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静脉注射

与 静脉注射 相关的网络例句 [注:此内容来源于网络,仅供参考]

Rabbits received naloxone HCl intracerebroventricularlyor naloxone methiodide IV, a quaternary compound that does notcross the blood–brain barrier, 5 min before FENT treatment(5 or 50 g/kg, IV).

使用 FENT 治疗(5或50 g/kg ,静脉注射)前5min ,向家兔脑室内注射盐酸纳洛酮或静脉注射甲硫丁氨酸纳洛酮(一种四元化合物,不能透过血脑屏障)。

Objective: To prepare antidote of the type of dimercaptosuccinic acid for the treatment of animal with heavy lewisite intoxication through intravenous injection.

目的:制备抗重度路易氏剂中毒静脉注射用的二巯基丁二酸类抗毒药。方法:采用两步纯化法提纯口服胶囊中DMSA粗品,供动物实验中静脉注射用。

Intra-and extracellular concentrations of calcium, natrium, and potassium in fetal rat brains were measured in each group.

在不同时间内静脉注射bFGF,监测静脉注射bFGF后胎鼠脑细胞内外钙、钠、钾含量的变化。

Intravoneous injection of 0.5mg/kgBW jingsongling cause the 60 minutesrelaxation of goat gastric motor.The effects of jingsongling on the gastric motor ofgoat is major of effects of α2 -adrenoceptor agonist,as well as that of α1 -adrenoceptor agonist,the effects of α1 -adrenoceptor agonist concentrate at thecardic and pylorus;the function of α2 -adrenoceptor exit widely at cardic,pylorus,rumen,reticulum,omasum and abomasum,the function at different part is not the same.The lowestdose of jingsonglinginhibiting the amplitude and frequency of the gastricmyoelectric is 0.01mg/kgBW;the influence of jingsonglingto the gastricmotility of goat is dose-dependent,and the amplitude regain after the frequency ofgastric myoelectriv wave.When the goat revived,the gastric motor function is notreach the level of the normal;the major reason ofjingsongling cause the goat gastricrelaxation is the amplitude renew slowly.The intravoneous injection ofjingsonglingfirst,after 5 minutes,intravoneous injection the antagonist,the effect of prazosin and idazoxan mixed is best,which can fast reversal of theinhibition of jingsongling on the gastric motor of goat,the effects of idazoxan isgood,and the prazosin block the effection of jingsongling is better than that ofxylazine,especially at cardia and pylorus.When intravoneous injection of0.5mg/kgBW idazoxan and/or prazosin only,the amplitude of gastric myoelectric ingoat increases at a certain degree,but the frequency is not changed.

静脉注射0.5mk/kgBW的静松灵引起山羊胃蠕动弛缓长达60分钟;静脉注射0.5mg/kgBW的静松灵对山羊胃蠕动机能的影响是以α2肾上腺素受体作用为主的,兼有α1肾上腺素受体的作用,而且静松灵的α1肾上腺素受体的作用主要集中在幽门和贲门;α2肾上腺素受体的作用广泛存在于贲门、瘤胃、网胃、瓣胃、皱胃和幽门中,不同部位间有功能上的差异;静注0.01mg/kgBW的静松灵是对山羊的贲门、瘤胃、网胃、瓣胃、皱胃和幽门的肌电波频率和振幅均产生影响的最低剂量;静注一定剂量的静松灵对山羊胃蠕动的抑制作用与剂量呈相关性;而且山羊胃肌电波振幅的恢复比肌电波频率的恢复慢;苏醒时山羊的胃蠕动机能尚未恢复到正常状态;静松灵引起山羊胃弛缓的主要原因是由于胃肌电波的振幅恢复慢;静注0.5mg/kgBW的静松灵,5分钟后静注0.5mg/kgBW的拮抗剂时,咪唑克生+哌唑嗪的作用最好,能迅速地逆转静松灵对山羊胃蠕动的抑制作用,咪唑克生也有很强的拮抗作用,而哌唑嗪对静松灵的拮抗效果好于对隆朋的拮抗;单独静注0.5mg/kgBW的咪唑克生和/或哌唑嗪时,山羊的瘤胃、网胃、瓣胃、皱胃、贲门和幽门的肌电波振幅有一定程度的增强,而肌电波频率未见变化。

Methods The rat models of ARF were made with mercury perchloride by intraperitoneal injection.Five minutes later,the rats in nmhaFGF group and model group were respectively injected with 40 μg·kg-1 nmhaFGF and normal saline by lingual vein.The rats in control group were injected with normal saline.At the time of 30 min,90 min,24 h and 48 h,the contents of blood urea nitrogen,creatinine and urine protein were detected respectively.

腹腔注射氯化汞复制大鼠急性肾功能衰竭模型,5 min后,nmhaFGF组经舌静脉注射nmhaFGF 40 μg·kg-1,模型组经舌静脉注射等剂量的生理盐水,对照组大鼠麻醉后注射生理盐水,在30 min、90 min、24 h、48 h取血检测大鼠血液中尿素氮、肌酐和尿液中蛋白含量的变化。

Rats living over 96h were considered survival. To observe the protective effects of ALR gene on acute liver injury rat, thirty-six rats that were injected peritoneally with 50% CCl4 of 2ml/kg were divided into the flowing 6 group according to doses (50?g/kg and 200?g/kg ) and administration routes of pcDNA3-ALR DNA 4h after CCl4 injection: group 1, model group; group 2, ALR gene of 50μg/kg was injected venally; group 3, ALR gene of 200?

另取36只大鼠按2ml/kg腹腔注射50% CCL4,染毒后4h按不同剂量(50μg/kg 和200μg/kg )、不同注射途径(尾静脉注射、腹腔注射和联合注射)随机分为六组。1组:模型组,不注射ALR基因; 2组:尾静脉注射ALR基因 50μg/kg; 3组:尾静脉注射ALR基因 200μg/kg; 4组:腹腔注射ALR基因 50μg/kg; 5组:腹腔注射ALR基因 200μg/kg; 6组:联合注射(尾静脉和腹腔各注射ALR 基因100μg/kg)。

Atural killer cells promote CD8 Tm cell generation by regulating the extent of contraction.To generate CD8Tm cells,we adoptively transferred Thy-1.1 OT-I CD8 T cells in to host C57 mice(Thy-1.2) followed by injection of a virulance attenuated stain of Listeria Monocytogenes(ΔactA LM-OVA)via the tail veil 24h later.

K细胞通过调节初次应答收缩期而促进CD8Tm细胞产生为诱导产生CD8Tm细胞,我们将Thy-1.1~+ OT-I TCR转基因小鼠的CD8 T细胞通过尾静脉注射过继回输到野生型C57小鼠(Thy-1.2)体内,24h后通过尾静脉注射表达OVA的李斯特菌毒力减弱株(△actA LM-OVA)进行感染。

Convulsion was induced by intravenous strychnine 0.25 mgkg^(-1. Normal saline and the different dose of ketamine were intravenously administered, respectively. Mortality and duration of tonic state were observed. The EEG activity was monitored continuatively.

耳缘静脉注射士的宁0.25mgkg^(-1)制作惊厥模型,分别静脉注射氯胺酮或相应剂量的生理盐水,观察氯胺酮对家兔的死亡率和强直持续期的影响。

Objective The anticogant to the endotheliotoxin receptor is a promising drug to treat with the pulmonary arterial hypertension therotically.But in our country there was few systemical clinical trial about the drug used in the disease.We study the curative effect and the side effect of an anticogant to the endotheliotoxin receptor-Tetramethylpyrazine on the pulmonary arterial hypertension.Methods The64newborn patients were continually main line the tetramethylpyrazine by the mini pump and stimutaneously treated with the respiratory machine.The injec-tion speed was controlled from5μg/kg·min to10μg/kg·min,or to20μg/kg·min.

目的 探讨内皮素受体拮抗剂—阿魏酸钠微量泵持续静脉注射治疗新生儿肺动脉高压的疗效和不良反应方法对64例肺动脉高压的新生儿在呼吸机械通气的情况下同时用微量泵持续静脉注射阿魏酸钠,阿魏酸钠微量泵静脉注射的速度由5μg/开始,在保证体循环血压的基础上可以逐渐加量至10μg/,直至20μg/。

Methods: Twenty-four male SD rats were randomly divided into normal control (NC, n=6), protein C activator (PCA, n=6), lipopolysaccharide (LPS, n=6) and LPS+PCA group (n=6). NC group were given normal saline and PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. LPS group were administered lipopolysaccharide (10 mg/kg, 4h) by intraperitoneal injection method to make the model of septic shock rats. After the animal model was made, LPS+PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. 30 minutes after the injection, each rat was tested by Medlab biosignal analyzing system for observing mean arterial pressure and left ventricle function, the activity of LDH and iNOS in myocardium and the cuttings of myocardial tissues.

取SD雄性大鼠24只随机分成对照组、PCA组、LPS组和LPS+PCA组四组,对照组尾静脉注射生理盐水,PCA组尾静脉注射蝮蛇毒蛋白C激活物,LPS组腹腔注射LPS (10 mg/kg, 4h)的方法建立败血症休克的实验动物模型,在建立败血症的实验动物模型的基础上,尾静脉注射PCA (0.4 mg/kg)为LPS+PCA组,每只大鼠均于药后30 min时运用Medlab生物信号采集处理系统记录平均动脉压和左心室功能指标,并测定心肌中乳酸脱氢酶活性、诱导性一氧化氮合酶活性及心肌病理切片。

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