血缺氧的

AbstractObjective:to analyze the relationship between normal term newborn and the degree of asphyxiation.

目的：分析足月新生儿高胆红素血症与窒息缺氧程度的关系。

Although the pathologic changes of ICP are similar to those of placental ischemia,these alterations influenced directly the plaˉcental blood and oxygen supply function to result in bad pregnancy.

虽然ICP时胎盘病理改变与胎盘缺血缺氧时的改变相比无特异性，但形态学改变直接影响了胎盘的供血和供氧功能，导致了不良妊娠结局。

Results Compared with that of the control group, the dark adaptation time and the saturation of blood oxygen of trial group were significantly different, i.e.

高压氧干预可增加人体在高原缺氧条件下的血氧饱和度，并提高人体的暗适应能力。

The damage of hepatic kupffer cells function and intestine mucosae protective screen function caused cooperatively by ischemia, hypoxia, microcirculatory disarrangement, inflammatory mediators (O〓, OH〓 etc) and cytokines (TNF-α, IL-1, IFN-γetc), the functional damage easily results in bacterial translocation and endotoxemia, as the damage mitigates the clearance of bacteria and toxin.

缺血、缺氧、微循环障碍、炎症介质及细胞因子（TNF-α、IL-1、IFN-γ等）共同作用，使肝脏Kuppfer细胞和肠粘膜屏障功能受损，对内毒素清除能力减弱致内毒素血症，是肝功能衰竭的重要病生环节，亦是继发多脏器衰竭的始动因素，但ALF致病确切机制目前尚不十分清楚。

Therefore, tumors in general are always exposed to hypoxia and poor nutrition because of insufficient vascularization.

同时，由于血供不足，肿瘤细胞通常处于一种缺氧和营养缺乏的环境中。

In hypoxia/reoxygenation model(Na2S2O4), cell survival was enhanced and LDH in culture medium was reduced(P.05, P.01) after treated with 60, 30μgmL^(-1) Sanguis Draconis flavones. Enhanced cell survival and reduced LDH in culture medium(P.05, P.01) also observed in cytotoxicity model induced by H2O2 after incubated with 60, 30, 15μgmL^(-1) Sanguis Draconis flavones.

在缺氧／复氧(Na2S2O4)模型中，60、30μgmL^(-1)龙血竭总黄酮能显著降低培养液中的LDH浓度，增强细胞活力(P.05， P.01)。60、30、15μgmL^(-1)龙血竭总黄酮能抑制H2O2引起的损伤，降低培养液中LDH的浓度，增强受损细胞活力(P.05， P.01)。

METHOD: The following tests were made:①test with of hemopoietic inhibition induced by cyclophosphamide on mice,②Carban particles clearance test,③Erythrolysin formetien test,④Swimming test,⑤Hypoxia test,⑥Sedation test,⑦Hypnosis test.

①由环磷酸胺引起的小鼠造血系统抑制模型；②小鼠碳粒廓清试验；③小鼠溶血素形成试验；④小鼠游泳试验；⑤小鼠耐缺氧试验；⑥小鼠旷野活动试验；⑦由戊巴比妥钠引起的小鼠催眠模型。

Fant by use of serial CK-BB isoenzyme measurement.

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MethodsFocal cerebral ischemia was produced by permanent occlusion of the proximal of the right middle cerebral artery. The neurologic status of each rat was carefully evaluated at 24 h after surgery. The cerebral infarction numbers of neurons and nissls stains ,the water content were measured. Acute incomplete cerebral ischema was induced by ligaturing bilateral carotid arteries. Anoxia was produced by close normobaric hypoxia.

方法电凝大鼠一侧大脑中动脉造成局灶性脑缺血，观察脑血舒通对大鼠行为、大脑皮质内脑梗塞区神经细胞数量、尼氏小体含量、脑组织含水量的影响；采用结扎小鼠双侧颈总动脉形成急性脑缺血模型，并进行了小鼠常压耐缺氧实验，观察小鼠存活时间。

Sleep apnea hypopnea is one of the most important factors for exacerbating hypoxemia,carbon dioxide retention and acidosis.

睡眠呼吸紊乱是造成OS患者高碳酸血症及缺氧难以纠正的重要原因。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。