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轴突

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Immunohistochemically, the new crescent yellowish endings and fibers of trigeminal axon were SP and CGRP radioactive with granular, pearl and reticular delineations, mainly located at the 1rt and 2nd layers of lateral nucleus caudalis spinalis nervi trigemini.

免疫组化结果可见:P物质和降钙素基因相关肽的免疫阳性结构为轴突终末和纤维,呈现颗粒状、串珠状或交织成网状,主要集中分布在三叉神经脊束核尾侧亚核Ⅰ、Ⅱ层内,呈现新月形棕黄色显色。

In the suboesophageal ganglion, 5-HT/TPH-likeimmunoreactive neurons (ca.15) mainly distributed in the lateral and ventro-lateral area, withprocesses projecting into the suboesophageal ganglion neuropil.

咽下神经节处5-HT/TPH阳性神经元数目相对较多,每侧大约有15个,主要分布于髓区侧细胞体层和腹侧细胞体层,轴突投射进入咽下神经节。

The amount of neuron at 2st week for EG and CC were 36.50±1.78/EF and 19.70±3.71/EF respectively, with the percentage of (43.60±4.83)% and (39.04±3.87)% for EG and CG, which had significant difference (P.05) Conclusion: Self-assembled IKVAV peptide nanofibers, which can degrade the neuron mortality as well as promote neurite sprouting and growth, is a bioactivity scaffold, it can serve as a superordinary nerve tissue engineering scaffold.

自组装IKVAV多肽纳米支架能降低神经元的死亡率,并诱导轴突的发生和生长,具有支架及生物活性双重作用,可作为神经组织工程支架材料。

After embeding routingly,serial 10μm sections were prepared for modified trichromic staining with improve-ments to manifest axons and myelin sheaths simultaneously,immunohistochemistryand in situ hybridization.

组化染色用新的改良三色法同时显示神经轴突和神经髓鞘。

The abnormality of neurofilaments plays a possible role in axonal degeneration of Alzheimer''s Disease,but the mechanism remains unclarified.

阿尔茨海默病患者脑中神经细丝异常可能在其轴突退化中起重要作用,但机制不明。

The mice were ob served afe r poisoning the axons, found the typical Wallerian degeneration.

在电镜下观察小鼠中毒后的神经轴突,发现典型的 Wallerian 变性。

At present, SC could be obtained and cultured from infancy peripheral nerve or adult Wallerian degenerated nerve.

目前多从幼年动物周围神经或成年动物瓦勒变性神经获取、培养 SC,并与聚乙醇酸、聚乳酸等材料粘附、复合,预构成含 SC的人工神经导管,引导轴突再生。

BACKGROUND:Ola rats is a kind of rats with genovariation, who displays Wallerian degeneration after peripheral neuroaxonal damage that is slower than that normal 6J rats, thereby additional damage factor may help fully understand the property of Ola rats.

背景:Ola鼠是一种基因变异鼠,其周围神经轴突损伤后华勒氏变性速度比正常的6J鼠缓慢,增加损伤因素可能有助于了解Ola鼠的特性。

Results: Dynamic observation with histopathology and immunohistochemical staining showed that, after the nerve root nerve root compression, the myelinated nerve fibers decreased and there was ultrastructure changes of the nerve fibers, such as Schwann cell cytoplasm swelling and edema, Wallerian degeneration, and demyelinated degeneration of axons.

结果:通过对压迫神经根及神经根节大体观,组织病理学和免疫组织化学的光学显微镜的动态观察,可见神经根压迫后其有随神经纤维数目减少和超微结构的改变,如神经模细胞胞质肿胀和细胞水肿、华勒变性、神经轴突脱髓鞘变等。

Only 1 single 5-HT/TPH-like immunoreactive interneuron was localized near the dorsal antennal lobe, contributing to most immunoreactivity in the antennal lobe.

咽下神经节处5-HT/TPH阳性神经元数目相对较多,每侧大约有15个,主要分布于髓区侧细胞体层和腹侧细胞体层,轴突投射进入咽下神经节。

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