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DATA SYNTHESIS: With the increase of ultraviolet radiation caused by environmental pollution, DNA in human epidermal cells is damaged. Cyclobutane pyrimidine dimmer and 6-4 photoproducts are the main forms of DNA lesions.

资料综合:环境污染使得紫外辐射增强,而紫外线辐射对机体表皮细胞的DNA具有损害作用,可导致环丁烷嘧啶二聚体和6-4光产物的形成。

Ozone College depletion in stratosphere has led to the increase of solar UV-B radiation reaching to the earth surface, which would damage DNA of plant epidermal cells and produce cyclobutane pyrimidine dimmers (CPD and 6-4 photoproducts.

臭氧衰减导致地表紫外辐射增强,而紫外线辐射对植物表皮细胞的DNA具有损害作用,可导致环丁烷嘧啶二聚体和6,4-光产物的形成。

Objective To choose a better matter interface for the models of UVA induced human epiderm keratinocytes irradiation damage.

目的 选择一种建立长波紫外线(ultrayiolet,UVA)照射人表皮角质形成细胞损伤模型的理想载物界面。

The skin erythema and the rough level were observed during the experiment; the appearance and thickness of epiderm and the number of fibroblast were observed under light microscope.

实验观察皮肤红斑形成、粗糙程度等外观变化及HE常规染色后光镜下表皮厚度和真皮中成纤维细胞数量的变化。

Objective To choose a better matter interface for the models of UVA induced human epiderm keratinocytes irradiation damage.

作者:马林琳张玲罗敏邓杨林李玛琳目的选择一种建立长波紫外线(ultrayiolet,UVA)照射人表皮角质形成细胞损伤模型的理想载物界面

Methods To irradiate the human epiderm keratinocytes with an identical dose of UVA,which is primary cultured and is occupying exponential growth phase.

目的 选择一种建立长波紫外线(ultrayiolet,UVA)照射人表皮角质形成细胞损伤模型的理想载物界面。

Objective To choose a better matter interface for the models of UVA induced human epiderm keratinocytes irradiation damage.Methods To irradiate the human epiderm keratinocytes with an identical dose of UVA,which is primary cultured and is occupying exponential growth phase.

作者:马林琳张玲罗敏邓杨林李玛琳目地选择一种建立长波紫外线(ultrayiolet,UVA)照射人表皮角质形成细胞损伤模型地理想载物界面。

Result: The average healing time of superficial second degree burn wound was 7 days with epidermidalization; deep second degree burn wound achieved physiological healing in 22 days; while superficial third burn wound were healed in 35±days. The total cure rate was up to 98.92%, and scarring rate accounted for about 12%.

结果:浅Ⅱ度创面治愈时间,平均7d全部表皮化愈合,深Ⅱ度创面平均22d也生理性愈合,浅Ⅲ度创面在35d±5d愈合;总治愈率达98.92%,瘢痕形成率约占12%。

OBJECTIVE: To explore effects of epithelial growth factor on fibroblastic colony forming units from the non-adherent BMSCs.

目的:探讨表皮生长因子对骨髓中存在的非黏附骨髓间充质干细胞产生成纤维细胞集落形成单位的影响。

The proliferation and kinetics, cell cycle, clonalforming rate, and apoptosis of MEC- 1 cells, before and after FHIT gene transfection in vitro, and tumor loci formedon mice after injection of transferred MEC- 1 cells in vivo were observed by immunochemical staining, flow cytometryanalysis, and so on.

结果外源性FHIT基因转入黏液表皮样癌MEC- 1细胞后,S期细胞显著减少,G1期细胞增多;细胞倍增时间从(21.03±0.41)h延长为(26.86±0.71)h;软琼脂克隆形成率显著降低;细胞发生退行性改变和出现凋亡早期表现。

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