血肠

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过"肠—脑轴"传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

The results showed: the calcium, phosphate and ALP in blood in the osteoporosis rats induced by DXM were normal, the bone ash wight, bone dense and intestinal Ca2+ absorption were declined compared with these of the normal rats (p.05). Afterwards, the osteoporosis rats were treated with replenishing kideny herbs or calcium.

实验结果发现，地塞米松诱发的骨质疏松大鼠的血钙、磷、碱性磷酸酶及尿羟脯氨酸均在正常水平，骨密度、骨灰重下降，肠钙吸收减少，与正常大鼠比较有统计学差异（P.05），血中雌二醇、1，25-2-D3水平降低（P.01，P.05），PGE2升高（P.05），经过补肾中药或钙剂治疗后，血雌二醇、1，25-2-D3水平回升而PGE2回降，均接近正常水平。

Anatomical study proved:①.4~8 nutritional vessels of sural nerve –small saphenous vein were the intermuscular perforators of peroneal artery, the furthermost one located at(1.0±1.3)cm above the tip of external malleolus with diameter (0.6±0.2)mm;②.2~6 musculocutaneous perforators were given off by the artery of outer head of gastrocnemius muscle to supply nutrition to the sural nerve-small saphenous vein;③.intermuscular perforators from peroneal artery supplied blood to fibula and outer head of soleus muscle.the perforators with an average outer diameter of （1.3-1.6）cm gave off cutaneous branch ,fascial branch and nutritional vessels to the sural nerve-small saphenous vein,which formed the vessel chain of the sural nerve-small saphenous vein ,sub-fascial and suprafascial vessel net.

结果：1、解剖实验证实：①、腓肠神经-小隐静脉营养血管来自腓动脉肌间隔穿支4～8支，最远的跟外侧动脉穿支距外踝尖上(1.0±1.3)cm，外径(0.6±0.2)mm。②、腓肠肌外侧头动脉沿途发2-6支的肌皮穿支营养腓肠神经-小隐静脉。③、腓动脉肌间隔穿支营养腓骨、比目鱼肌外侧半。上述穿皮支平均外径在0.3～1.6mm，发出皮支、筋膜支、腓肠神经-小隐静脉营养血管，形成腓肠神经-小隐静脉血管链以及深、浅筋膜血管网。

METHODS: The 144 BALB/c male mice were abdominally irradiated with 1150 cGy 60Coγray in 1-3 h, and then they were injected intestinal RNA from normal rats on jejunum. On day 1, 3 and 5 after irradiation,the mice were sacrified after anesthesia for determining endotoxin in blood and bacterial metathetic rate, and intestinal crypt survival rate.

选用BALB/c雄性小鼠144只，用1150 cGy 60Coγ射线进行腹部一次照射，于照后1~3 h采用局部肠腔扩张注入法给小鼠空肠肠腔内注入正常大鼠小肠RNA，分别于照后1，3，5 d麻醉后解剖，取血，测定内毒素含量；取肠系膜淋巴结，测定细菌移位率；取空肠段，计数肠腺存活率。

METHODS: The 144 BALB/c male mice were abdominally irradiated with 1150 cGy 60Coγray in 1-3 h, and then they were injected intestinal RNA from normal rats on jejunum.

选用BALB/c雄性小鼠144只，用1150 cGy 60Coγ射线进行腹部一次照射，于照后1~3 h采用局部肠腔扩张注入法给小鼠空肠肠腔内注入正常大鼠小肠RNA，分别于照后1，3，5 d麻醉后解剖，取血，测定内毒素含量；取肠系膜淋巴结，测定细菌移位率；取空肠段，计数肠腺存活率。

The hemolysin consists three components,B,L1,and L2(35.36.and 45kDa,respeotively),combinations of all three moieties produed the unique ring-shaped zone of hemolysis,increased rabbit vascuolar permeability,caused fluid accumulation in rabbit ileum loop and appeared cellulotoxic reaction on Vero cells.

溶血素由B、L1和L2 3种成份组成，分子量分别为35、36和45kDa 3种成份的协同作用在血琼脂上引起靶状溶血，使兔皮肤血管通透性增加，兔小肠肠段结扎产生积液反应，Vero细胞形态学改变。

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过&肠—脑轴&传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

Piglet edema were first discovered in Britain, is hemolytic E. coli intestinal toxemia, so capillaries or systemic damage to the small blood vessels, increasing permeability and fluid extravasation too much water, to the head, eyelids, Equality ear edema, acute ataxia and death as the main feature of acute infectious diseases, also known as gastrointestinal edema pigs or pig intestinal toxemia E. coli, a highly lethal disease.

仔猪水肿病最早发现于英国，是由溶血性大肠杆菌引起肠毒血症，而使全身毛细血管或小血管受到破坏，通透性增大，水液外渗过多，造成的以头部、眼睑、耳部等处水肿、共济失调和急性死亡为主要特征的急性传染病，又称猪胃肠水肿或猪大肠杆菌肠毒血症，有高度致死性疾病。

P.01 . conclusion:(1) IgA of the lamina propria mucosae would lessoning while the endotoxin in portal veinwas heightening, and the lesson of the I L-6mRNA was the main reason of the lesson of IgA in the lamina propria mucosae, so, the impairment of barrier of the intestinal mucosa was caused by the impairement of the organic immunological function descent while there was obstructive jaundice existing.

(1) 肠粘膜固有层内IgA表达减少伴随门静脉血内毒素浓度的增加，而肠粘膜内IL-6mRNA表达的减少是导致肠粘膜固有层IgA表达减少的重要原因之一，因此可推论肠粘膜免疫屏障功能的受损与梗阻性黄疸时机体的免疫功能受抑有关。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。