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Nonbicarbonate and intracellular buffers contribute to the buffering of newly formed acid so that the actual drop in [HCO3-] is usually less than the increase in the anion gap due to a simple elevated-gap metabolic acidosis.

非碳酸氢盐和细胞内缓冲液有利于缓冲新形成的酸,以致实际[HCO3-]的下降低于单纯性AG增高型代谢性酸中毒引起的阴离子间隙的增加。

Nonbicarbonate and intracellular buffers contribute to the buffering of newly form ed acid so that the actual drop in [HCO3-] is usually less than the increase in the anion gap due to a simple elevated-gap metabolic acidosis.

非碳酸氢盐和细胞内缓冲液有利于缓冲新形成的酸,以致实际[HCO3-]的下降低于单纯性AG增高型代谢性酸中毒引起的阴离子间隙的增加。

The purpose of this study is to discuss its action mechanism from the point of view of modern medicine, starting with clinical and experimental study,utilizing modern experimental technology.

消瘤方是导师焦中华教授治疗非小细胞肺癌常用的方剂,临床观察安全有效,本研究的目的是从临床和实验研究两方面入手,利用现代实验技术,从现代医学的角度探讨它的作用机制。

Questions plus testingHBV, HCV, HIV, HTLV, syphilisTesting onlyWNV, T. cruzi,Questions onlyCJD, vCJD, HAV, malaria, babesia, leishmaniaQuestions assumed to have impactHHV-8, tropical infections, emergent situations e.g., SARS

需要质疑和检测的内容乙型肝炎病毒,丙型肝炎病毒,艾滋病毒,人类嗜T细胞病毒,梅毒螺旋体只需要检测的内容西尼罗河病毒,克氏锥虫,只需要质疑的内容克雅氏病,变种克雅氏病,甲肝,疟疾,巴贝西虫,利什曼原虫需要质疑假设可产生影响的内容疱疹病毒-8,热带传染病,紧急情况如非典

Histone deacetylase 6(HDAC6) is microtubule and HSP90 associated protein, which control genetic transcription and change cellule motor ability by deacetylased microtubule and HSP90,and then participate tumorigenesis, growth, metabasis, and the production of tumor vessel.

研究方法: 1。应用激光捕获显微切割术联合逆转录-聚合酶链反应技术,检测36例行根治性切除的原发性肝细胞癌中心处非坏死癌组织及24例正常肝组织的HDAC6 mRNA在组织中的表达,并对HDAC6与肝癌发生发展过程中相关的临床病理因素间相关性进行研究。

9The sieve that scanning of 9mTc-MDP nuclide bone can check in installment as lung cancer checks the method; progress as lung cancer course of diseases, tumor labels the level of content CEA, CA125, NSE and CYFRA21-1 rises gradually. Other people of number of tumour of combination of 99mTc-MDP bone scanning has fundamental value in be not cellule lung cancer to be diagnosed in installment.

99mTc-MDP核素骨扫描可作为肺癌分期检查的筛查手段;随着肺癌病程的进展,肿瘤标记物CEA、CA125、NSE和CYFRA21-1的水平逐渐上升。99mTc-MDP骨扫描联合肿瘤标记物在非小细胞肺癌分期诊断中具有重要的价值。

The patient presented with hemoptysis. The chest images and bronchoscopic washing cytology showed bilateral lung involvement, so chemotherapy was prescribed, as stage IV non-small cell lung cancer with lungto-lung metastasis was suspected. After 3 cycles of chemotherapy with docetaxol and carboplatin, the bilateral lung tumors responded discordantly. An aggressive surgical intervention was performed, and the pathology showed synchronous squamous cell carcinoma and a carcinoid tumor.

病人一开始以咳血表现,因为胸部影像学检查及支气管镜抽洗液细胞学检查显示两侧肺部皆有癌侵犯,所以在第四期非小细胞肺癌合并有肺部转移的情形下开始接受化学治疗,经过三个疗程的克癌易及卡铂之化学治疗,两侧肺肿瘤的反应不一致,因此采取积极的手术处理,而病理报告显示两侧肺部肿瘤分别为鳞状上皮癌及类癌。

During the mitosis, the correct separation of sister chromatids is necessary in normal transition from anaphase to telophase and it guarantees exact distribution of DNA. The non-disjuction is the important reason for forming the aneuploid.

在有丝分裂时,姐妹染色单体的正确分离既起到保证遗传物质准确分配的作用,又是分裂细胞从后期向末期过渡所必需的;而姐妹染色单体不分离则是产生非整倍体细胞的重要原因。

Monosomic 3, monosomic 5, monosomic 10, trisomic 4 and trisomic 5 as experiment materials, their meiosis behavior of pollen mother cell, pollen characteristics, physiological and biochemical characteristics of anther, section observation of pollen development and the structure of embryo sac were studied, and the effects on the seed set and transmission rate were analyzed.

本文以大白菜同源四倍体小孢子培养产生的且已鉴定为3号单体、5号单体、10号单体、4号初级三体、5号初级三体的非整倍体株系及二倍体株系等为试材,对各材料的结籽率、雄配子传递率进行了统计,并对花粉母细胞减数分裂行为、花粉特性、花粉发育和胚囊结构以及花药生理生化特性等方面进行了观察研究,进而分析了其对大白菜单体n-1雄配子和三体n+1雄配子传递率的影响。

Result:After removal of the bilateral lower molars, the remodeling and degenerative activities in different layers of the condyles increased, such as vesiculation of endoplasmic reticulum, distortion of mitochondria and increase of lysosome. Furthermore, the sequential arrangement of collagen fibrils was disturbed, the collagen network broke down and the proteoglycan granules decreased.

结果:细胞发生了不同程度的改建及退行性反应,如粗面内质网的扩张,线粒体肿胀,溶酶体增多等;基质中胶原原纤维的有序排列结构消失,胶原网受到破坏,同时蛋白多糖颗粒减少;基质中脂质小体及基质小囊等成分增多;负重区较非负重区的改变更显著。

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