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The effect of CPI-deficiency on the photophysical and photochemical characteristics of the mutant alga were studied in detail.

本文首先详细地研究了CPI的部分缺失对突变型衣藻的光物理和光化学反应的影响。

The effect of the CPI-deficiency on the photophysical and photochemical characterization of the mutant alga was further studied.

进而详细地研究了CPI的部分缺失对突变型衣藻的光物理和光化学反应的影响。

Through comparing the growth and exogenous gene expression of recombinant Anabaena sp.

比较无选择压力下连续传代的转基因鱼腥藻 712 0在不同培养基中的生长和外源基因表达,证实没有发生质粒部分缺失,但转基因鱼腥藻在无选择压力下会降低重组质粒拷贝数。

QKI is an RNA binding protein essential for normal myelination, and are abundantly expressed in the central nervous system.

QKI是一类RNA结合蛋白,在神经系统中表达含量很高,基因部分缺失小鼠由于神经髓鞘发育障碍,在出生后10天出现严重的震颤表型。

Meta analysis shows, only sex of 3 heredity much condition and male not Yo is remarkable and relevant, namely AZFc division share is short of break, androgen accepts put oneself in another's position genen length and methylene 4 hydrogen folic acid is reductive enzymatic gene much condition.

Meta分析显示,仅有3个遗传多态性和男性不育显著相关,即AZFc区部分缺失、雄激素受体基因n长度和亚甲基四氢叶酸还原酶基因多态性。

Methods Amplify the cDNA sequence encoding truncated HCV gene, with a part of carboxyl-terminus deleted, by PCR. Synthesize the mimic epitope at E2 region of HCV and seven T or Th cell epitope genes of NS3-NS5 respectively. Bind HCV core gene with a part of carboxyl-terminus deleted to the synthetic epitope gene by PCR, then clone into eukaryotic expression vector pcDNA3.1 and transiently transfect COS7 cells.

用PCR方法扩增核心区羧基端部分缺失的基因片段;分别合成HCV E2区模拟表位和NS3~NS5 7个T或Th细胞表位基因;PCR方法将羧基端部分缺失的HCV核心区基因与合成的表位基因串联,克隆入真核表达载体pcDNA3.1,并通过脂质体瞬时转染COS7细胞。

In vitro\% peritrophic membrane assay showed that all three truncated enhancin lost their mucin degrading ability, while a full length recombinant enhancin was active in the assay.

通过体外降解围食膜的方法检测这些部分缺失的增强蛋白的活性,结果证实这三种蛋白均失去了增强蛋白的降解围食膜粘蛋白的活性。

Using CCA, karyotype abnormalities were found in 18 (37.5%) of 48 cases, among which complex karyotypes were found in 4 (8.3%) cases, a deletion of chromosome 5/a deletion of the long arm of chromosome 5 (-5/5q-) in 5 (10.4%) cases,-7/7q- in 5 (10.4%) cases, trisomy 8 in 8 (16.6%) cases, 20q- in 2 (4.6%) cases, unconsistent translocations in 3 (6.2%) cases.

细胞遗传学分析检出染色体异常18例(37.5%),其中复杂异常4例(8.3%)、5号染色体缺失或5号染色体长臂部分缺失5例(10.4%)、7号染色体缺失或7号染色体长臂部分缺失5例(10.4%)、8号染色体增加8例(16.6%)、20号染色体长臂部分缺失2例(4.6%)、不一致的易位3例(6.2%)。

Objective To study the effects of 20/21 bp partial deletion mutation (from nt 1 748 or nt 1 747 to nt 1 767) in the core promoter region of hepatitis B virus genome complicated by precore stop condon mutation at nt 1 896 on the expression of the viral antigens.

目的为研究乙型肝炎病毒核心启动子20/21bp部分缺失(nt1748/1747至nt1767)及同时存在的A1896点变异对病毒抗原表达的影响。方法利用前期构建的HBV全基因的重组载体转染HepG2细胞后,对病毒抗原进行ELISA检测及Western-blotting分析。

Refractors are unstable or even missing in the middle and there is an abnormal near surface belt in the east.

中东某稀疏三维工区表层条件复杂,工区中部折射层不稳定且部分缺失,工区东部有一地表异常区带,模型静校正和折射静校正都未能很好地解决该区的静校正问题。

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