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钙血症

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Hypercalcemia is an abnormally high level of calcium in the blood.

钙血症是一个异常高水平的钙在血液中。

Diuresis can also increase calcium excretion; the development of hypocalcemia will make signs and symptoms of hypermagnesemia worse.

但是利尿也能使钙排出增加,低钙血症反会使高镁血症的病情恶化。

Methods 82 infants with hyperbilirubinemia were divided into two groups by gravidity time: preterm group and term group. Serum calcium was measured before and after phototherapy.

方法将82例新生儿高胆红素血症患儿按胎龄分为足月儿及早产儿两组,足月儿47例,早产儿35例,测定光疗前后血清钙水平,比较两组低钙血症的发生率。

While deficiency of vitamin D impacts health negatively, ingestion of extremely high doses of vitamin D can cause hypercalcemia, a condition in which the blood's calcium level is above normal.

虽然缺乏维生素D的健康产生不利的影响,摄取的极其高剂量的维生素D可导致高钙血症,一个条件,其中血液的钙水平高于正常。

Results Incidence rate of hyponatremia,hyperpotassemia and hypocalcemia was increased with the number of organ dysfuntion.

结果 随着器官障碍数的增加,低钠血症、高钾血症、低钙血症的发生率也显著增加。

The causes of hypocalcemia are either from increased loss of calcium from the circulation or from insufficient entry of calcium into the circulation.

钙血症的原因是无论从增加钙流失的流通不足的钙进入到流通。

Many consumers believe that calcium and vitamin D can only choose one product, this is a misunderstanding, due to individual differences in the prevalence of the human body, not all at the same time also the lack of both calcium deficiency of vitamin D, some long-term use of vitamin D it will inhibit the body's own vitamin D formation, or trigger a large number of vitamin D intake and Yun Ji-poisoning, poisoning symptoms of hypercalcemia caused by liver and kidney dysfunction and soft tissue calcification, manifested as loss of appetite, unresponsive, rhythm arrhythmia, difficulty in breathing, nausea and vomiting, polydipsia polyuria, constipation and so on.

许多消费者认为,补钙只能选择与维生素D合一产品,这是一种误解,由于人体个体差异普遍存在,并不是所有的人同时既缺钙又缺维生素D,部分人长期服用维生素D反而会抑制体内自身维生素D的形成,或是引发维生素D大量摄入而积蕴中毒现象,中毒症状为高钙血症引起的肝肾功能损伤和软组织钙化,表现为食欲减退、反应迟钝、心律不齐、呼吸困难、恶心呕吐、烦渴多尿、便秘等。

Lymphopenia and hypocalcemia were the prominent laboratory findings. Severe thrombocytopenia was rarely observed.

实验室发现最明显的特征是淋巴细胞减少症和低钙血症,很少观察到严重的血小板减少症。

PTH gene and protein expression levels were up-regulated in parathyroid gland and thymus in 1αase knock-out mice with hypocalcaemia.

1αase-/-小鼠表现为低钙血症?与正常血钙的WT小鼠相比,PTH基因和蛋白的表达水平在1αase-/-小鼠胸腺组织中均明显升高?

Indeed, patients with more seere hypomagnesaemia (grade 3 to 4) should be offered intraenous magnesium replacement with a goal of normalising serum magnesium concentrations to decrease the risk of cardiac eents, constitutional symptoms, and risk of hypocalcaemia.

当然,较严重低镁血症(3-4级)患者应实施静脉镁补充替换替换治疗,使血镁浓度达到正常标准,降低心血管事件、低钙血症发生风险、改善患者全身症状。

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