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Blood-brain barrier in tumors and peritumoral oedematous regions had the same variations in ultrastructure.

瘤体和瘤周水肿区血脑屏障超微结构变化无明显差异,内皮肿胀,毛细血管腔狭小,胶面有稀疏的绒毛样突起,内皮为无孔型,胞质内胞饮泡较多,内皮紧密连接增长,细胞连接间隙增宽,基膜完整,胶质膜缺损。

The mice in experimental group and control group were exposed to 350 cGy radiation produced by 60Co. After 3 h, karyocytes at different concentrations in the fresh human umbilical cord blood were injected into the mice in experimental group A, B, C via their tail veins, and the equal volume of normal sodium was also injected into control group via tail veins. After one month, carbon tetrachloride (CCl4) was injected into experimental group A, B and control group via abdominal cavity, and the equal volume of normal sodium was injected into experimental group C. After two months, immunohistochemistry and reverse transcriptase polymerase chain reaction were used to detect the expressions of human cytokeratin-18 (CK18), cytokeratin-19 (CK19) and albumin in liver tissues of all mice.

采用60Co治疗仪γ射线对实验组和对照组行350cGy的亚致死剂量照射,实验组A、B、C3组照射后3h内经尾静脉分别输入1.0×10^7个/只、2.0×10^7个/只和3.0×l0^7个/只人新鲜脐血有核细胞,对照组经尾静脉注入等体积无菌生理盐水。1个月后对实验A、B组和对照组裸鼠经腹膜腔注射四氯化碳(CCl4),实验C组注射等体积生理盐水。2个月后采用免疫组化和RT-PCR方法检测裸鼠肝组织人源性CK18、CK19和人源性白蛋白的表达。

The polygelation and crystalloid solution were injected during the operation, and retransfusion self-blood was taken after operation.

术中静脉补充约与失血量等量的多聚明胶和晶体液,待手术关闭髋关节腔时回输自体血。

Or, how does VEC influence these characters and functions of its neighboring TC. These are all unclear. On the other hand, How does TC adhere to and destruct the vascular endothelium to enter and come out of the vascular cavity during the tumor haematogenous metastasis?

反之,位于TC邻近的VEC对TC的生物学特性、功能又如何影响以促进肿瘤血管系统的建立,尚不清楚;另一方面,在肿瘤的血行转移中TC如何粘附、破坏血管内皮以进出血管腔?

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。