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The same is true for hypertension. Here again you look at relaxations to increasing concentrations of acetylcholine.

高血压也一样,在这张片子中诸位可以再一次看到乙酰胆碱浓度增加时血管的舒张反应。

Objective To study the endothelium-dependent dilation function in patients with Takayasu arteritis by color Doppler ultrasound.

目的 利用高分辨力超声技术对大动脉炎患者的血管内皮依赖性舒张功能进行研究和分析。

Methods: Fresh saphena rings were sudied in organ chamber after incubating with Krebs-Henseleit bicarbonate solutions, FWM, KH with 0.1mmol/L NCR, FWM with 0.1mmol/L NCR at 37℃ for 1 hour. Precontraction induced by prostaglandin F2 (U4661930nmol/L) and endothelium-dependent relaxation induced by calcium ionophore A23187 l0^(-10~l0^(-6) mol/L were measured in the presence of indomethacin (7μmol/L) and LNNA (300μmol/L). Result: No significant changes were found in the 4 groups under the light microscope.

采用器官槽法研究在37℃有氧条件下用重碳酸盐缓冲液、含0.1mmol/L NCR的KH液、FWM液、含0.1mmol/L NCR的FWM液中浸泡血管环1h后,再用7μmol/L吲哚美辛和300 μmol/L N-硝基-L-精氨酸作用后,检测30 nmol/L U46619及不同浓度A23187引发的血管收缩舒张反应。

Meanwhile,concentrations of KP and ANP in atrium and aorta of the rats were observed as well.

KP不仅具有和ANP一样的利尿、舒张血管、抑制肾素-血管紧张素系统的作用,还具有抑制心肌Na+-K+-ATP酶的作用〔2〕。

In 66 anaesthetized rats, the effects of local injection of agmatine on femoral, renal, and mesenteric vascular beds were investigated respectively by constant flow perfusion method.

在66只麻醉大鼠,分别采用后肢、肾脏和肠系膜动脉在体恒流灌注法,观察了向灌流环路中直接注射胍丁胺(agmatine, AGM)的血管效应,以所引起的灌流压增减反映血管的收缩和舒张。

Methods There were to patients of diabetes and 35 patients of non-diabetes in this study. The diameter, inter median thickness, peak systolic velocity, end diastolic velocity, pulsatility index, and resistance index of bilateral common femoral artery, superficial femoral artery, profunda femoral artery, popliteal artery,and pedal artery,and whether there was arteriosclerotic plaque or occlusive diseases were examined with color Doppler ultrasonogh.

糖尿病组70例,非糖尿病组35例,分别应用彩色多普勒超声观察双侧股总动脉、股浅动脉、股深动脉、腘动脉、足背动脉的血管内径、内-中膜的厚度、收缩期最大峰值血流速度、舒张期末血流速度、搏动指数、阻力指数;观察血管内有无硬化斑块、有无完全性和不完全性动脉闭塞。

The trophoblast blood flow spectrum could be easily detected with the guide of CDE.The ultrasonic findings were characterized by broad arterial flow spectrum,high diastolic component of the low resistance trophoblastic flow,RI=0.40~0.51 with single or biphasic spectrum.The salpingian ring with high blood flow indicated early and complete salpingian pregnancy.

结果:①异位妊娠包块血流信号丰富,血管连续性好,能较完整显示血管床、血管网及范围广的低流速血流信号;②脉冲多普勒取样为滋养层周围性血流频谱,其特征为:频谱增宽、单相或双相持续存在、阻力降低(RI=0.44±0.04)、舒张期血流丰富的动脉频谱;③具有鲜明丰富的滋养层周围性彩色血流频谱特征的&输卵管环&是早期输卵管妊娠的特征性表现,并提示是输卵管完整存在的标志。

SI can inhibit vasomotion in vaso-dilation status, but its mechanism is not mediated by endogenous NO.

血管运动得以恢复;在SI的有效作用时间内应用L—NMMA则不能使处于舒张状态的血管收缩。

On the other hand, we also analyzed and identified the proteins with changed expression upon the stimulation with carbachol in the cultured human coronary artery endothelial cells.

在10一7mol.L一'5一HT预收缩平衡的内皮完整的脐静脉血管环上, ATPlo一'mol 。L一'可以诱发用的血管的内皮依赖性舒张反应。

objective to observe the protective effect of qiliqiangxin capsule in rabbit heart ventricle reconstitution of chronic heart failure.methods using the method of giving rabbit abdomen aorta half ligations to build the chronic heart failure model to observe qiliqiangxin capsule the effect to blood flow mechanics index,room wall thickness,heart exponent and heart ventricle reconstitution biochemistry index.results qiliqiangxin capsule could reduce effectively bp,lvsp,lvedp,t wave,improve room wall thickness and heart exponent,reduce angii and alt,all the paramenters ahd notable difference,composed to those of model group (p<0.05,p<0.01).conclusion qiliqiangxin capsule can improve the blood flow mechanics change of chronic heart failure rabbit,which has notable inhibitory action on heart ventricle reconstitution which originate from chronic heart failure.inhibition of ang ii,alt of immune nerve endocrine system may be a mechanism of qiliqiangxin capsule.

观察血流动力学、标志心室重构的室壁厚度及心脏指数及肾素―血管紧张素―醛固酮系统相关生化指标的变化。结果芪苈强心胶囊可有效降低血压、左室压、左室末期舒张压、t波,改善室壁厚度和心脏指数,降低血清血管紧张素ii及醛固酮水平,与模型组比较均有显著性差异(p<0.05,p<0.01)。结论芪苈强心胶囊可改善慢性心力衰竭兔血流动力学的变化,对慢性心力衰竭引起的心室重构具有显著抑制作用,其机制可能与该药发挥抑制神经内分泌免疫系统rass中angii、ald的水平有关。

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