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Preliminary in vitro results showed that some of these compounds can promote osteoblast proliferation.

体外抗骨质疏松筛选结果表明,该类化合物能促进成骨细胞的增殖。

Along with the continuing of fixation period, osteoid thickness and osteoid area gradually decline.

随着固定时间延长类骨质厚度及面积有逐渐下降的趋势。

In different stage after operation, there were more new formed blood vessels, fibroblast and fibrotissue and osteoid than control groups.

术后不同时期,实验组新生血管的量、新生纤维组织及纤维母细胞、类骨质等均较对照组多。

Not having enough calcium is associated with osteoporosis, osteomalacia, and rickets.

没有足够的钙是与骨质疏松症,骨软化症,佝偻病。

Chronic deficiency may be a factor in the development of osteoporosis or osteomalacia and may contribute to hypertension and colon cancer.

钙缺乏可致骨质疏松或骨质变软,还可能和高血压、结肠癌的发生有关。

It may not only contribute to the deelopment of osteoporosis and osteomalacia/rickets but also play a role in cancers, multiple sclerosis, infection, hypertension, and diabetes mellitus.

它可能并不仅仅导致骨质疏松和骨软化症/佝偻病,同时也与癌症、多发性硬化症、感染、高血压和糖尿病的发病有关。

Ultraviolet light is needed to activate production of vitamin D in the body, which prevents rickets, osteomalacia and osteoporosis.

在人体内部,紫外线被用来刺激维生素D的生成,这种维生素能预防驼背、骨软化和骨质疏松症等骨骼问题。

Vitamin D deficiency is linked to development of bone-softening diseases, such as rickets among children, and osteomalacia and osteoporosis among adults, Garden-Robinson said.

维生素D缺乏联系的发展,骨软化疾病,如佝偻病儿童中,而软骨病和骨质疏松症在成年人中,花园-鲁宾逊说。

Absorption is reduced, possibly contributing to iron deficiency, osteoporosis (also related to calcium deficiency), and osteomalacia due to lack of vitamin D and absence of exposure to sunshine.

吸收功能减退,可能引起缺铁、骨质疏松症及缺乏维生素D缺乏和阳光照射所致的骨软化症。

Prevention of osteoporosis, muscle cramps, osteomalacia, lumbar back pain in the elderly acid belly.

预防骨质疏松,肌肉抽蓄,骨软化,腰酸腹背痛的中老年人。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。