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Lung Ang Ⅱ correlated positively with MIP-2 in BALF in all study rats. These results indicate that local angiotensin system is involved in the pathogenesis of VILI and suggest that blockade of Ang Ⅱ might have potential therapeutic implications in alleviating VILI.

这些结果表示局部血管紧缩素系统与机械通气导致肺伤害的病理生理机转有关,并且建议阻断血管收缩素可能有治疗机械通气导致肺伤害的潜在功效。

Compare the lung injury extent and the important physiological parameters to determine the optimal condition for rabbit ventilator induced lung injury, Choosing another 24 new Zealand rabbit, randomly divided into 4 groups:(1)conventional ventilation+PEEP group;(2)ventilator induced lung injury group: employ the experimental condition determined by the first part experiment;(3)VILI+HO-1 inducer hemin group:pre-treated with HO-1 inducer, the ventilation method is the same with VILI group;(4)VILI+HO-1 inhibitor Zn-PP group(Zn-PP group):pre-treated with HO-1 inhibitor, the ventilation condition is the same with VILI group.

第二部分24只家兔随机分为4组:(1)常规潮气量+PEEP组;(2)呼吸机所致肺损伤组:使用实验第一部分确定的最适条件;(3)呼吸机所致肺损伤+HO-1诱导剂干预组:预先给予氯铁血红素,通气条件同VILI组;(4)呼吸机所致肺损伤+HO-1阻断剂干预组:预先给予锌原卟啉,通气条件同VILI组。

Traditional machinery aerates send lung ventilator dependency pneumonic , lung ventilator (the happening of the complication such as ALI), remove machine difficulty to lung ventilator is depended on and be being sent. In June 2005 ~2007 year in December, we are used without achieve machinery to aerate auxiliary cure spends organic phosphor again toxic choline can high resemble and ARF 30 exemple, effect satisfaction.

传统机械通气致呼吸机相关性肺炎、呼吸机相关肺损伤等并发症的发生,并对呼吸机依靠而致撤机困难。2005年6月~2007年12月,我们采用无创机械通气辅助治疗重度有机磷中毒胆碱能危象并ARF 30例,效果满足。

Breath fails (Respiratory Failure, because lung aerates,RF) is obstacle of function of insufficient, dispersion and lung aerate / blood stream scale is maladjusted wait for an element, low oxygen blood appears when making static interest position issues breath disease and carbon dioxide retention, cause a series of physiology functions and metabolization thereby disorder clinical and integrated proof.

概要: 呼吸衰竭(respiratory failure,RF)是由于肺通气不足、弥散功能障碍和肺通气/血流比例失调等因素,使静息状态下呼吸时出现低氧血症和二氧化碳潴留,从而引起一系列生理功能和代谢紊乱的临床综合征。

In 1/8 patients HFJ was ineffectie; the patient (no. 8) was intubated and entilated conentionally.

在机械通气前及通气后每30mins测定PaO2、PaCO2、Ph和氧饱和度以及记录呼吸困难的情况。

The pattern of aerenchyma formation could originate by the combination of cortical cell autolysis and enlarged intercellular space. The process occurred in some stages: cortical cells stopped expansion and the intercellular space became larger at the initial stage. Cortical cells to collapse and autolysis were found with the disintegration of cell contents. After the evacuation of cell contents, cortical cells became shrinking and invaginated.

形成过程大体分为:皮层细胞生长停止,细胞间隙增大;皮层细胞衰老,并发生自溶,内含物解体;解体物逐渐撤走,皮层细胞开始收缩、内陷,当细胞内含物全部撤走,相邻细胞的残留细胞壁叠合即在两侧形成通气空腔;径向上相邻的皮层细胞逐步发生凹陷和细胞壁叠合,结合方形的细胞间隙一起构成通气组织。

The results and the correlations are of particular relevance to the design and operation of hot sparged three-phase reactors.

本文结果及通气功率、气含率关联式对于工业热态通气三相搅拌反应器设计和操作具有一定的参考价值。

Multiphase reaction engineering (530.2440);RPD;gas holdup;hot sparged;gas-liquid-solid three- phase system;stirred tank

多相流反应工程(530.2440);通气功率;气含率;热态通气;气-液-固三相体系;搅拌槽

In conscious, mechanically ventilated patients, sleep architecture was highly abnormal, with a short REM stage and a high degree of fragmentation. The ventilatory mode did not influence sleep pattern, arousals, awakenings, and ineffective efforts.

对于清醒的机械通气患者来说,睡眠结构严重异常,快眼动相睡眠较短,而片断睡眠较多,不同的机械通气模式对睡眠模式、微觉醒、觉醒和无效呼吸的影响没有显著差异性。

Obstructive sleep apnea hypopnea syndrome is a clinic syndrome charactered with repeated apnea hypopnea,intermittent hypoxia,frequent arousals during sleeping at nightwhether companied with hypercapnia or not.

阻塞性睡眠呼吸暂停低通气综合征(obstructive sleep apnea hypopnea syndrome,OSAHS)以睡眠时反复呼吸暂停、间歇性低氧、睡眠结构紊乱和反复微觉醒为特征,伴或不伴低通气的临床综合征。

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