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After pretreatment with Ca(NO3)2,W7 and TFP in improving and blocking Ca2+*CaM messenger system, membrane damage in strawberry leaf was relief by Ca(NO3)2treatment, at the meanwhile, salt damage index was reduced, the content of prolinc and chlorophyll were enhanced, Pn, EVAP and GS were improved, ABA content in root and leaf was reduced, the content of ZTJAA, GA and ZT/ABA were enhanced, the content of unaturated fatty acid increased, whereas, saturated fatty acid content in leaf and root was reduced, the rate between unaturated fatty acid and saturated fatty acid and index of unaturated fatty acid were enhanced.

在Ca(NO_3)_2、W7和TFP喷叶预处理促进或阻碍Ca~(2+)·CaM信使系统传导后,发现Ca(NO_3)_2处理能减轻叶片的膜伤害,降低盐害指数,增加脯氨酸含量和叶绿素含量,提高净光合速率、蒸腾速率、气孔导度,降低根系和叶片中的ABA含量,增加ZT、IAA和GA含量和ZT/ABA比值;提高叶片和根系中的UFA含量、U/S比值和IUFA,降低根系和叶片中的SFA含量。

An added thoracoplasty to the thoracotomy approach, however, resulted in even greater residual reduction in PFTs at follow-up, with declines in percent-predicted FVC of 15%, percent-predicted FEV1 of 14%, and percent-predicted TLC of 8%.

然而,在开胸手术基础上增加的胸廓成形术反而导致术后肺功能的恢复减慢,预期最大通气量降低了15%,预期用力呼气量降低了14%,肺总量降低了8%。

An added thoracoplasty to the thoracotomy approach, howeer, resulted in een greater residual reduction in PFTs at follow-up, with declines in percent-predicted FC of 15%, percent-predicted FE1 of 14%, and percent-predicted TLC of 8%.

然而,在开胸手术基础上增加的胸廓成形术反而导致术后肺功能的恢复减慢,预期最大通气量降低了15%,预期用力呼气量降低了14%,肺总量降低了8%。

With the EMI shield, 66% of EMI can be reduced for more stable signals; while the Covered Chokes guarantee more efficient power and less heat generation in comparison with traditional Toroidal Coil Chokes. Additionally, the low RDS mosfets lower temperatures by 10–15°C in comparison with the traditional thicker versions, and last but not least, the Stabilizer Solid Polymer Aluminum Capacitors reduces power loss for more stable operations and durability—even under long-term use.

通过EMI shield技术,可以使电磁干扰冲突降低66%,让显卡更加稳定;与传统的电感线圈相比,全封闭电感能够在提高转换效率的同时降低发热量;而低阻抗MOSFET与传统电感相比,温度可以降低10~15度;另外更加稳定的贴片式铝聚合物固态电解电容能够提供更加稳定纯净的电流和更长的使用寿命——甚至是长时间满载使用。

The results indicate seawater concentration has no effect on absorbency, pH, relative density, dry matter and water contents in Aloe vera leaves. While, seawater stress causes increased MDA and membrane penetration, decreased phytochroms, prolines and NO_3~--N. It is beneficial to plant growth when irrigation was done with 10% seawater, because of higher ratio of K~+/Na~+ and lower ratio of Na~+/Ca~(2+). In addition, the polysaccharide contents in Aloe vera leaves increase twice and triplication under 25% and 50% seawater irrigation respectively, the barbaloin contents increase over triplication in var. 75% seawater and over twice in var. 50% seawater and var. 100% seawater. However, when seawater concentration exceeds 25% the growth of plant was inhibited, biomass decreased, flowering time was deferred, Ca~(2+) content decreased caused by Na~+ accumulation in leaves.

结果表明:在海水胁迫下,库拉索芦荟全叶汁的吸光度、酸度、相对密度、总固形物和水分含量总体上受海水浓度的影响不大;但芦荟叶的丙二醛含量和质膜透性增加,叶绿体色素、脯氨酸和硝态氮含量减少;在10%海水浇灌下,芦荟叶细胞具有较高的K~+/Na~+比和较低的Na~+/Ca~(2+)比,对库拉索芦荟的生长有一定的促进作用;在25%海水和50%海水浇灌下,芦荟多糖含量提高了2-3倍;在75%海水浇灌下芦荟甙含量增加了3倍多,在50%海水和100%海水灌溉下,其含量也增加了2倍多;当海水浇灌浓度超过25%后,库拉索芦荟的生长明显受到抑制,生物量降低,抽苔期推迟,同时,海水浓度的增加也导致芦荟叶中累积大量Na~+,从而使得芦荟叶中Ca~(2+)含量降低,但Mg~(2+)并没有受到海水浓度的影响;用浓度低于75%的海水浇灌库拉索芦荟可以降低剖面深度在0-100cm之间的土壤盐度;其抗氧化保护酶中超氧化歧化酶的酶活力相对保持稳定,过氧化物酶的酶活力在10%海水浇灌下明显提高,随后则下降,而过氧化氢酶的酶活力则随海水浓度的升高而下降。

12H treatment group compared with 12h model group: the level of SOD, MDA of the Gardenoside group is no different with the model group; the level of the SOD of the Baical in group increase and the level of MDA is no different; the level of SOD of the Concha Margatitifera Usta is no different and the MDA of it decreased than the model; the level of SOD of the Cholic Acid group increased and the level of MDA decreased; the level of SOD of the Hefang Group increased and the level of MDA of it decreased than the model group.

考虑nNos可能只是参与缺血早期的神经毒性作用。到缺血24小时这一时程已经减低或耗竭。大鼠局灶性脑缺血12小时模型组缺血脑组织P53光密度值含量较正常组明显增高(p<0.01)。珍珠母治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低。胆酸治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低(p<0.05)。栀子苷治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低(p<0.05)。

Wenxin capsule can promote endolethial cells releasing CO and synthesing 〓, enhance activity of HO, induce angiectasis through activating guanylic cyclase and adenglate cyclate of intracellar of vessel smooth muscle cell to enhance content of intracellar cGMP and AMP. 3. Wenxin capsule can inhibit releasing of platelet activiting factor 〓 which inhibit platelet aggregation and have antithrombosis function. 4. Wenxin capsule can promote activity of t-PA, reduce activity of PAI-1, strengthen fibrinolytic function, therefore inhibit fibrin disposition. 5. Wenxin capsule can inhibit synthesis of smooth muscle cellular DNA. Its function don't kill it directly but prevent transformation from period Go to peried S. 6. Wenxin capsule is an effective prescription of anti-coronary atherosclerosis atherosis. The results of image pattern analysis tastifies that area of lipid plaque in treating group reduce 54% and 58% in contrast with that in control group. The results of light, electro- microscopy show that structure of endolethial cell and smooth muscle cell in treating group is more integral than that in control group.

其作用机制之一是由于该药具有良好的调脂降脂作用,其不仅能明显降低血清LDL-c及升高HDL-c含量,且能有效阻止TC沉积于血管壁,从而保护内皮细胞结构完整;此外,温心胶囊尚具有明显的抗氧化作用,该药不仅能明显降低血浆LPO含量,且能提高SOD活性,同时其对血红素在细胞内代谢的调节作用也参与了抗氧化过程。2 温心胶囊能明显促进内皮细胞释放CO及合成〓,提高HO活性,通过激活血管平滑肌细胞内鸟苷酸环化酶及腺苷酸环化酶引起细胞内cGMP、AMP浓度增加而引起血管舒张。3 温心胶囊能明显抑制血小板激活因子〓的释放,从而抑制血小板聚集,抗血栓形成。4 温心胶囊能明显提高t-PA活性,降低PAI-1活性,增强纤溶功能,从而抑制纤维蛋白沉积。5 温心胶囊能明显抑制平滑肌细胞DNA合成,其抑制作用不是直接杀伤抑制,而是阻止细胞由Go期向S期转化。6 温心胶囊是抗冠状动脉粥样硬化的有效制剂,图象分析结果证实,该药治疗组脂斑面积比对照组缩小了54%和58%,光电镜结果显示内皮细胞及平滑肌细胞结构较为完整。

Results There was a downtrend in weight of dams on GD20 as the DBTD exposure dose increased. The weight of dam's utero significantly decreased in 30 mg/kg group. A significant decrease in fetal weights was observed in all DBTD groups, and fetal sizes in 20, 30 mg/kg groups. Exposure to DBTD from GD 12-19 resulted in a distinct increase in normalized anogenital distances in female fetuses, but no effects were seen in male ones.

结果 妊娠20d时,孕鼠体重随DBTD剂量升高呈下降趋势,但与对照组比较,差异无统计学意义,30mg/kg组孕鼠子宫重量显著降低;所有DBTD染毒组雄性、雌性胎鼠体重均显著降低,20、30m/kg组胎鼠体长显著降低;所有DBTB染毒组雌性胎鼠肛门距生殖器的标化距离显著增长,而雄性胎鼠无显著变化。

In 300mg/kg JSS treatment, the yolk color was 6 degree too. While added 500mg/kg JMH into the basal diet, the egg yolk color was up to 9 degree. Supplement of oxytetracycline + JMH or chlortetracy dine + JMH in the basal diet, the egg yolk color was up to 8 and 6 degree respectively and decreased one and three color unit respectively than that of JMH treatment. Added oxytetracyclimne + JSS or chlortetracycline + JSS, the egg yolk color was up to 8 and 6degree respectively. When added arsanilic acid + JSS, the egg yolk color was up to 7 degree and improved one color unit than that of JSS treatment. When added arsanilic acid +JMH to basal diet, the egg yolk color was up to 8 degree and decreased one color unit than that of JMH treatment.

饲料中添加300mg/kg的金闪闪,蛋黄颜色也是6级,没有产生增色效果,添加500mg/kg的加美黄,可使蛋黄颜色达到9级,提高3个比色单位,土霉素与加美黄配合使用,蛋黄颜色为8级,比加美黄组降低1级,金霉素与加美黄配合使用蛋黄颜色为6级,比加美黄组降低3级,土霉素与金闪闪配合使用可使蛋黄颜色升到8级,比金闪闪组提高2级,金霉素与金闪闪配合使用蛋黄颜色仍为6级,阿散酸与金闪闪配合使用,蛋黄颜色为7级,比金闪闪组提高1级,阿散酸与加美黄配合使用蛋黄颜色为8级,比加美黄组降低1级。

The cancers studied here were throat cancer, aute non- lymphoblas He leukem ia and acut lymphoblastic leukemia. The result s indicated that,(1)the levels of Leu, Lys, Ser Asn,Ile,Gly,and Thr were obviously decreased whea lse the levels of orn, Trp evidently increased in cultural liquid of throat can cer cell line indicating that the growth of HepⅡ cell consumed 7 amino acids and releaced 2 amino acids into cultural liquid.(2)the levels of Glu, Gls, Leu, Phe, Tyr and Trp were evidently increased while the levels of Thr , His, Ala decreased in plasma from patient with ANLL. These r esults were consistent with the previous report.(3)the levels of Gly, Trp and Phe in plasma from patients with ANLL evidently de creased and the levels of Ala, His increased after therapy indicating that cance r cells were growing in an anaerobic catabolism state.

这些癌肿包括喉癌HepⅡ细胞,急性非淋巴细胞白血病和急性淋巴细胞白血病,结果表明:(1)喉癌细胞株培养过程中亮氨酸、赖氨酸、丝氨酸、天冬酰胺、异亮氨酸、甘氨酸以及苏氨酸等水平明显降低,而色氨酸水平明显增加,说明喉癌细胞的生长繁殖必须依赖以上7种氨基酸同时释放了色氨酸;(2)急性非淋巴细胞白血病患者血浆中的谷氨酸、甘氨酸、亮氨酸、苯丙氨酸、酪氨酸和色氨酸等水平明显升高,而苏氨酸、组氨酸、丙氨酸等水平明显降低,这些结果与国际报道相一致;(3)经治疗后,ANLL患者血浆中甘氨酸、色氨酸和苯丙氨酸等水平明显降低,而丙氨酸、组氨酸等水平明显升高,表明肿瘤细胞处在无氧代谢。

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