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静脉注射

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ABSTRACT] Objective: To analyze the clinical properties of nonsensitive treatment of Kawasaki disease with intravenous injection of immunoglobulin.

目的:分析静脉注射丙种球蛋白对川崎病治疗不敏感的临床特点。

G at a low dose, sign of clinical infection was not observed and case of death was not found, mimicking the nonsymptomatic bacteremias model.

结论兔髂动脉球囊剥脱术后,给予或不给予高脂饮食,通过小剂量P.g静脉注射,可建立伴无症状菌血症的兔动脉粥样硬化模型。

Nonsynchronous electrical cardioversion together with intravenous loading dose 150mg of amiodarone was administed,and it would be repeated once after 10min when the treatment had no effect,followed by 0.5~1.5mg/min infusion for 24~36 hours,and amidarone was given to patients orally.The curative effect and side effect were observed.

对急性心肌梗死突然发作持续性室速和室颤的9例患者,在非同步电转复的同时静脉注射胺碘酮150mg,无效者10min后再重复一次,继之以0.5~1.5mg/min静点维持24~36h,同时口服胺碘酮,观察其疗效和副作用。

These conjugates were injected i.v. into immunodeficient mice bearing human KB tumors that overexpress the folic acid receptor.

将这些共轭化合物通过静脉注射入具有过表达叶酸受体人KB肿瘤的免疫缺陷鼠体内。

Sensory impairment and urinary dysfunction also had developed, and paraparesis persisted after the hypokalemia was corrected during hospitalization. An optokinetic nystagmus test showed bilateral sick waves and at the same time a cerebrospinal fluid analysis was normal. A blood examination disclosed borderline anti-nuclear antibodies and an elevated serum IgG level. Her cervical spinal cord showed mild swelling and contrast-enhancement when examined by magnetic resonance imaging. Neither a brain stem nor an optic nerve's lesion was found. Based on the above, acute transverse myelitis was diagnosed. All the symptoms present in the patient relieved gradually after 5-days of pulsed intravenous methylprednisolone therapy.

一50岁女性,因为眩晕、前额头痛、视力模糊、恶心、呕吐及两侧下肢无力而求诊,於低血钾矫正后,两侧下肢无力依然持续,并发生胸部以下感觉缺损及排尿困难等现象,视运动性眼振检查显示两侧波形障碍,脑脊液检查正常,血液学检查显示抗核酸抗体及血清IgG指数偏高,显影剂增强磁振造影显示颈段脊髓肿大且略为显影,并无脑干或视神经的病灶,诊断为急性横断性脊髓炎,给予静脉注射类固醇后症状逐渐缓解。

Results: six patients with vagal reflection were cured by intravenous atropine, 2 with pericardial hemorrhage were treated by pericardiocentesis, 1 patient developed hypotension due to occlusion of the mitral valve orifice by the catheter was corrected by shortening the duration of the catheter in left ventricle, the blood pressure of 1 patient with rapid atrial flutter was elevated by intravenous cedilanid, the blood pressure of 1 patient with hypotension of unknown origin was elevated by vasopressor and atropine.

结果:其中迷走神经反射6例,经静脉注射阿托品纠正;心包出血2例,心包穿刺抽液血压至正常;球囊导管阻塞二尖瓣口1例,缩短导管停留在左心室时间,可以避免低血压反应;快速心房颤动1例,使用西地兰减慢心室率,血压回升;原因不明1例,经使用升压药及阿托品后血压改善。

Increased intraocular pressure can occur as a consequence of oral, intravenous, inhaled, topical, periocular, or intravitreal corticosteroid therapy .

口服、静脉注射、吸入、局部应用、球周或玻璃体内应用皮质类固醇药物均可引起眼内压的升高[1–6,7·,8··, 9–11]。

Methods After peripherial intravenous administration of contrast material, spiral scan of portal venous system of 21 normal and 34 patients with cirrhosis were acquired with Elscint CT-Twin.

经外周静脉注射对比剂后,采用Elscint CT-Twin 对21例正常对照组和34例肝硬化组行门脉系的SCTA检查。

The colon temperatures were measured by a thermistor and the cytosolic free calcium concentration ([Ca2+]i) in dissociated brain cells was measured by Fura 2-AM. The results demonstrated that the pyretic action of egtazic acid (0.6 μmol, icv) was markedly inhibited by DEX (5 mg/kg,iv), but DEX (60-120 μmol/L) did′t affect [Ca2+]i in dissociated hypothalamus cells. Actinomycin D, which interferes with gene transcription (3 nmol, icv), completely abolished the antipyretic action of DEX on egtazic acid-induced fever.

结果发现:脑室灌注乙二醇双(2-氨基乙醚)四乙酸(0.6 nmol)引起家兔结肠温度明显升高,静脉注射地塞米松(5 mg/kg)显著抑制家兔乙二醇双(2-氨基乙醚)四乙酸性发热,地塞米松(60-120 μmol/L)并不影响下丘脑细胞内[Ca2+]i,而事先脑室灌注抑制基因转录的放线菌素D(3 nmol)则完全取消了地塞米松对乙二醇双(2-氨基乙醚)四乙酸性发热的解热作用。

Methods The blood stasis model was made by adrenaline administrated and coldness stimulation in rats, injected by ranine vein to observe the effect of PDS on quaque index of acute blood stasis mesenterium microcirculation and thrombogenesis in rats.

皮下注射肾上腺素加冰水冷浴法制备大鼠急性血瘀模型,舌下静脉注射给药,观察PDS对大鼠肠系膜微循环及体外血栓各指标的影响。

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