钙
- 与 钙 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Hypocalcemia is thought to be caused by immaturity of parathyroid glands that make calcium.
低钙血症被认为是不成熟造成的甲状旁腺,使钙。
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In addition to a complete medical history and physical examination, diagnosis of hypocalcemia is made by testing the blood for the amount of calcium.
除了完整的病史和身体检查,诊断低钙血症是由测试血液中钙的数量。
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Absorption of HF may cause hypocalcemia due to HF's fixation of blood calcium .
HF的吸收会由于HF对血钙固定导致低血钙症。
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Absorption of HF may cause hypocalcemia due to HF's fixation of
HF的吸收会由于HF对血钙固定导致低血钙症。
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Results:The incidence of hypocalcemia in FDP group was 64.10%, and the one in nonFDP was 15.58%.
结果::1, 6二磷酸果糖组低钙发生率64.10%,非果糖组低钙发生率15.58%,,用四格表资料的χ2检验,果糖组与非果糖组的χ2=38.01,P.05,两组比较差异有统计学意义。
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Free calcium was unchanged in group A, but there were fifty percent of patients in group B had postoperative hypocalcemia. The duration of temporary hypocalcemial was from 3 days to 49 days (mean=16.5 days).
在A组中术前术后血钙离子都没有显著变化,但在B组却有50%的病例发生暂时性的低血钙症,发生的时间最短3天,最久49天,平均16.5天。
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Therefore, evaluate serum magnesium when hypocalcemia is present and particularly if hypocalcemia is refractory to initial calcium therapy.
因此,低钙时应估计血清镁浓度,尤其是低钙对治疗反应不佳时。
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In the setting of renal failure, there are a number of abnormalities of the calcium-vitamin D metabolic pathway, such as hypocalcemia, hyperphosphatemia, and increased PTH levels, that ultimately lead to renal bone disease.
在肾功能衰竭的设置,有一些异常的钙的数量维生素D的代谢途径,如低血钙,血磷,甲状旁腺素水平的增加,最终导致肾性骨病。
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There are two stages in the collapse of CaSiO_3 perovskite under decompression: the breakage of SiO_6 octahedron and the breakage of CaO_(12) icosahedron.
钙钛矿型CaSiO_3解压缩成非晶态时,存在两个结构破坏阶段:破坏硅氧八面体和破坏钙氧二十面体。
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And 1% in parenchyma, extracellular fluid and blood, called the Immiscibility calcium pool.
其余1%的钙存在于软组织,细胞外液及血液中,称之为混溶钙池。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。