段脊髓用来组织病理学检查，同时用5-溴脱氧尿嘧啶核苷和神经细胞标记物进行免疫组化染色。结果两组大鼠在伤后一周运动功能有明显的恢复。不过，到损伤后28天，我们观察发现，损伤治疗组中的大鼠bbb评分比损伤对照组中的评分高出7.0分左右(group 2∶16.94±1.18，group 1∶9.89±1.29；p.05)，光镜和电镜检查发现wnt-3a蛋白对髓鞘形成和轴突再生有一定的修复效果。

A synapse is a junction between the axon and dendrites of two neurons.

许多神经元内的轴突被包裹在髓鞘内。

Methods Clinical laboratory and electrophysiologic date on 62 patients with CBS were reviewed. Patients were classified as having acute inflammatory demyelinating polyneuropathy or acute motor axonal neuropathy based on electrodiagnosis criteria.

根据电生理分型将62例GBS患者分为急性炎性脱髓鞘型多神经病(acute inflammatory demyelinating polyneuropathy， AIDP)与急性运动轴索型神经病acute motor axon neuropathy， AMAN）两组，对两组患者的临床特征进行比较分析。

The structure of axon was completed, and the arrangement of regenerated nerve fibers with rotund or elliptoid shapes was closed.

电镜超微结构观察可见再生的神经纤维较粗大，排列紧密，呈均匀一致圆型或椭圆型，髓鞘较厚，轴突结构完整。

CD4+T cells directly injected into the endoneurium caused conduction block followed by macrophage infiltration and demyelination.

CD4+T细胞直接注射到神经细胞内，引起巨噬细胞浸润和脱髓鞘，随后引起神经传导阻滞。

On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面，更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值，分为几个相等的区间作为横轴，并将各区间内所测定值依所出现的次数累积而成的面积，用柱子排起来的图形，也叫做柱状图。