被抑制的

The results indicated that the content of cytosolic CaM in cells treated with exogenous Ca2+ has increased indistinctively, while fluorescence intensity in cells treated with TFP decreased. So we believed that exogenous Ca2+ has little effect on the expression of CaM. High concentration of TFP can enter yeast cells and combine to CaM to make it inactive, which is the reason that TFP restrain the growth of yeast cells. 5mmol/L EGTA could completely arrest the cell proliferation of MFP7 after 28h, when the fluorescence intensity in cells wasobviously increased with flow cytometry and LSCM.

被较高浓度TFP处理过的MFP7胞内荧光强度则相对较弱，推测是因为TFP进入胞内与CaM结合从而使其失活，这也是高浓度TFP抑制酵母细胞增殖的原因。5mmol／L EGTA处理28h左右后，酵母细胞的生长被抑制在G1晚期，此时通过细胞流式法和在激光扫描共聚焦显微镜下观察到胞内荧光强度显著高于对照，说明CaM的表达水平在G1后期开始上升；回加10mmol／L Ca~(2+)处理24h后，细胞开始恢复生长，细胞流式法测定胞内荧光强度有所下降，表明多数细胞完成G1／S转换，进入生长对数期。

The inward current in OHCs was totally inhibited after the superfusion of 100μmolIL and 200μmo1/L linopirdine respectively. The outward rectifier K(superscript +) current I(subscript K was the dominant K(superscript +) current in the whole cell currents in Deiters' cells. In the presence of 200μmol/L linopirdine, the current was not significantly affected.

在细胞外液中，加入100μmol/L利诺吡啶后，OHCs中的四乙基二乙胺敏感的钾电流峰电流成分被抑制，稳态电流幅值减小，且电流的失活时间常数明显延长；在细胞外液中加入100μmol/L利诺吡吮后，OHCs的内向性钾电流I被完全抑制；而细胞外液中利诺吡啶终浓度为200μmol/L时，Deiters 细胞的外向整流性钾电流幅值无明显变化。

This subset is hyporesponsive and anergic to antigen stimulation. Recently, it has been demonstrated to participate in the immune deficiency in human immunodeficiency virus infection and has close relationship with the development of acquired immunodeficiency syndrome. Meanwhile, the CD4(superscript +)D25(superscript +)Treg is also infected when the human body is invaded by HIV. The CD4(superscript +)CD25(superscript +)Treg will finally be killed by the mechanism of antibody-dependent cell-mediated cytotoxicity. Because of the decrease of the CD4(superscript +)CD25(superscript +)Treg, it can't function as a immune suppressor as a result of the excess of activation of HIV and gradually exhausting of the T cell, which shows that the CD4(superscript +)CD25(superscript +)Treg plays a inhibitory role in the different stages of the development of the HIV as well as different effects in the pathogenesis of AIDS.

它能够抑制自身免疫病的发生和发展，参与肿瘤免疫的调节，同时在感染和移植免疫中也发挥着极其重要的作用。T细胞的这一亚群具有免疫调节和免疫抑制的特性，新近发现它亦与爱滋病的发生、发展关系密切HIV进入人体后，CD4D25调节性T细胞抑制了机体的免疫效应但它也同时被感染，最终由于细胞毒的作用而死亡由于调节性T细胞数量的减少不能有效的发挥其抑制作用，HIV持续的过度活化使得T细胞逐渐耗竭说明在HIV发生、发展的不同阶段Treg细胞可能都发挥了免疫抑制作用，但是却对HIV感染与爱滋病发病的进程产生了不同的效应。

Dipole source analysis found cingulate may be involved in executive inhibition processing.

这说明与低焦虑被试相比，高焦虑被试对正性图片采取了和负性图片相同的抑制策略，存在着过度抑制的倾向。

The effect of daphnetin and daphnetin-Fe complex on COX and RNR activity of P.falciparum was tested by ultraviolet spectrophotometer and electron spin resonance respectively.

结果 体外同步培养的恶性疟原虫经瑞香素（100μmol／L）作用2、4、8和12h后，COX活性分别被抑制了0、6％、73％和80％；在瑞香素浓度为0.1、1、100和1000μmol／L，作用6h后，COX活性分别被抑制3％、31％、53％和84％；而瑞香素-Fe复合物对COX的影响几乎消失。

The results are as follows:(1) Long train electrical stimulation at the vMNF inhibitited the myoelectric activities of genioglossus markedly.

结果如下：(1)长串电脉冲刺激vMNF导致颏舌肌肌电活动明显被抑制；(2)在vMNF内微量注射谷氨酸钠，颏舌肌肌电活动出现明显的抑制效应；(3)单脉冲电刺激vMNF引起颏舌肌抑制反应的潜伏期为(20.6±0.4)ms。

The outward rectifier K+ current current was the dominant K+ current in the whole cell currents in Deiters' cells. In the presence of 200μ M linopirdine, the Ik current was not significantly effected. Our findings demonstrated that the KCNQ heteromeric or homomeric potassium channel may be the molecular basis for the peak outward K+ current and testified that the inward IKn current is mediated by KCNQ potassium channel.

在细胞外液中，加入100 μmol/L利诺吡啶后，OHCs中的四乙基二乙胺敏感的钾电流峰电流成分被抑制，稳态电流幅值减小，且电流的失活时间常数明显延长；在细胞外液中加入100 μmol/L和200 μmol/L利诺吡啶后，OHCs的内向性钾电流IKn被完全抑制；而细胞外液中利诺吡啶终浓度为200 μmol/L 时，Deiters细胞的外向整流性钾电流幅值无明显变化。

No considerable effect was observed on the COX activity of P.falciparum treated with daphnetin-Fe complex. The tyrosyl free radical was tested to reflect the RNR activity of P.falciparum at various times by ESR.

结果 体外同步培养的恶性疟原虫经瑞香素（100 μmol/L）作用2、4、8和12 h后，COX活性分别被抑制了0、6%、73%和80%；在瑞香素浓度为0.1、1、100和1 000 μmol/L，作用6 h后，COX活性分别被抑制3%、31%、53%和84%；而瑞香素-Fe复合物对COX的影响几乎消失。

Onthe other hand, with increased isoflurane or halothane concentration,responses of ventral horn neurons were depressed by 60% and45%, respectively.

另一方面，随着异氟醚或氟烷浓度的提高，腹侧角神经元的反应为从被抑制60％变为了被抑制45％。

Greater than -10 mV and shows little inactivation during a 200 ms depolarization. It is completely reprimed at a holding potential of -60 mV, and can be isolated by using a more depolarized h.p.(-40 mV) to inactivate the other two types of calcium currents. 3. Component T can be seen in isolation with weak test pulses.

在测试电位大于-10mV时，L电流被产生并不会在200ms除极化的过程中被抑制。L电流在-60mV时被可被重新发起，并可在-40mV时以抑止其它两种钙离子电流被隔离。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。