过氧化反应

objectiveto study the effects of alcoholic extracts of averrhoa carambola l. root on blood glucose level and lipid peroxidation in stz-induced diabetic mice.methodsdiabetic model were induced by once intraperitoneal injection of stz.

目的研究阳桃根醇提物对链脲佐菌素(streptozocin， stz)致糖尿病模型小鼠的血糖及抗脂质过氧化反应的影响。

Activity of liver sod in high-dose of alcoholic extracts of averrhoa carambola l.root group significantly increased（p＜0.05）, while concentration of mda in liver decreased markedly（p＜0.01）.conclusionthe alcoholic extracts of averrhoa carambola l.root could relieve the reaction of lipid peroxide and has significant hypoglycemic effect in stz-induced diabetic mice. it is a potential hypoglycemic agent of treatment diabetes and its complication.

结论阳桃根醇提物能够减轻stz诱导的糖尿病小鼠脂质过氧化反应，降低血糖，是一种具潜在开发意义的治疗糖尿病及其并发症的药物。

Betulin has obviously protective effects on alcoholic liver injury in rats.The mechanism may be the induced decrease of lipid peroxide.

白桦脂醇能抑制酒精诱导脂质过氧化反应对肝组织的损伤，对酒精性肝损伤有明显的保护作用。

The staining of F-actin in the ischemia reperfusion group was decreased significantly （P.05） compared with the normal control group. There was a marked loss of canalicular microvilli and distension of canalicular lumen in the ischemia reperfusion group. The staining of F-actin in the LA group was significantly increased than those in the I/R group （P.05）. Conclusions: Ischemia reperfusion induces an disruption of bile canalicular F-actin microfilaments and a loss of microvilli. Alpha-lipoic acid can protect bile canalicular F-actin microfilaments against IRI.

肝脏I/R可造成胆小管F-actin微丝破坏、微绒毛丧失，导致胆小管收缩减弱，胆汁排泄功能受损，这可能是大鼠肝脏I/ R后缺血型胆道病变发生的主要机制；α-硫辛酸通过清除氧自由基，降低脂质过氧化反应，调节细胞内氧化剂-抗氧化剂的含量，保护胆小管F-actin微丝结构免遭缺血再灌注损伤破坏，进而预防和治疗缺血型胆道病变。

Results Expression of CYPⅡE1 in rat nonalcoholic steatosis by high fat diet wasprominent in hepatic acinar zone...

结论非酒精性脂肪肝肝细胞色素P450ⅡE1在腺泡3区的表达增强，并呈弥散性的分布，与酒精性脂肪肝的表达相似，并可能与脂质过氧化反应有关。

Its mechanism may be related to its effects of improving SOD activity, decreasing l ipid peroxidation, and promoting angiogenesis of skin flaps.

围手术期大鼠腹腔注射蜕皮甾酮能促进超长宽比例随意皮瓣成活，其机制可能与其提高SOD 活力、降低脂质过氧化反应、促进皮瓣新血管形成有关。

Objective: To study the effects of exam stress on lipid peroxidation in undergraduates.

目的 ：探讨考试应激对大学生脂质过氧化反应的影响。

The MDA content and SOD activity play an important role in the process of hepatocyte apoptosis and lipid peroxidation.

MDA含量和SOD活力在酒精性肝病的肝细胞凋亡过程及脂质过氧化反应中发挥重要作用。

Se deficiency also aggravated the tume faction of thyroid gland caused byIdeficiency.

缺Se缺I加重了心肌脂质过氧化反应，缺Se缺I造成了心肌的结构损伤作用。

Experimental Center of National Level One, Hubei Traditional Chinese Medica l College, Wuhan 430061）Abstract To investigate the protective effects of Jiaweisinisa n on acute liver injury induced by intraperitoneal injection of D-ga lac-tosamise, serum superoxide dismutase, malondialdehyde , glutathioe contents were measured. The pathological changes of liver wer e observed simultaneously. The results showed that JWSNS could decrease serum MD A content, but increase serum SOD and GSH activities in acute liver injury induc ed by D-GalN. It was also found that JWSNS could alleviate the degeneration and necrosis of the hypatocytes.

为研讨加味四逆散防治肝损伤的作用机理，通过动物试验，先用加味四逆散预防给药，再采用D-氨基半乳糖造成大鼠急性肝损伤模型，然后检测大鼠血清超氧化物岐化酶、丙二醛、还原型谷胱甘肽水平，同步观察该方对急性肝损伤大鼠体内脂质过氧化反应及肝脏病理改变的影响，结果表明该方可明显升高大鼠血清SOD、GSH水平，降低MDA水平，肝脏病理学检查亦表明该方使肝细胞变性坏死程度减轻。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。