诱导性

Methods: Twenty-four male SD rats were randomly divided into normal control (NC, n=6), protein C activator (PCA, n=6), lipopolysaccharide (LPS, n=6) and LPS+PCA group (n=6). NC group were given normal saline and PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. LPS group were administered lipopolysaccharide (10 mg/kg, 4h) by intraperitoneal injection method to make the model of septic shock rats. After the animal model was made, LPS+PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. 30 minutes after the injection, each rat was tested by Medlab biosignal analyzing system for observing mean arterial pressure and left ventricle function, the activity of LDH and iNOS in myocardium and the cuttings of myocardial tissues.

取SD雄性大鼠24只随机分成对照组、PCA组、LPS组和LPS+PCA组四组，对照组尾静脉注射生理盐水，PCA组尾静脉注射蝮蛇毒蛋白C激活物，LPS组腹腔注射LPS (10 mg/kg， 4h)的方法建立败血症休克的实验动物模型，在建立败血症的实验动物模型的基础上，尾静脉注射PCA (0.4 mg/kg)为LPS+PCA组，每只大鼠均于药后30 min时运用Medlab生物信号采集处理系统记录平均动脉压和左心室功能指标，并测定心肌中乳酸脱氢酶活性、诱导性一氧化氮合酶活性及心肌病理切片。

Elegans were significantly decreased after exposure to ENIs（p.05）; also the brood size and the lifespan of C. elegans were significantly reduced after exposure to ENIs（p.05）. Moreover, after exposure to ENIs, the lifespan of mev-1 mutant hermaphrodites was shorter than that of wild type（p.05）, indicating that mev-1 gene mutant could result in more sensitivity of the mutant hermaphrodites to ENIs exposure than the wild type.

结果表明：空气负离子暴露可显著加速野生型和mev-1突变型线虫发育（p.05），缩短寿命（p.05），与野生型相比，空气负离子暴露对mev-1突变线虫寿命影响更大（p.05）；空气负离子暴露可显著降低线虫后代数量（p.05），原因可能在于空气负离子暴露显著诱导性腺细胞凋亡的增加（p.05）。

Behavioral stress was evoked by 10 foot shocks (1sec, 0.8 mA, 2-3 min inter-shock intervals) in a Skinner box.

电生理实验是在脑冠状面切取的400微米厚脑片上对海马CA1区LTP和LTD的诱导性进行测定。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。