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The results obtained are as follows:(1) intrarenal artery injection of capsaicin (20, 40, and 60 nmol/kg) increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2) pretreatment with ruthenium red (40 mmol/kg), a capsaicin receptor antagonist, completely abolished the effect of capsaicin; and (3) pretreatment with a nitric oxide synthase inhibitor L-NAME ( N6-nitro-L-arginine methylester, 0.1 mmol/kg), significantly enhanced the ARNA response to capsaicin.

结果表明:(1)肾动脉内注射辣椒素20、40和60nmol/kg可呈剂量依赖性地兴奋肾传入纤维的活动,而动脉血压不变;(2)静脉内预先应用辣椒素受体阻断剂钌红(40 mmok/kg),可完全阻断辣椒素对肾传入纤维的兴奋作用。(3)静脉内预先注射一氧化氮合酶抑制剂L-NAME(0.1 mmok/kg),能延长并增强肾传入神经对辣椒素的反应。

The results obtained are as follows:(1) injection of 50, 100, and 200 nmol/kg adenosine into the renal artery increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2) pretreatment with 8-cyclopenthl -1,3-dipropylxanthine (DPCPX, 160 nmol/kg), an adenosine A1 receptor antagonist, partly abolished the effect of adenosine; and (3) pretreatment with a nitric oxide synthase inhibitor N w-nitro- L -arginine methylester (L -NAME, 0.1 mmol/kg) significantly enhanced the ARNA response to adenosine.

结果表明:(1)肾动脉内注射50, 100和200 nmol/kg腺苷可呈剂量依赖性地兴奋肾神经传入纤维的活动,而动脉血压不变。(2)肾动脉内预先应用选择性腺苷A1受体阻断剂DPCPX (160 nmol/kg),可部分阻断腺苷对肾神经传入纤维的兴奋作用。(3)静脉应用一氧化氮合酶抑制剂 L -NAME(0.1 mmol/kg)预处理,延长并增强了肾神经传入纤维对腺苷的反应。

The results obtained are as follows:(1) injection of 50, 100, and 200 nmol/kg adenosine into the renal artery increased the renal afferent nerve activity in a dose-dependent manner with unchanged arterial pressure;(2)pretreatment with 8-cyclopenthl-1,3-dipropylxanthine (DPCPX, 160 nmo//kg), an adenosine A1 receptor antagonist, partly abolished the effect of adenosine; and (3) pretreatment with a nitric oxide synthase inhibitor Nω-nitro-L-arginine methylester (L-NAME, 0.1 mmol/kg)significantly enhanced the ARNA response to adenosine.

结果表明:(1)肾动脉内注射50,100和200nmol/kg腺苷可呈剂量依赖性地兴奋肾神经传入纤维的活动,而动脉血压不变。(2)肾动脉内预先应用选择性腺苷A1受体阻断剂DPCPX(160nmol/kg),可部分阻断腺苷对肾神经传入纤维的兴奋作用。(3)静脉应用一氧化氮合酶抑制剂L-NAME(0.1mmol/kg)预处理,延长并增强了肾神经传入纤维对腺苷的反应。

Finally, we found that MK801, a complete antagonist of NMDA receptor, abolished increasing cell surface expression of NMDA receptor afforded by carnosine or clobenpropit.

最后我们还发现NMDA受体完全阻断剂MK801能阻断肌肽和clobenpropit引起的NMDA受体膜上表达增加。

The inhibitory effect of GABA could be blocked by GABAAreceptor antagonist bicuculline bicuculline methiodide and Cl- channel blocker picrotokin.

GABA的抑制效应大部分可被GABAA受体选择性拮抗剂荷苞牡丹碱甲基碘化物和Cl-通道阻断剂印防己毒素所阻断,而单独灌流BMI和PTX对RVLM神经元主要起兴奋作用。

ConclusionCattail pollen decoction can increase the contraction of isolated uterine smooth muscle strips in nonfertile rats.

结论蒲黄能增加子宫平滑肌条收缩活动,其作用可被L型Ca2 通道阻断剂异搏定阻断。

Results:The results showed that the apoptosis of neutrophiles was promoted by the treatment of 1 μg/ml LPS and 1 μm A23187,in 6 hours and 12 hours respectively.The apoptosis of neutrophil was markedly delayed by a PLA2 inhibitor,4-bromophanacyl bromid (p-BPB 0.01~1 mM).

结果:脂多糖(LPS 1μg/ml)和钙离子载体(A23187 1 μmol/L)等PLA2激动剂分别在共同培养6 h和12 h之内促进中性粒细胞凋亡,而PLA2阻断剂4-溴苯甲酰溴甲烷(p-BPB 0.01~1 mmol/L)可明显减弱上述激动剂的促凋亡作用。

These effects could be blocked or attenuated by their corresponding antagonists or antibodies.

这些作用能被相应的阻断剂或抗体所阻断或抑制。

Intracerebral ventricular injection of M receptor blocker,Atropine(10 μ L 1× 10- 7 mol/L), failed to block the gastric contraction effect of cerebral SP.

侧脑室注射 M 受体阻断剂阿托品 10 μ L(1× 10- 7 mol/L),不能阻断脑内 P物质对胃的收缩效应。

Pretreatment with KATP channel blocker glibenclamide (Gli; 20μmol/L) could partially block the effects of H2S pretreatment (100μmol/L).

应用KATP通道阻断剂格列苯脲( Gli; 20μmol/L ),可部分阻断H2S对乳头状肌细胞的电生理效应。

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Finally, according to market conditions and market products this article paper analyzes the trends in the development of camera technology, and designs a color night vision camera.

最后根据市场情况和市面上产品的情况分析了摄像机技术的发展趋势,并设计了一款彩色夜视摄像机。

Only person height weeds and the fierce looks stone idles were there.

只有半人深的荒草和龇牙咧嘴的神像。

This dramatic range, steeper than the Himalayas, is the upturned rim of the eastern edge of Tibet, a plateau that has risen to 5 km in response to the slow but un stoppable collision of India with Asia that began about 55 million years ago and which continues unabated today.

这一引人注目的地域范围,比喜马拉雅山更加陡峭,是处于西藏东部边缘的朝上翻的边框地带。响应启始于约5500万年前的、缓慢的但却不可阻挡的印度与亚洲地壳板块碰撞,高原已上升至五千米,这种碰撞持续至今,毫无衰退。