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豆状核

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Results: In the 13 cases, the CT diagnosis show that five cases of low-density lesions of Bilateral hepatolenticular, five cases of globus pallidus, one case of Bilateral hepatolenticular with corpus callosum, one cases of External capsule putamen, one case of Putamen with the caudate nucleus, three cases of low-density of Bilateral lobi frontalis and lobi parietalis, one case of unilateral putamen hemorrhage, three cases of cerebral atrophy.

结果 13例中,CT示双侧豆状核低密度5例、苍白球5例、双侧豆状核伴胼胝体1例、壳核伴外囊1例、壳核伴尾状核1例;双额顶叶低密度3例、单侧壳核出血1例、大脑萎缩3例。

At 6h after ischemia both MRI were found to indicate definite infarct foci in the caudate nucleus and the lentiform nucleus,mass effect was seen too.Histopathologic findings:At 2h after ischemia,electro microscope revealed the changes of ischemia and edema.At 4h after ischemia,the photomicroscope demonstrated the changes of ischemia edema and the electromicroscope indicated the damage to blood brain barrier.

结果 梗塞2h MRI T2加权可见尾状核头部、豆状核信号增高;6h MRI可见尾状核、豆状核形成明确梗塞,并出现占位征象。2h电镜下已有缺血水肿改变,4h光镜下出现脑缺血水肿改变,电镜下见到血脑屏障受损。

Objective To discuss the VEEG changes in hepatolenticular degeneration patients.Methods VEEG was performed in 28 patients with hepatolenticular degeneration and 20 control subjects.

目的 探讨肝豆状核变性患者视频脑电图改变方法对28例肝豆状核变性患者及20例健康对照组视频脑电图监护检查。

In the original studies,[4,5] all the acute stroke patients with angiographically proven internal carotid artery or M1 occlusion had ALN, presumably because the lentiform nucleus is fed by lenticulostriate arteries from M1 without collateral flow from cortical anastomoses.

先前的研究报道:所有经脑血管造影证实存在颈内动脉闭塞或大脑中动脉M1段闭塞的急性卒中患者,都会出现豆状核低信号征;可能系豆状核是由大脑中动脉M1段发出的豆纹动脉供血,缺乏来源于皮层吻合支的侧枝循环供血所致。

On the other hand, if the medial lenticulostriate arteries escape from ischemia, the medial part of the lentiform nucleus may be spared ischemic injury, and early CT signs may be restricted to the lateral or posterior part of the lentiform nucleus.

另一方面,如果内侧豆纹动脉群(medial lenticulostriate arteries)没有受累发生缺血,那么豆状核内侧区可能就不会发生缺血损伤,早期CT征象可能就仅限于豆状核的后部或外侧部

The 8 cases of 12 patients examined by cranial CT showed abnormal low density lesion in basal ganglia,lentiform nucleus,and so on.The 5 cases of 8 patients examined by cranial MRI showed abnormal signs in basal ganglia,lentiform nucleus,and so on.

12例患者行头颅CT检查,8例发现基底节,豆状核等异常低密度灶;8例行头颅MRI检查,5例显示基底节,豆状核等处异常信号。

The results of in situ hybridization and fluorescence quantitative PCR showed that the gene expression of dopamine receptors(including D1 and D2) in the prefrontal cortex and DRD1 in lentiform nucleus of SHR from the Kongsheng Zhenzhong Pill group is higher than that in the methylphenidate hydrochloride group and both groups are lower than that in the control group:the gene expression of dopamine receptors D2 in the lentiform nucleus from the methylphenidate hydrochloride group is higher than that in the Kongsheng Zhenzhong Pill group while both groups are lower than that from the control group.

原位杂交和荧光定量PCR表明,SHR大鼠前额叶皮质的多巴胺受体(包括D1和D2)和豆状核受体D1的基因表达,孔圣枕中丹组比盐酸哌甲酯组要高,且两者均低于对照组;豆状核多巴胺受体D2的基因表达,盐酸哌甲酯组要比孔圣枕中丹组高,且均低于对照组。

Results:Sensitivity of dense artery sign,lentiform nucleus sign,insular ribbon sign,space occupying effect,hypodensity lesion and cortical sign were 20.8%,23.6%,8.8%,11.6%,56.8%and 14.8%respectivelyP<0.05 as compared with normal control group.

结果:正常组:致密动脉征占11%,豆状核征占5%,脑岛带征占1%,其它征象均为阴性;病变组:致密动脉征占20.8%,豆状核征占23.6%,脑岛带征占8.8%,低密度灶占56.8%,占位征占11.6%,皮质征占14.8%(与正常组相比P均<0.05)。

Methods:102 whole brains were serially transected with each section 2mm in thickness.82 red nuclei,substantia nigra and subthalamic nuclei,110 amydaloid nuclei,122 lenticular nuclei and thalami,and 114 caputs nuclei naudati were located in three dimension.

对102个整脑作三维切面的2mm厚连续切片,分别对82个红核、黑质、丘脑底核,110个杏仁核、122个豆状核、丘脑和114个尾状核头等脑内主要神经核团进行定位研究。

Results: In the 13 cases, the CT diagnosis show that five cases of low-density lesions of Bilateral hepatolenticular, five cases of globus pallidus, one case of Bilateral hepatolenticular with corpus callosum, one cases of External capsule putamen, one case of Putamen with the caudate nucleus, three cases of low-density of Bilateral lobi frontalis and lobi parietalis, one case of unilateral putamen hemorrhage, three cases of cerebral atrophy.

目的 探讨双侧基底节区对称性低密度病变的CT诊断价值。方法回顾性分析肝豆状核变性5例、中毒性病变5例、Wernicke脑病2例、Huntington氏大舞蹈病1例的CT平扫表现及临床意义。结果 13例中,CT示双侧豆状核低密度5例、苍白球5例、双侧豆状核伴胼胝体1例、壳核伴外囊1例、壳核伴尾状核1例;双额顶叶低密度3例、单侧壳核出血1例、大脑萎缩3例。

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