血管收缩的

The first part consists of three experiments:(1) The rings were incubated in KH, 20, 50 mmol/L 〓 for 1 house, relaxation in response to the EDHF stimuli A23187 in 30nmol/L U46619-induced preconrtaction in the presence of 7 μ mol/L indomethacin, a cyclooxygenase inhibitor, 300μmol/L LNNA, a nitric oxide biosyhnthesis inhibitor, and 1mmol/L tetraethylammonium , a 〓 blocker, or 3 μmol/L glibenclamide , a 〓 blocker, was compared with the control;(2) After the arteries were incubated in KH, UW solution or HTK solution at 4℃ for 4 hours, endothelium-derived relaxation (percentage of 30nmol/L U46619 precontraction) was induced by A23187 in the present of 7 μmol/L indomethacir and 300μmol/L LNNA;(3) After incubation with KH, UW solution and STH (either at 37℃ in oxygenated organ chamber or at 4℃ in a refrigerator for 4 hours), endothelium-derived relaxation (percentage of 30nmol/L U46619 precontraction) was induced by A23187 in the present of 7 μ mol/L indomethacin and 300 μmol/L LNNA.

第一部分研究结果：（1）单纯浸泡于KH的冠状动脉A23187能引发66.67％的血管舒张反应，经TEA及20mmol／L、50mmol／L钾离子作用后，血管舒张反应程度显著降低，但经GBM作用后改变不明显；（2）与保存于KH的冠状动脉相比，A23187引发的血管舒张反应程度，保存于UW液的明显下降，保存于HTK液的无明显变化；（3）在37℃条件下，血管环浸泡于STH出现缓慢轻微的舒张反应，浸泡于UW液初期出现短暂收缩反应，但此后主要以舒张为主：在37℃条件下，血管经UW液保存后，U46619引发的收缩反应程度降低；不论在37℃或4℃条件下，A23187引发的血管舒张反应，经UW液保存后明显下降，但用STH保存后变化不明显。

Endovascular restenosis is an abnormal repairing response of vessel against injury. Following stent implantation, vessel contractibility reconstitution, local inflammation and thrombogenesis, intima hyperplasia, mainly vascular smooth muscle hyperplasia, are found.

血管内再狭窄是血管对抗损伤的一种异常修复反应，表现为支架植入后血管的收缩性重构，局部的炎症与血栓形成，以及以血管平滑肌细胞增殖为主的内膜增生。

To measure the flow velocity of the lateral ascending cervical arteries and the small vessel into femoral epiphysis and RI of arteries.

测量髋关节外侧颈升动脉及股骨头骨骺终末血管的血流速度，动脉血管流速为收缩期峰值流速。

In contrast, the dilatory responses to the nitric oxide donor sodium nitroprusside and olume expansion were enhanced.

与此相反，对硝普纳的扩血管效应则提高。而此些收缩反应和扩血管效应能够被NO合成抑制剂N-硝基-L-精氨酸甲酯所纠正。

Both ACEI with or without sulfhydryl groups could prevent the H2O2 induced decrease in contraction responses to PE in intact aortic rings. The increase of NO and activation of HO-1 might be involved in the mechanism.

含有和不含筑基的ACEI均具有对抗H2O2引起的血管收缩功能下降的作用，其主要机制可能是通过引起NO产生增加和诱导HO-1而实现的。

Classically, Ang Ⅱ is well known to cause potent increases in systemic and local blood pressure via its vasoconstrictive effect, to influence renal tubules to retain sodium and water, and to stimulate aldosterone release from the adrenal gland.

众所周知，Ang Ⅱ通过它的血管收缩效应导致系统和局部的血压增高并影响肾小管的钠水潴留以及刺激肾上腺的醛固酮释放。

The information can also transfer via a signal transduction pathway, therefore ET-1 can cooperate with some growth factors such as bFGF, TGF-β and PDGF. ET-1 is not only a potent vasoconstrictive peptide, but also a strong cellular division factor. It is an important regulator to intimal hyperplasia.

因为ET-1不仅是最强的血管收缩物质，而且还具有强烈的细胞分裂作用，所以，ET-1是内膜增生的重要调节因子，但目前对于静脉移植术后不同时期ET-1的动态变化及其与内膜增生相互关系的研究尚不多见。

Endothelin-1 (ET-1) is a potent vasoconstrictive peptide that was isolated initially from the conditioned medium of cultured endothelial cells. It is a pathogenic mediator with a number of deleterious effects, including vasoconstriction, fibrosis, vascular hypertrophy,and inflammation.Atrial natriuretic peptide, angiotensin II and nitric oxide also takes an important role in cardiovascular system.

内皮素-1(endothelia-1，ET-1)是一种由内皮细胞合成和分泌的、具有强烈收缩血管作用和促进细胞增生的血管活性多肽，心钠素(atrialnatriuretic peptide，ANP)、血管紧张素Ⅱ(angiotensin Ⅱ，AⅡ)、NO等在心血管系统功能的调节中也起着重要作用。

Results Both inhibitions of PKC with 6.8 mmolL^(-1) hypericin and PKA with 0.112 mmolL^(-1) KT5720 shifted the dose-response curve of 15-HETE induced pulmonary vasoconstriction to the right, and more powerful inhibition was observed with PKC inhibitor (P.01). Denuding endothelia decreased the response of PA ring to 15-HETE in both normoxic and hypoxic PA rings (P.05, P.01, respectively).

结果 用6.8 mmolL^(-1) hypericin阻断PKC途径可使15-HETE收缩缺氧大鼠肺动脉量效曲线右移(P.01)；用0.112 mmolL^(-1) KT5720阻断PKA途径使15-HETE收缩缺氧大鼠肺动脉量效曲线右移不明显；除去血管内皮细胞可降低对照组和缺氧组肺动脉血管环对15-HETE的反应性。

Effects of H_2O_2 on the tension of isolated aortic rings of rat As above methods,Our results indicated that (1) aortic rings with endothelium precontracted by NE to 10000μmol/L H_2O_2 induced a relaxation; aortic rings with endothelium to 100μmol/L H_2O_2 induced a significant contraction;H_2O_2 induced the same effects in aortic rings without endothelium, there is significant difference in presence and absence of endothelium.

双氧水对大鼠离体血管环张力的影响方法同2，结果：①NE预收缩后，加入10000μmol／L H_2O_2，内皮完整血管环表现为舒张作用；加入100μmol／L H_2O_2后血管环表现为收缩作用；H_2O_2对去内皮的血管环同样具有上述作用，与内皮完整组相比差异显著；②10000μmol／L H_2O_2可使得内皮完整及去内皮血管环中NO含量升高；100μmol／L H_2O_2可使得NO含量降低。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。