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The coronary ectasia may be a compensatory mechanism to resist coronary artery atherosclerosis, while coronary atherosclerosis is the result of reduced compensatory function.

冠脉扩张可能是抵抗动脉粥样硬化发生而启动的机体正常代偿功能,而冠状动脉粥样硬化是机体失去正常代偿功能所导致的血管损伤。

Results The lesion located at the basal segments of left lower lobe in 9 cases (90%), furthermore, 7(70%) at the posterior basal segments; The remainder 1 case (10%) was found at the posterior basal segment of right lower lobe; The lesion present as multiloculated cyst containing fluid and/or air in 7 cases(70%) without contrast enhancement of cyst wall, 3 cases (30%) presented as irregular mass with moderate enhancement, in which bronchiectasis with mucous impaction, calcification or cyst containing fluid may be seen; MRP, MIP and VR reconstructions showed anomalous branches arising from descending aorta in all 10 patients(100%) with draining into left atrium via pulmonary vein,which was consistent with surgery; Ipsilateral hilar lymphadenopathy was demonstrated in 7 cases (70%).

结果 9例(90%)病灶位于左下叶基底段,其中7例(70%)位于左下叶后基底段,1例(10%)位于右下叶后基底段;7例(70%)病灶表现为含液/气的薄壁多房囊腔,增强后囊壁无明显强化,3例(30%)表现为不规则团块,增强后有中等强化,其内有粘液嵌塞的扩张支气管,也可有钙化或液性区;MPR、MIP及VR等三维重建显示10例(100%)均为降主动脉发出异常分支供血,并通过肺静脉回流入左房,血管显示情况与手术相符;7例(70%)患侧肺门淋巴结有增大。

Among the various endovascular techniques that have been tested so far, we propose self-expandable covered stents as ideal for treating arterial aneurysms and pseudoaneurysms of the petrous and cavernous carotid segments.

迄今为止各种已经检验过的血管内操作方法中,我们建议植入自我可膨胀扩张盖,这是颈动脉颞骨岩部和壁内段动脉瘤和假动脉瘤的理想治疗方法。

Previous studies showed that before the pial artery large dilation which were widely observed in CSD, there is also a small constriction occurring in pial arteres.

已有研究表明,在普遍被观察到的CSD过程中软脑膜动脉血管大幅度舒张之前,还存在一个较小幅度的软脑膜动脉扩张和收缩。

It is proposed that the vasodilating and hypotensive effect of propofol be probably mediated via the increase of plasma CGRP and NT.

推测异丙酚扩张血管、降低血压的作用可能与CGRP及NT的增加有关。

SI could abolish the effect of L-NMMA on vasomotion but L-NMMA did not influence the effect of SI on vasomotion.

在L—NMMA的有效作用时间内应用SI可使被抑制于收缩状态的血管重新扩张。

Nitric oxide increases the flow of blood to tissues by dilating the arteries that penetrate those tissues.

通过扩张深入组织的动脉血管,一氧化氮增加了流向组织的血液。

Accordingly, this class of drug decreases the blood pressure by simultaneously dilating the peripheral arteries and increasing the body's loss of salt.

相应地,这种药物可以通过扩张血管降低血压的同时提高身体盐分的排除量。

Pupils dilate, arteries constrict, core temperature rises, heart races, blood pressure skyrockets, respiration becomes rapid and shallow, the brain fires bursts of electrical impulses from nowhere to nowhere, and secretions spit out of every gland, and the muscles tense and spasm like you re lifting three times your body weight.

学生扩张,血管收缩,核心温度的升高,心跳,血压焰火,呼吸变得快速,浅,脑火电脉冲连发从无到有从无到有,分泌物吐出每腺,肌肉紧张和痉挛等你取消你的3倍体重。

BACKGROUND: Restenosis and lumina loss limit further application of balloon extension and stent implantation. Effect of tunica intima proliferation and apoptosis in restenosis and the intervention method are exploring.

摘要背景:球囊扩张及支架置入后再狭窄、管腔丢失等问题制约了支架置入治疗的进一步发展,血管内膜增殖和凋亡在再狭窄中的作用及干预方法尚在积极探索中。

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如果有任何问题,你可以随时联系我。

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