髓细胞的

The results indicated that 100μg/ml LPS had significant damage on the ultrastructure of HDPCs . After 5μg/ml Chinese nutgall water extract were added , the damage of HDPCs'ultrastructure was decreased significantly.

结果显示100μg/ml内毒素对牙髓细胞超微结构有不同程度的损伤，加入5μg/ml五倍子水提取物后，牙髓细胞超微结构损伤明显减轻，细胞器结构基本恢复正常。

HDPCs are dominating cells in dental pulpal tissue. HDPCs can not only synthesize dental pulp organic ingredient, but also differentiate into odontoblast and protect dental pulp.

HDPCs是牙髓组织中的主体细胞，HDPCs不仅具有合成牙髓有机质的能力，而且必要时还能分化为成牙本质细胞保护牙髓。

The females incubated at 33 , their ovarium has powerful cortex, the germinal epithlium produce oocyte, its medulla degenerate into ovarian cavity. During sexual differentiation, mitochondria and smooth endoplasmic reticulum in cells of ovary increase in quantity, they disintegrate after sexual differentiation. There are a lot of big grana in cells of ovary before sexual differentiation, in the course of sexual differentiation the quantity of the big grana decrease, while small grana increase and peakafter sexual differentiation. The males incubation at 26 , cortices of their testes degenerate into a thin albuginea. Medulla are formed by testicular cords in which scatter spermatogonium.

33℃下孵化的小龟卵巢皮质发达，由原始生殖细胞产生卵原细胞，髓质退化成为卵巢腔，分化时卵巢细胞的超微结构显示线粒体和内质网的数量大大增加，它们在分化结束后解体，分化前细胞内有较多的大颗粒，在分化中大颗粒减少，小颗粒增多，在分化后小颗粒达到最多。26℃下孵化的小龟精巢皮质退化成为一层薄的白膜，髓质有许多睾丸管构成，管腔内有散落排列的精原细胞。

The result showed that the exogenetic TGF-βhad significantly inhibiting effect on both LPS's restraining the proliferation of pulp cell and inducing the apoptosis of pulp cell.

为探讨TGF-β对LPS介导的人牙髓细胞损伤修复的调控机制，首先采用了免疫组化染色技术检测了人牙髓细胞内源性TGF—β的表达变化。

Specific siRNA was used to target LMO2 in Molt-4 cells. Results Higher expression of LMO2 was detected in some of patients with acute myeloblastic leukemia or acute lymphoblastic leukemia. However, enforced expression of LMO2 in K562 cells decreased cellular clonogenecity, suggesting the activation of the gene may not be important in the pathogenesis of myeloid leukemia.

结果 LMO2基因在很多急性髓系和淋巴系白血病患者的表达水平高于慢性白血病患者及正常骨髓细胞，但LMO2转染的K562细胞克隆形成率下降，提示该基因在髓系白血病细胞的表达异常是一种伴随结果而不具有致癌作用。

Dental pulp cell is the main cellular component of the pulpal tissue. It plays important roles in nutrition, metabolism, sense and repair of the teeth.

牙髓细胞是牙髓组织中的主要细胞成分，担负着牙齿的营养代谢、感觉和修复等重要功能。

Apoptosis are found in rat normal and inflammatory dental pulp, and it may have a important role in pulpitis.

正常牙髓和炎症牙髓中存在细胞凋亡，细胞凋亡在牙髓炎的转归和预后方面可能有一定的作用。

Apoptosis are found in rat normal and inflammatory dental pulp, and it may have a important role in pulpitis.Key words apoptosis;pulpitis;TUNEL method;rat

正常牙髓和炎症牙髓中存在细胞凋亡，细胞凋亡在牙髓炎的转归和预后方面可能有一定的作用。

A case is made that areas of chronic demyelination arise because of concurrent loss of oligodendrocyte progenitor cells and oligodendrocytes and that because of the slow rate of repopulation that occurs in old individuals the recruited OPCs are not exposed to the acute inflammatory environment required to generate remyelinating oligodendrocytes.

一种说法是慢性脱髓鞘的区域产生系因少突胶质原始细胞和少突胶质细胞并存，而且因老年个体中缓慢的再生率，补充的OPCs没有暴露于产生髓鞘再生少突胶质细胞需要的急性炎症的环境。

A study has indicated the expression of Smad2 in human dental pulp cells, but there are no reports about the functions of Smad2/3 in TGF- P modulating dental pulp repair and tooth development.

本研究的目的是观察人牙髓细胞内Smad2和Smad3分子在TGF-β1信号转导中的作用，以及TGF-β1对二者表达水平的调控方式，并检测Smad2和Smad3在牙胚发育的不同时期和正常、龋坏、炎症牙髓组织中的表达及其变化，旨在从细胞内信号转导水平探讨TGF-β调节成牙本质细胞分化和牙本质形成的分子机理，为揭示牙胚发育和牙髓损伤修复过程中基因表达的调控机制提供一定的实验依据。

By the time of its fall, most of the prisoners were writers who had written against the corruptions of the government.

到它被攻陷的时候，里面多数的犯人是写了反对政府贪污文章的作家。

The most obvious variation to ovum morphological character was that the color was changed from light green to sepiaceous in embryonic development, and all the ovums were almost hatched after 96h.

在胚胎发育过程中卵的形态特征最明显的变化是颜色从淡绿到深褐色，卵在发育96h后卵基本全部孵化。