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髓形成

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RESULTS On the experimental side, there was new bone formation in the bone medullary cavity, the endosteum and the section surface of the cortex at 2 weeks. At 4 weeks, at the center of the blood clot in the tube there was new bone formation and bone defect was completely healed at 8 weeks.

结果 实验侧术后2 周即在骨断端髓腔、骨内膜及皮质断面处有新骨形成,并长入管内血肿,术后4周新骨长入血肿中心,8周完全骨愈合。

These results support the idea that in the human fetal forebrain axonal PSA-NCAM expression is inversely related to primary myelination.

这些结果支持人的胎儿前脑轴突中PSA-NCAM的表达反而与最初的髓鞘形成有关的观点。

SCI can cause hematomyel ia, dropsy, necrosis, capsular space, gummosis and so on, which can lead to neuron deprivation, demyelination of neurite. Clinical symptoms mainly manifest funct ion disorder of movement, sense, reflectance, and musculus sphincter below injury plan.

SCI后出现脊髓出血、水肿、坏死、囊腔形成、退变胶质化等一系列病理改变,以致神经元丧失、轴突脱髓鞘,临床表现为受损平面以下的运动、感觉、反射及括约肌功能的障碍。

After twelve weeks new bone had converted to lamellar bone, some of which formed bone-shell. Marrow cavity was seen.

第12周,新骨组织形成板层状结构,髓腔明显,内含造血和脂肪细胞,第24周,新骨结构典型。

While in the BMP/HA group, although the fibrous callus was appeared in the 4th and 8th weeks, the HA was not absorbed. In the 12th and 16th weeks the change was similar to that in the 8th week and no recanalization of bone marrow cavity.

而BMP/HA复合组,虽然在第4、8周也有纤维性骨痂形成,但HA不被吸收;第12、16周与第8周相似,无髓腔再通现象。

The forming time was incompletely consistency with the developing time of neurons in the medulla oblongata.

结果表明:(1)第6周山羊胚胎延髓处在组织发生末期和核团形成初期,是其内部核团构建的关键时期。

The function of the adrenal medulla is to form nonadrenaline and adrenaline.

肾上腺髓质的功能是形成去甲肾上腺素和肾上腺素

Ischemia injury of oligodendrocyte could make obstacle on methylation of myelin sheath alkali protein after cerebral ischemia.

维生素B12参与了髓鞘形成过程,在中枢神经系统白质损伤的恢复以及脑缺血后痴呆和锥体系病变的恢复方面显示了良好疗效。

Essential fatty acids such as GLA are utilised by the body to make up our basic cellular structure and to form the myelin sheath that insulates nerve cells and ensures the transmission of information to vital organs.

必需脂肪酸,如亚麻酸是人体所利用,以弥补获得的治疗效果我们的基本细胞结构,并形成髓鞘的隔离神经细胞,并确保信息传输的重要器官。

Maybe it has to do with suboptimal myelin formation, and that's a very different mechanism.

或许这与髓鞘形成不佳有关,但那是非常不同的机转。

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