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酪蛋白酸

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To get full health benefits from tea, however, don't use milk in it - some casein-related proteins in the milk bind the polyphenols - but instead acidify it with a little lemon (or even take your vitamin C with the tea) to enhance absorption of the tea polyphenols.

获得全面的健康的好处,从茶叶,不过,不要使用牛奶在它-一些酪蛋白相关蛋白的牛奶约束,多酚类物质-而是酸化,它与少许柠檬(或什至采取您的维生素C与茶)加强吸收的茶多酚。

The present experiment was conducted to determine the optimum dietary zinc requirement of juvenile cobia with zinc methionine as zinc sources, by using a purified diet based on casein-gelatin as the protein sources.

利用酪蛋白和明胶为蛋白源的精制饲料,以有机态的蛋氨酸锌做为锌源,来研究军曹鱼幼鱼对锌的最适需要量。

Methods Of 219 early pregnancy women taking RU486,30 cases as effective group,23 cases as ineffective group and 16 cases as curette abortion group,and they were labeled immunohistochemically with PR,ER and EGFR,PKC,raf-1,MEK-1 and Bcl-2,respectively.

219例服用RU486早孕药物流产者,对其有效组30例,失败组23例,人流组对照16例,以免疫组化法分别标记孕激素受体、雌激素受体、表皮生长因子受体、酪氨酸蛋白激酶C、分裂激活蛋白激酶(MEK-1)、细胞转化分裂介质(raf-1)和抗凋亡蛋白(Bcl-2)。

Methods Of 219 early pregnancy women taking RU486,30 cases as effectiv e group,23 cases as ineffective group and 16 cases as curette abortion group,and t hey were labeled immunohistochemically with PR,ER and EGFR,PKC,raf-1,MEK-1 and Bcl-2,resp ectively.

219例服用RU486早孕药物流产者,对其有效组30例,失败组23例,人流组对照16 例,以免疫组化法分别标记孕激素受体、雌激素受体、表皮生长因子受体、酪氨酸蛋白激酶C、分裂激活蛋白激酶(MEK-1)、细胞转化分裂介质(raf-1)和抗凋亡蛋白(Bcl-2)。

Methods The activity of PTK, PKCα,βI ,βII ,γ,ε,ξ and the active station of NF-κB in large bowel carcinoma tissues was detected by using membraneous conjugated method, laser scanning confocal microscopy and immuriohistochemistry respectively in the animal model of nude mouse transplantation tumors of large bowel carcinoma.

以大肠癌裸鼠移植瘤为动物模型,用膜结合法、激光共聚焦显微镜和免疫组织化学分别测定了大肠癌组织蛋白酪氨酸激酶、蛋白激酶Cα、βI、βⅡ、γ、ε和ξ的活性以及核因子-κB的活化状况。

By contrast, dexamethasone, a synthetic glucocorticoid, induces a chromaffin cell phenotype characterized by increased cell flattening, loss of neuritic processes, increased chromogranin A and tyrosine hydroxylase proteins, and decreased amounts of secretogranin II and neurofilaments.

作为对照,地塞米松诱导嗜铬细胞表型,具有特征的增加细胞变扁、丧失神经突起,增加嗜铬粒蛋白A和酪氨酸羟化酶蛋白,减少分泌粒蛋白II和神经丝的数量。

The excess fraction is crucial in effecting sufficient CMC adsorption during the mixing process of yoghurt and CMC solution. Pasteurism had no influence on the CMC adsorbed amount in direct acidified milk drinks; however, it led to the decrease of CMC adsorbed amount in yoghurt drinks.

生产酸性乳饮料的巴氏杀菌过程,对调酸型乳饮料中CMC在酪蛋白上的吸附量没有显著影响,但是这一加热杀菌过程可以降低CMC在发酵型乳饮料中蛋白颗粒的吸附百分比及吸附量。

We also showed that coadministration of AG825 or AG1478 will enhance the efficacy of cytotoxic AGent cisplatin.

了解5种人类非小细胞肺癌细胞系A549、H322、H358、H460、H1299的HER2和EGFR蛋白表达水平,探讨5种细胞系HER2蛋白表达水平与顺铂IC50之间的关系;观察HER2和EGFR小分子酪氨酸激酶抑制剂AG825和AG1478对5种细胞系的生长抑制作用;观察AG825、AG1478联合顺铂对5种细胞系的协同生长抑制作用;研究AG825、AG1478联合顺铂对H322、H1299细胞系细胞增殖、细胞周期、细胞凋亡和坏死的影响;研究AG825、AG1478联合顺铂所致H322、H1299细胞系EGFR、磷酸化ERK和总ERK表达的变化,探讨协同生长抑制作用的机制。

CML is cytogenetically marked by the philadelphia chromosome, which originates from a reciprocal translocation between chromosome 9 and 22 and is molecularly marked a chimeric bcr-abl gene, resulting from juxta-positive of the abl proto-oncogene on chromosome 9 with the bcr gene, which is normally located on chromosome 22. The chimeric bcr-abl gene expression an 8. 5kb hybrid mRNA transcript giving rise to a 210-KD fusion protein (P210〓) with increased tyrosine kinase activity. P210〓 plays a key role in the pathogenesis of CML. The continuous cell line K562 was established from the pleural effusion of a 53-year-old female with CML in terminal blast crisis, and was a human erythroleukemia line, contained Ph chromosome.

绝大多数慢粒患者白血病细胞中具有Ph染色体,是由9号染色体长臂3区4带和22号染色体长臂1区1带相互易位形成,即t(9;22),使位于9q〓的c-abl原癌基因在第二外显子的5'端断裂并易位到22 q〓的M-bcr基因第2或第3外显子的3'端,形成异常的bcr-abl嵌合基因,该基因转导出异常的mRNA,编码并翻译出P210蛋白,该蛋白具有很强的酪氨酸激酶活性,使粒细胞发生恶性增殖。K562细胞属于慢粒急变、红白血病细胞株,具有Ph染色体。

Prophenoloxidase is a key enzyme for mediating melanotic encapsulation of oocysts,which may be transformed into active phenoloxidase by limiting proteolysis.

而前酚氧化酶(Prophenoloxidase,PPO)是介导卵囊黑化包被反应的一种关键酶,活化后可催化酪氨酸羟基化为二羟基苯基丙氨酸,二羟基苯基丙氨酸和多巴胺可进一步氧化为具有活性的苯醌活性,苯醌即可介导蛋白交联并聚合成黑色素层固定、隔离或杀死入侵的病原体。

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