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阻断剂

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The results obtained are as follows:(1) intrarenal artery injection of capsaicin (20, 40, and 60 nmol/kg) increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2) pretreatment with ruthenium red (40 mmol/kg), a capsaicin receptor antagonist, completely abolished the effect of capsaicin; and (3) pretreatment with a nitric oxide synthase inhibitor L-NAME ( N6-nitro-L-arginine methylester, 0.1 mmol/kg), significantly enhanced the ARNA response to capsaicin.

结果表明:(1)肾动脉内注射辣椒素20、40和60nmol/kg可呈剂量依赖性地兴奋肾传入纤维的活动,而动脉血压不变;(2)静脉内预先应用辣椒素受体阻断剂钌红(40 mmok/kg),可完全阻断辣椒素对肾传入纤维的兴奋作用。(3)静脉内预先注射一氧化氮合酶抑制剂L-NAME(0.1 mmok/kg),能延长并增强肾传入神经对辣椒素的反应。

The results obtained are as follows:(1) injection of 50, 100, and 200 nmol/kg adenosine into the renal artery increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2) pretreatment with 8-cyclopenthl -1,3-dipropylxanthine (DPCPX, 160 nmol/kg), an adenosine A1 receptor antagonist, partly abolished the effect of adenosine; and (3) pretreatment with a nitric oxide synthase inhibitor N w-nitro- L -arginine methylester (L -NAME, 0.1 mmol/kg) significantly enhanced the ARNA response to adenosine.

结果表明:(1)肾动脉内注射50, 100和200 nmol/kg腺苷可呈剂量依赖性地兴奋肾神经传入纤维的活动,而动脉血压不变。(2)肾动脉内预先应用选择性腺苷A1受体阻断剂DPCPX (160 nmol/kg),可部分阻断腺苷对肾神经传入纤维的兴奋作用。(3)静脉应用一氧化氮合酶抑制剂 L -NAME(0.1 mmol/kg)预处理,延长并增强了肾神经传入纤维对腺苷的反应。

The results obtained are as follows:(1) injection of 50, 100, and 200 nmol/kg adenosine into the renal artery increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2)pretreatment with 8-cyclopenthl-1,3-dipropylxanthine (DPCPX, 160 nmo//kg), an adenosine A1 receptor antagonist, partly abolished the effect of adenosine; and (3) pretreatment with a nitric oxide synthase inhibitor Nω-nitro-L-arginine methylester (L-NAME, 0.1 mmol/kg)significantly enhanced the ARNA response to adenosine.

结果表明:(1)肾动脉内注射50,100和200nmol/kg腺苷可呈剂量依赖性地兴奋肾神经传入纤维的活动,而动脉血压不变。(2)肾动脉内预先应用选择性腺苷A1受体阻断剂DPCPX(160nmol/kg),可部分阻断腺苷对肾神经传入纤维的兴奋作用。(3)静脉应用一氧化氮合酶抑制剂L-NAME(0.1mmol/kg)预处理,延长并增强了肾神经传入纤维对腺苷的反应。

Finally, we found that MK801, a complete antagonist of NMDA receptor, abolished increasing cell surface expression of NMDA receptor afforded by carnosine or clobenpropit.

最后我们还发现NMDA受体完全阻断剂MK801能阻断肌肽和clobenpropit引起的NMDA受体膜上表达增加。

The inhibitory effect of GABA could be blocked by GABAAreceptor antagonist bicuculline bicuculline methiodide and Cl- channel blocker picrotokin.

GABA的抑制效应大部分可被GABAA受体选择性拮抗剂荷苞牡丹碱甲基碘化物和Cl-通道阻断剂印防己毒素所阻断,而单独灌流BMI和PTX对RVLM神经元主要起兴奋作用。

ConclusionCattail pollen decoction can increase the contraction of isolated uterine smooth muscle strips in nonfertile rats.

结论蒲黄能增加子宫平滑肌条收缩活动,其作用可被L型Ca2 通道阻断剂异搏定阻断。

Results:The results showed that the apoptosis of neutrophiles was promoted by the treatment of 1 μg/ml LPS and 1 μm A23187,in 6 hours and 12 hours respectively.The apoptosis of neutrophil was markedly delayed by a PLA2 inhibitor,4-bromophanacyl bromid (p-BPB 0.01~1 mM).

结果:脂多糖(LPS 1μg/ml)和钙离子载体(A23187 1 μmol/L)等PLA2激动剂分别在共同培养6 h和12 h之内促进中性粒细胞凋亡,而PLA2阻断剂4-溴苯甲酰溴甲烷(p-BPB 0.01~1 mmol/L)可明显减弱上述激动剂的促凋亡作用。

These effects could be blocked or attenuated by their corresponding antagonists or antibodies.

这些作用能被相应的阻断剂或抗体所阻断或抑制。

Intracerebral ventricular injection of M receptor blocker,Atropine(10 μ L 1× 10- 7 mol/L), failed to block the gastric contraction effect of cerebral SP.

侧脑室注射 M 受体阻断剂阿托品 10 μ L(1× 10- 7 mol/L),不能阻断脑内 P物质对胃的收缩效应。

Pretreatment with KATP channel blocker glibenclamide (Gli; 20μmol/L) could partially block the effects of H2S pretreatment (100μmol/L).

应用KATP通道阻断剂格列苯脲( Gli; 20μmol/L ),可部分阻断H2S对乳头状肌细胞的电生理效应。

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