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记忆增强

与 记忆增强 相关的网络例句 [注:此内容来源于网络,仅供参考]

A tapered incident wave is used to reduce the truncated error. The angular correlation function is calculated by Monte-Carlo generated rough surface, the enhancement on nonmemory line of the ACF can be used to detection a target placed over or under the rough surface.

在散射目标存在时,目标与粗糙面的复合多次散射场的角相关函数,在不满足记忆线条件下也有普遍较高的角相关函数值,即在非记忆线上的增强可以用于检测粗糙面上方、下方的散射目标。

Its main role, however, should not be as a reinforcer of memories, but as a tool to establish or re-establish memories in cases of poor recall.

它的主要作用,但是,不应该被视为一个记忆增强剂的,但作为一种工具来建立或重新建立召回记忆差案件。

With the deficit in the information processing on facial emotion perception of schizophrenic patients, the neurocognitive training program is based mainly on the Memory Processing Model, Treisman's Attenuation Model, signs of the eye movement, Ellis's Information Processing Model of Face Recognition, Zhuo's Morphological-Tuning Model, Wang's Two-Stage Face Recognition Model, and Anderson's Adaptive Control of Thought. The contents of the training contain self-instruction, feature abstraction, mnemonics, and positive reinforcement.

本研究以记忆历程模式为主轴,参考减弱模式的感官登录与减弱控制机转、眼球凝视现象、脸部辨识讯息历程模式、双阶段脸孔辨识模式的型态侦测模块、二阶段脸孔辨识模式的脸孔与表情表征、思考适应控制模式的陈述与程序知识等部分,并举证精神分裂病患在表情讯息处理历程各阶段-注意、编码、储存、提取的缺损现象,以自我引导、特征抽取、记忆增强、立即回馈等原则,设计神经认知行为取向的脸部表情辨认训练方案。

The mechanism may be related to reducing brain cholinesterase activity, inhibiting the accumulation of aluminium ions in brain, maintaining the normal level of Glu/GABA.Key words:Prepared Radix Rehmanniae; Learning and memorizing; Dysmnesia

结论熟地黄对于化学药品所致记忆障碍模型小鼠具有增强其学习记忆和空间记忆能力的作用,其作用机制可能与改善中枢胆碱能神经系统功能,降低脑内Al3 含量,保护脑组织及维持脑内Glu/GABA的正常水平有关。

The hippocampus is the most important encephalic region relative to function of learning and memory.

海马是学习和记忆的关键脑区,海马长时程增强(long-term potentiation,LTP)是NMDA受体依赖性突触传递效能的持续性增强,是脑学习和记忆功能在突触水平的研究模型和细胞基础。

Maher hit upon the beneficial effects of fisetin when she screened a collection of flavonoids, substances with anti-oxidant activities found in many plants, for their neuroprotective abilities in tissue culture models of neurodegenerative disease.

通过实验观察到,非瑟酮对健康实验鼠的神经细胞的生长,增强记忆有保护和促进作用。Maher说:&这是中央神经系统分子水平的自然物质的作用首次被发现,而且表明能增强LTP和长期记忆。&

The serum levels of TChE and SOD and CAT in hippocampus of mice in Cold group was also significantly higher than those in the Control group and Warm group(P 0.05或P 0.01),while the serum level of MDA in the Control group and Warm group were higher than that in the Cold group(P 0.05或P 0.01). Conclusion:(1)Long proper winter swimming could improve learning and memory abilities of mice.(2) Winter swimming could improve the ability of learning and memory by coordinate the content of acetylcholinesterase (3) This effect may be related to the decreased activity of TChE and the increased activity of anti-oxidatio.

1长期适量的冬泳运动可以促进小鼠学习记忆能力。2冬泳运动可以通过增加小鼠脑内海马组织中ACh的含量而影响TChE的活力。3冬泳运动促进学习记忆的机制可能是:3.1冬泳运动通过冷应激引起的神经内分泌系统的反应,来改变中枢神经系统的功能,使小鼠脑内乙酰胆碱的代谢增强、含量增加,从而促进学习记忆能力。3.2冬泳运动通过增强机体清除自由基的能力,来减轻脂质过氧化的程度,对小鼠的脑损伤起到保护作用,从而促进学习记忆能力。

These results suggest that morphine withdrawal selectively impairs the cued fear extinction through inhibiting the ERK phosphorylation in the amygdale and mPFC. Histamine deficiency not only enhances acquisition the cued and contextual fear conditioning and but also potentiates the impairment of extinction of cued fear memory.

这些结果提示:吗啡戒断能选择性抑制恐惧记忆的消除,此作用是通过抑制杏仁核和mPFC脑区内ERK蛋白磷酸化来实现的;组胺缺乏能增强线索和背景恐惧记忆的获得以及增强吗啡戒断对线索恐惧记忆消除的抑制作用。

Histamine deficiency markedly potentiated the acquisition of learning in cued and contextual fear conditioning in the HDCKO mice,but had no effect on cued and contextual fear extinction.However,histamine deficiency potentiated the inhibitory role of morphine withdrawal in cued fear extinction.Consistent with the behavioral results,morphine withdrawal decreased ERK phosphorylation in the amygdala and mPFC but not in the hippocampus of WT and HDCKO mice,and ERK might not be phosphorylad in the amygdale and mPFC of HDCKO mice after the 4th training of cued fear extinction.

但HDCKO小鼠增强了吗啡戒断对线索恐惧记忆消除的抑制作用,与行为学实验结果相一致是,,吗啡戒断能明显降低WT和HDCKO小鼠在线索恐惧记忆消除第4次训练后杏仁核和mPFC脑区的ERK磷酸化水平,但对海马脑区的ERK磷酸化没有作用;HDCKO小鼠在线索恐惧记忆消除训练的第4次训练后杏仁核、mPFC脑区的ERK蛋白没有明显磷酸化激活。

Effects of chronic multiple stress on learning and memory and the expression of Fyn,BDNF,TrkB in the hippocampus of rats[J].

慢性复合应激性学习记忆增强大鼠海马神经细胞增殖和突触后酪氨酸激酶表达的变化[J]。

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