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Acetylsalicylic acid, a nonspecific cyclooxygenase inhibitor, was also found to suppress viral reactivation in the heat-stressed mouse model.

然而,塞来昔布比非反非特异的非特异的单氨氧化酶2的抑制剂阿司匹林能更为有效抑制复发而且付作用更小。

To study the protective effects of polypeptide from Chlamys farreri on thymocytes damaged by H_2O_2 and antioxidative mechanisms of PCF,we made a thymocytes damaged-H_2O_2 model.

在有氧代谢过程中,机体会产生少量的活性氧,这些ROS可以被机体内存在的抗氧化酶和非酶抗氧化剂所清除。

These results show the level of diacetyl produced in Enterobacter aerogenes UV-3 would be dependent on the nonenzymatic oxidative decarboxylation of ALA.

通过主要代谢酶的酶学性质分析,确定产气肠杆菌突变株UV-3积累丁二酮的代谢途径是由α-乙酰乳酸的非酶氧化直接生成。

It catalyzes the oxidation of dopa rapidly to dopaquinone, which in turn polymerize non-enzymetically into insoluble melanin.

它是一种含铜的酶,能够催化单酚羟化成二酚,把二酚氧化成醌;醌在非酶促条件下形成最终的反应产物黑色素。

On the basis of this system andpreviously morphological, physiological and biochemical studies on cucumber in vitrogynogenesis. Materials from both typical gynogenesis using M99 as induction medium andnon gynogenesis using W5 as induction medium were collected. The activities anddistribution of peroxidase and its isoenzymes, content of soluble protein and distribution of allprotein during early stage of cucumber in vitro gynogenesis and non gynogenesis wereinvestigated by biochemistry and histochemistry technology.

本试验在前期有关离体黄瓜雌核发育形态学和生理生化研究的基础上,以诱导离体雌核发育的 M99 培养基上和非雌核发育的 W5 培养基上培养的黄瓜未受精子房为试材,采用生物化学技术和组织化学技术,深入研究了离体雌核发育早期外植体内的标志酶——过氧化物酶及其同工酶的活性及分布的变化、可溶性蛋白质的含量和总蛋白质分布的变化及核酸和多糖分布的变化,为揭示离体雌核发育早期的细胞分化机制提供科学依据。

The antioxidative activities of these model compounds depend on their scavenging effects on reactive oxygen species in enzymic or nonenzymic manner.

硒酶模型化合物的这些抗氧化活性与它们通过酶或非酶方式清除各种活性氧的作用有关。

2 A kind of enzyme and mediator modified electrode is devised by immobilyzing both N-methyl phenazine sulfate and glucose oxidase on the surface of carbon glassy electrode (GOD-NMP-GC electrode), which successfully realizes the electron transfer process between glucose oxidase and carbon glassy electrode.

将NMP-GC电极与葡萄糖氧化酶结合,制作了GOD-NMP-GC电极,该电极可快速实现电极=NMP介体=GOD活性中心=葡萄糖之间的电子长程转移,并且用循环伏安法对酶催化反应进行了研究;用含丰富乳酸脱氢酶的乳酸杆菌与NMP-GC电极相结合,制作了介体微生物电极,并检测到GC电极=NMP=LDH活性中心=乳酸之间的非均相电子转移。

In summary, the nonphagocytic NADPH oxidase has emerged as a potentially important target to focus on to explore the mechanisms underlying the pathogenesis of diabetic nephropathy.

现就其与糖尿病肾病的关系进行综述。1 NADPH氧化酶的分子结构非吞噬细胞中的NADPH氧化酶在结构上与吞噬细胞中的NADPH氧化酶相似,均由膜单位和胞浆内单位组成。

There were five groups in the examination of cellular levelK562 group,K562/A02 group,K562+ADM group,K562/A02+ADM group and K562/A02+TTD+ADM group).The non-cytotoxicity doses to cell lines K562 and K562/A02 of TTD were got by MTT assay.Using flow cytometry (FCM assay to examine the intracellular ADM concentration.There were three groups in the examination of genic,zymologic and protein levelsK562 group,K562/A02 group and K562/A02+TTD group).The mRNA expression of MDR was measured by fluorescent quantitative reverse transcriptase polymerase chain reaction(RT-PCR.The expression levels of glutathione-S-transferase and topoisomerase Ⅱ were determined by immunohistochemical technique.

细胞水平检测实验分5组(K562组、K562/A02组、K562+ADM组、K562/A02+ADM组和K562/A02+TTD+ADM组),采用MTT法检测TTD对K562和K562/A02细胞的非细胞毒性剂量,流式细胞术检测细胞内阿霉素的浓度,基因、酶学、蛋白水平检测实验分3组(K562组、K562/A02组和K562/A02+TTD组),采用RT-PCR法检测mdr1 mRNA的表达,免疫细胞化学方法检测谷胱甘肽S转移酶π和拓扑异构酶Ⅱ的表达水平,Western-blotting法检测P-糖蛋白和bcl-2表达。

Free radicals generated and are scavenged continuously to keepdynamic equilibrium in the body.

机体内是通过酶触反应和非酶触反应不断产生自由基的。

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