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The variation of plasma vasoactive peptide concentration was not only related to UAP attack but also significant to the maintance coronary diastole and corculation.

UAP的发生与血管活性肽的变化密切相关,血管活性肽对维持冠脉舒张和循环有重要意义。

The purpose of this study was to investigate the vasorelaxing effect of vasonatrin peptide on human intramammary artery.The vasorelaxing effect of VNP on HIMA was measured by means of perfusion in vitro. The effects of HS-142-1, TEA, 8-Br-cGMP and methylene blue were also observed.

为了研究血管钠肽对人乳内动脉(human intramammary artery, HIMA)的舒张作用及其机制,采用离体血管灌流的方法,观察VNP对内皮完整和去内皮HIMA的舒张作用,以及HS-142-1、TEA、8-Br-cGMP和镁蓝对这一过程的影响。

Objective Endothelial dysfunction, characterized by impaired endothelium-dependent vasodilatation, is now considered as the common path toward cardiovascular disease.

内皮细胞损伤导致的血管内皮功能障碍目前被认为是诱发许多心脑血管疾病的共同环节,其中血管内皮舒张功能的受损是血管内皮功能障碍的重要特征之一。

The isolated vascular rings perfusion assay was used to examine the effect of EDDF on the vasodilation.

用离体大鼠血管环微量灌流法观察h/p-EDDF对大鼠肠系膜二级阻力血管和脑基底动脉和/或胸主动脉的舒张作用;(4)。

As a new messenger factor,carbon monoxide can activate soluble guanyl cyclas,relax angio-smooth muscle,prevent platelet from aggregation and inhibit angio-smooth muscle cells proliferation,therefore it can maintain normal blood stream in coronary artery.

一氧化碳是一种新的信使因子,通过激活可溶性鸟苷酸环化酶,舒张血管平滑肌,抑制血小板聚集和抑制血管平滑肌细胞增殖,从而恢复冠状动脉的正常血流。

As a new messenger factor,carbon monoxide can activate soluble guanyl cyclas,relax angio-smooth muscle,prevent platelet from aggregation and inhibit angio-smooth muscle cells proliferation,therefore it can maintain normal blood stream in coronary artery.

一氧化碳是一种新的信使因子,通过激活可溶性鸟苷酸环化酶,舒张血管平滑肌,抑制血小板聚集和抑制血管平滑肌细胞增殖,从而恢复冠状动脉的正常血流。血红素氧合酶通过其抗动脉粥样硬化作用对冠状动脉起保护作用。

By using β-NADPH-diaphorase histochemical staining technique,wefound for the first time that NOS activity was detected in the epithelium of airpathway and endothelium of vessels but was not detected in the small vessels(ID<100μm),and the NOS express in pulmonary vascular endothelium of preascitic andascitic broilers were elevated,especially in the endothelium and smooth muscle layer ofsmall vesseles with thicker walls and of muscularized nomuscular artery in asciticbroilers.

本研究从田间自然发生腹水症的肉鸡群中选取正常肉鸡、亚临床腹水鸡和腹水鸡为实验对象,通过比较这三种肉鸡血流动力学、心电图、动静脉血血气、肺外动脉舒张反应性、肺血管结构以及肺组织一氧化氮合酶的活性和分布等方面的不同,探讨了肺动脉舒张能力在肉鸡肺动脉高压形成中的作用和机制。

The vasodilating mechanism of AE from CM ma he related to promoting the release of NO from endothelium of blood vessel and synthesis of NOS and NOS in aortic tissue.

桑白皮丙酮提取物的舒张血管作用机制可能与血管释放NO和促进cNOS、NOS合成有关。

Glyceryl trinitrate induced dilatation: GTN, high sensitie C reactie protein, tHcy, serum folate, red cell folate and lipids.

干预前、后分别检测血管内皮功能、血管平滑肌功能(硝酸甘油介导的血管平滑肌舒张GTN)、高敏C反应蛋白、血浆总高半胱氨酸、血浆红细胞叶酸盐和血脂。

Then the responses of artery to phenylephrine acetylcholine, sodium nitroprusside and norepinephrine were studied.

结果:转基因血管内皮细胞的增殖较对照组活跃,动脉的内皮依赖性舒张反应较强,10 -6mol/L 的乙酰胆碱引起的舒张为33-08%±10-01%,对照组为- 17-57%±5-59%,P< 0-01 。

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