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血管瘤

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Cherry angiomas natural treatments are available, however, you must first learn the cause of cherry angioma.

樱桃血管瘤的自然疗法,可但是,你必须先了解的原因樱桃状血管瘤

Presented with early artery enhancement and immediate wash out ,Two cases presented with wash in quickly and wash out slowly .The enhancement pattern of 36 liver metastatics cases was divided into the little blood provides group and the much blood provides group . The 5 cases of haemangioma were enhanced centripetally in portal phase and washed out after several minutes without arterial enhancement.

通过造影后的表现还可明确提示以下几个方面:(1)肿块内的坏死区;(2)在延迟相发现了肝内小转移灶,从而改变了临床治疗方案;(3)滋养动脉的显示;(4)门脉内栓子的鉴别诊断。36个转移性肝癌病灶分为少血供转移癌组和多血供转移癌组。6例肝血管瘤中5例均表现为向心性环行持续缓慢充填,另一例9mm的小血管瘤表现为完全、快速向心填充。

Subsequently, more diagnostically challenging, atypical haemangiomas, papillary endothelial hyperplasia, angiomatosis and angiolymphoid hyperplasia with eosinophilia are considered.

接着综述了诊断具有挑战性的病变如不典型血管瘤、乳头状内皮增生、血管瘤并和伴有嗜酸细胞浸润的血管淋巴样增生。

Results (1) 6 cases of Tuberous Sclerosis. Diffused subependymal nodular calcification lesions were found in all cases on unenhenced CT. 4 patients are 2 pairs of mother/child relationship. Both of the two mothers are found to suffer from renal angiomyolipoma.(2)1 case of neurofibromatosis showed abnormal spinal canal: scoliosis of thoracic and lumbar spine, concave change of vertebral posterior border. Bilateral renal hypogenesis was found in this patient. Diffused hyper-density lesions were found in kidney and fatty accumulation was found in back skin.(3)6 cases of Sturge-Weber syndrome. On unenhenced CT, curving and strip-shaped calcifications were found along the parietal and occipital gyrus.

结果 ①结节性硬化6例,所有病例CT平扫见两侧脑室室管膜下多发小结节状高密度钙化灶,其中4例为两对母子关系,并见两位母亲合并有肾脏错构瘤,;②神经纤维瘤病1例,MRI表现为椎管异常,胸腰段脊柱侧弯,椎体后缘呈明显的切凹改变;伴有双肾发育不良,CT示肾内多个高密度影,背部皮肤多量脂肪堆积,③脑颜面血管瘤综合征6例, CT可见顶枕部沿脑回分布的弯曲的条状高密度钙化,部分延伸致侧脑室内,增强后见病灶内有扭曲的条状和结节状明显强化的血管影;④小脑血管瘤病4例,影像学表现为小脑内大囊、小结节样占位性病变。

Hemangioma model established in nude mice by hypodermically injection with human hemangioma endothelial cells retains all the biological characteristics of the human hemangioma and can be a good model for research on hemangioma.

皮下植入人血管瘤内皮细胞的裸鼠动物模型建模方法可靠,能较真实地反映人血管瘤的发展过程,是研究血管瘤的较理想的平台。

Methods:Six cases of large intestinal diffuse cavernous hemangiomas were surgically treated from 1994 to 2005, with whole Colorectal involved in 1 case, rectosigmoid colon in 4 cases, rectum in 1 case.

1994年至2005年手术治疗6例,其中全结直肠弥漫性海绵状血管瘤1例,直肠乙状结肠弥漫性海绵状血管瘤4例,直肠弥漫性海绵状血管瘤1例。

The expressions of Bax and TGFβ1 in 50 children cases of dermal hemangioma which were in proliferative phase or in involuting phase were detected by HE staining and immunohistochemical method. We also analysized the difference between the different phases of hemangioma , and try to find out the role of Bax and TGFβ1 in hemangiomatous proliferation and regression .

采用HE染色和免疫组织化学方法检测50例小儿皮肤血管瘤增生期、退化期Bax、TGFβ1的表达水平,并分析它们在不同时期血管瘤中的差别,近而探讨Bax、TGFβ1在血管瘤增生、消退中的作用。

Results: The positive expression rates of Matrix MetallProtinase-2 protein in the cases of proliferating groups, involuting group , vascular malformation group and normal skin group were respectively 32%、12%、8%、5%.

结果 在血管瘤增生期、血管瘤退化期、血管畸形、正常皮肤组织中MMP-2表达的阳性率分别为32%、12%、8%、5%,血管瘤增生期组明显高于其它3组(P.05);其差别具有显著性意义。

Its difference was significant. The positive expression rates of Tissue Inhibitors of Metalloproteinase-2 protein in the cases of proliferating group, involuting group , vascular malformation group and normal skin group involuting group were respectively10%、28%、6%、5%.

其差别具有显著性意义 1、增生期血管瘤组织中,MMP-2表达明显升高,可以认为MMP-2通过降解血管基膜和细胞外基质促进小儿血管瘤血管形成,从而促进血管瘤增殖。

The positive expression rates of Tissue Inhibitors of Metalloproteinase-2 protein in the cases of involuting groups were significantly higher than those in proliferating group and vascular malformation groups and normal skin group (P.05). Its difference was significant. Conclusions: Matrix MetallProtinase-2 and Tissue Inhibitors of Metalloproteinase-2 may play an important role in the pathogenesis of hemangiomas through angiogenesis. They may have no effect on vascular malformation and normal skin

血管瘤退化期,TIMP-2表达明显升高,说明 TIMP-2与血管瘤纤维化、消退有关,其机制可能是一方面通过抑制MMP-2介导的基底膜降解而在内皮细胞迁移、聚集中起作用,另一方面直接抑制内皮细胞的增殖及迁移,使内皮细胞高频率调亡,细胞外基质增多,组织出现纤维化,最终导致血管瘤的退化。2、血管畸形和正常皮肤小血管中MMP-2和TIMP-2不表达或表达较弱,说明血管畸形和正常皮肤小血管中并不存在异常的血管生成,据此可以对血管性疾病进行分型、分期,并指导临床治疗。

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