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Objective To observe effect of large blood vessel hypoxia on the Wister rat's NO activity.

目的 探讨大血管缺氧对Wistar大鼠一氧化氮活性的影响。

Objective: To observe thrombin activity in a rat model of aortic endothelial denudation.

目的:观察大鼠主动脉拉伤模型血管壁凝血酶活性的变化。

Objective: To study the effects of nitric oxide andvasoactive intestinal polypeptide in the pathogenesis of experimental reflux esophagitis.

目的:探讨一氧化氮及血管活性肠肽在实验性反流性食管炎发病机制中的作用。

Objective: To evaluate the changes of VIP in the pterygopalatine ganglion in rats with allergic rhinitis.

目的:探讨变应性鼻炎大鼠翼腭神经节中血管活性肠肽的变化情况。

MAIN OUTCOME MEASURES: Integrality of transplanted vessels, intimal thickness and nitric oxide synthase activity in the intima.

主要观察指标:静脉移植段血管内皮的完整性、内膜厚度及内膜一氧化氮合酶活性。

Furthermore, the maladjustment of vasoactive peptide is deemed to be an important aspect of VSA.

此外,血管活性肽失调被认为是VSA发病机制的又一个重要方面。

The treatment group consisted of 44 patients who received mutine thempv(①rapid and adequate fluid resuscitation,④)rapid restoration of important organ circulation bv treatment with cardiovascular drugs,⑧early enteral nutritional support to protect gastrointestinal mucosal barrier.et al·+CVVH.The control group consisted of 32 patients who received only routine therapv.

回顾性分析76例严重脓毒症伴脓毒性休克患者,分为两组,常规综合治疗组32例,采用快速而充分的液体复苏、血管活性药物改善脏器灌注、早期肠道营养保护肠黏膜屏障等综合治疗;综合治疗+CVVH组44例,在常规综合治疗基础上加CVVH治疗。

76 patients were divided into either the treatment group or the control group.The treatment group consisted of 44 patients who received mutine thempv(①rapid and adequate fluid resuscitation,④)rapid restoration of important organ circulation bv treatment with cardiovascular drugs,⑧early enteral nutritional support to protect gastrointestinal mucosal barrier.et al·+CVVH.The control group consisted of 32 patients who received only routine therapv.

回顾性分析76例严重脓毒症伴脓毒性休克患者,分为两组,常规综合治疗组32例,采用快速而充分的液体复苏、血管活性药物改善脏器灌注、早期肠道营养保护肠黏膜屏障等综合治疗;综合治疗+CVVH组44例,在常规综合治疗基础上加CVVH治疗。

In hypertensive patients with heart failure,the ratio of ADM/ADT decreased with the deterioration of heart failure Conclusion:The oversecretion of ADT and slow compensation of ADM is a characteristic deregulation of vasoactive peptide in the process of hypertensive heart failure.

ADM代偿缓慢和ADT分泌过度是高血压心力衰竭过程中血管活性肽调节紊乱的一个特点。

METHODS: Human premolars were collected and embedded in paraffin and cut into slices.

目的:研究血管活性肠肽在牙髓牙本质复合体中的表达,探讨其功能。

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