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Objective: To observe the curative effect of the Chinese medicine paved on the umbilicus and enema in the treatment of refractory cirrhosis ascites with azotemia.

目的:观察中药脐敷配合中药灌肠对难治性腹水诱发氮质血症的临床疗效,以及对肾素-血管紧张素-醛固酮、心房利纳肽的含量的影响;阐明中药脐敷配合中药灌肠治疗难治性腹水诱发氮质血症的部分机制。

Using of matrix assisted laser desorption/ionization time of flight mass spectrometry coupled with carboxypeptidase Y digestion for the analysis of C terminal peptide ladder by direct determination of the mixture of truncated peptide, the sequence of C terminus can be read out simplely from the mass differences between adjacent peptide peaks on the mass spectrum.

用羧肽酶Y水解蛋白质和多肽,结合飞行时间质谱测定酶解后缩短肽的质量,根据相邻两肽峰的质量差别读出蛋白质和多肽的C端序列,在pmol水平下测定了人促肾上腺皮质激素片段以及人血管紧张肽片段,其中人促肾上腺皮质激素片段测到了C端 15个氨基酸残基的顺序

After treatment with the above hormones, the rats were established anesthesia with urethan (1g/kg, i. p.); polyethylene catheters were inserted into the external jugular vein and the common carotid artery for drug administration, and then blood pressure was recorded. The arterial catheter was connected to a pressure transducer, and the blood pressure was displayed on the channel of BL-310 Experimental System of Biological Function through IBM computer. We assessed the changes of resting arterial blood pressure, the pressor responses of angiotensin Ⅱ(Ang Ⅱ, 0.01mg/kg), noradrenaline (NA, 0.01mg/kg) and adrenaline (Adr, 0.016mg/kg). The depressor responses of Ach (0.006mg/kg) and carotid sinus baroreflex were also discussed after treatment with the hormones in OVX rats.

用不同雌激素处理21天后,将动物用25%的氨基甲酸乙酯腹腔麻醉(1g/kg),做颈总动脉插管,通过压力换能器,将血压变化输入计算机,通过BL-310智能型生物信号显示与处理系统的压力测定模块,观察去势鼠及不同雌激素替代治疗大鼠静脉注射血管紧张素Ⅱ(AngⅡ,0.01mg/kg)、去甲肾上腺素(NA,0.01mg/kg)、肾上腺素(Adr,0.016mg/kg)的升压反应以及乙酰胆碱(Ach,0.006mg/kg)的降压反应和压力感受性反射的变化。

Methods: there were 40 youth Essential Hypertension patient examples who measure up , 20 cases as the group with electro-acupuncture Quchi and Taichong were observed vs 20 cases as the group with Catto pulley,observe the blood pressure and the principal clinical symptom;6 Essential Hypertension rats as the group with electro-acupuncture Quchi and Taichong were observed vs 6 Western medicine Catto pulley group、6 un-disposal sential Hypertension rats,observe the blood pressure、the plasmodia Noradrenalin and AngiotensionⅡlevel change.

临床试验将40例符合纳入标准的青年原发性高血压患者,按就诊顺序随机分为电针组和对照组各20例,分别给予电针曲池、太冲穴和口服卡托普利治疗。观察治疗前后血压、临床症状总积分及主要症状的变化。动物实验将自发性高血压大鼠18只随机分为3组,每组6只,分为电针组、卡托普利组和模型组,2周后观察各组的降压疗效,及血浆去甲肾上腺素、血管紧张素Ⅱ含量的变化。

Furthermore, recent studies have underlined that there are intricate interplays among adipocytes, the sympathetic nervous system, and the renin-angiotensin system, which participate in the obesity-associated dysmetabolic state.

最近的研究进一步揭示,在脂肪细胞、交感神经系统和肾素-血管紧张素系统之间存在密切的联系,其参与了肥胖相关的代谢失调状态。

We inestigated functional coupling of the receptors by measuring contractility in isolated trabeculae stimulated with increasing concentrations of angiotensin II.

我们增加血管紧张素II的浓度来刺激离体心肌小节,通过测量心肌小节的收缩性来探索受体的功能偶联。

A stressful unhealthy lifestyle causes constriction of fine blood vessels in the skin.

一种紧张的不健康的生活方式引起在皮肤里好血管的收缩。

Strong heart mao is still treatment CHF of tradition position, from 80's blood vessel strain ACEI drive clinical extensive application, get by its special pharmacology foundation quick development, particularly three new ACEI, make CHF treatment put off to a later time solution symptom ascension to lower death rate quality of change, is see by people as treatment cardiovascular disease new of turn.

强心甙类仍是治疗心衰的传统地位,自80年代以来血管紧张素转换酶抑制剂被临床广泛应用,以其独特药理基础得到迅速发展,尤其第三代新型ACEI,使心衰治疗从缓解症状上升到降低死亡率质的转变,被人们视为治疗心血管病新的转折。

In recent years,the role of Angiotensin-Converting-Enzyme Inhibitors in intervening IGT is attracting more and more attention from people.This article reviews the research progresses about IGT intreating and preventing IGT.

近年来,血管紧张素转换酶抑制剂类药物对IGT人群的干预作用越来越引起人们的关注,现就ACEI类药物防治IGT的研究现状进行回顾。

In vivo, this enzyme is involved in the metabolism of angiotensin III in the brain and peripheral organs, in the degradation of nociceptin and in the inactivation of enkephalins, in association with neutral endopeptidase NEP.

在体内该酶参与血管紧张素Ⅲ在脑部及外周器官的代谢、脑啡肽的降解和内啡肽的失活,并与中性内肽酶NEP有关。

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此刻,也许正是在通往天国的路上,我体验着这白色的晕旋。

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