钙

Endogenous fecal excretion was measured in a subset of children, and net calcium balance was calculated.

在部分孩子中测定钙的排泄量，计算钙的代谢平衡。

According to experimental results and etiopathogenisis and pathology of osteoporosis , and analyzing review investigation on hepcidin、associations of bone metabolism and iron metabolism and relations of ferri ion and calcium ion,we can make conclusions as follows: 1、We applied single manmade intervention approach to induce osteoporosis model in this study, there was no other influential factor. In the termination of test , rats bone density and bone histological anatomy indicated that osteoporosis model was creditable,meanwhile hepatic functional enzyme and hepatic microtome section demonstrated that hepatic function had noci-influence induced by medicinal herbs resource. Accordingly, we could believe that hepatic hepcidin gene expression and serum hepcidin contents can represent internal iron metabolism changes at selected test time. 2、In this investigation, different findings between test group and control group indicated that:according to changes of hepcidin gene expression measured by RT-PCR and changes of serum hepcidin contents determined by ELISA kit , there were correlations between hepcidin and rat osteoporosis model induced by retinoic acid. 3、Under the condition of this investigation,there was a interactive hypothesis as follows: Hepcidin iron metabolism←→ferriion←→calcium ion←→osteoporosis.

根据本次实验结果，对骨质疏松的病因病理的认识及铁代谢与铁调素、骨代谢与铁代谢关系、铁离子与钙离子的关系系列研究的回顾，经分析得出如下结论：1、本研究骨质疏松模型采用单一人工干预方法，没有其他影响因素；实验结束时大鼠骨密度、骨组织学检查表明骨质疏松形成，同时肝脏功能酶和HE切片显示肝脏功能未受药源性产生明显损伤性影响；因此，同期不同时间组大鼠肝脏铁调素基因表达、血清铁调素含量可以代表体内铁代谢不同时间的变化。2、在本研究中，模型组与对照组研究结果的差异表明：铁调素基因RT-PCR变化、血清铁调素ELISA测定含量变化与维甲酸制作的大鼠骨质疏松模型形成有实验相关性。3、在本研究平台下，形成了一个互相影响的假设图：铁调素←→铁代谢←→铁离子←→钙离子骨质疏松。

Endotoxin and tetrahydrobiopterin enhanced the activation of NO synthesis and ACO, but L-arginine, the precursor of NO, showed noticeable inhibition on the elevation of NO metabolites and the degree of ACO. In addition, L-arginine show prominent inhibition on myocardial fibrinosis.

诱导iNOS活性的药物也可加重动脉钙超负荷损害，然而NO合成的前体物质L-精氨酸对ACO早期血NO〓〓/NO〓〓升高有明显抑制，同时抑制ACO，尤其对心脏钙超负荷损害有明显的保护作用。

Sheet-like particles are mainly aluminosilicates and calcium compounds on which iron compounds are attached, and the majority of floccule particles contain calcium compounds.

片状颗粒主要是附有铁化合物的铝硅酸盐和钙盐，絮状颗粒则主要含钙化合物。c不同采样点单个颗粒的形貌、组分有明显差异。

However, in principle, three months of BB can be calcium, some doctors do not advocate prematurely because of calcium may lead to premature fontanel closure BB.

但原则上说，三个月个BB可以补钙，有些医生不主张过早补钙是因为过早可能会导致BB囱门早闭。

My baby is the case, post-mortem examination when the baby is small blood calcium deficiency, the doctor said that calcium deficiency led to baby later closed fontanel.

我家孩子情况也是这样，体检时验小血孩子是缺钙的，医生说孩子缺钙导致囱门迟闭。

However, simply not to fontanel closure without calcium, calcium deficiency is caused more harm than good, and open and four months from BB fontanel closure is also a long time ago.

但若只单纯为了囱门不早闭而不补钙，而引起缺钙则得不偿失了，旷且四个月的BB囱门离闭合还很早。

Patients with FOP do not have high levels of calcium, so it is fine to eat a diet with adequate calcium and vitamin D intake to preserve the health of the normal skeleton.

FOP患者不是因为钙的含量很高，因此吃足够的钙和维生素D的食物，以保持正常的骨骼健康是必要的。

Calcium formate may offer significant advantages as a dietary calcium supplement.

提示甲酸钙可能是一种极有优势的钙补充剂。

We discussed the influence of calcium fortifier, stabilizer and poly-phosphoric acid sodium on the stability of high calcium fluid milk.

探讨了钙剂的选择依据及其在牛乳中的合适添加量，阐述了钙剂、稳定剂和多聚磷酸盐对高钙液体乳热稳定性的影响机理，确定了生产高钙牛乳的最优配方是钙剂用量为60mg/100mL、CMC—Na为0.3％、多聚磷酸钠为0.2％。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。