血管作用的

The third part is to study the role of apoptosis in the mechanism of TTT in treatment of CNV.

提示TTT照射可以通过降低视网膜组织VEGF的表达来起到抑制新生血管的作用；TTT照射引起MMP-1的表达升高可能通过增加胶原成分的降解来促进CNV纤维膜的回退。

Standard treatment for septic shock includes antibiotics, intravenous fluids and vasopressor medicines (drugs to increase blood pressure)," explained the first study's lead author, Dr. Charles Sprung, director of the general intensive care unit, department of anesthesiology and critical care medicine at Hadassah Hebrew University Medical Center in Israel."Until our study, doctors in hospitals around the world were also routinely using steroids as adjunct therapy to help hundreds of thousands of patients in septic shock fight for their lives. Our study demonstrates that this adjunct therapy of hydrocortisone is not helpful for most patients with septic shock and, in fact, may be harmful.

标准的败血症性休克治疗包括抗生素、静脉补液和血管升压药物，"第一作者，以色列Hadassah Hebrew 大学医学中心麻醉学部与危重症医学部综合加强监护单元主任Charles Sprung博士解释道，"在我们的研究之前，全世界的医生常规使用类固醇激素辅助治疗成千上万的败血症性休克患者，但是我们的研究证实了氢化可的松对多数患者并没有辅助治疗的作用，实际上可能是有害的。

The small rat pulmonary tissue surface of ⅲ and ⅳ group can't see any abnormality; the capillary vessel of alveolar wall dilation appears a little hyperemia in different degree under the microscope.conclusion different measures of potentill anserine extract has different effects on endothelial cell of capillary vessel of small rats' alveolar wall. endothelial cell of capillary vessel of alveolar wall has hyperplasia and hypertrophy in different degrees; it leads to permeability of capillary vessel increasing in different extent.

不同剂量的蕨麻提取物对小鼠肺泡壁毛细血管内皮细胞的影响不同，肺泡壁毛细血管内皮细胞呈不同程度的增生肥大导致毛细血管通透性呈现不同程度的增加，表明可能与不同浓度的蕨麻提取物促进小鼠肺组织某些血管内皮细胞因子（vascular endothelial cell factor vecf ）的分泌作用有关。

More and more evidences have shown that traditional high 〓 cardioplegic solutions increase energy and oxygen demand during asystole; make more calcium entry into cells.

愈来愈多的研究表明：传统高〓心肌保护液于诱导心脏停搏期间增加心肌能耗、氧耗，介导〓内流并对传导系统抑制作用相对较弱，高〓所致的血管内皮细胞损伤也引起学者们的高度重视，如何减轻或避免高〓心肌保护液的不利影响，是心外科领域中一个亟待解决的难题。

3NE、MTT、LDH、ET-1、ICAM-1 and Tie-2experiment show: in the process of oxygen glucose deprivation/reoxygenation ,BMEC have subjected great damagement, I/R groups have significant differences compared with NC groups (p.05);polymorphonuclear leukocyte can aggregrate this injury, I/R+PMN groups have significant differences compared with I/R groups (p.05); after Sivelestat Sodium treatment, the effect of I/R injury has been decreased, I/R+PMN+Siv groups have significant differences compared with I/R+PMN groups (p.05), and NE、MTT、LDH experiment show that the three dose groups there was a significant dose-effect relationship.

3测定NE、MTT、LDH、ET-1、ICAM-1、Tie-2 mRNA均显示：氧糖剥夺/复氧复糖过程能对BMEC造成损伤，I/R组与NC组比较有明显的统计学差异(p.05)；中性粒细胞能加重对BMEC的损伤，I/R组与I/R+PMN组之间比较亦有统计学差异(p.05)；西维来司钠处理后，血管内皮细胞的损伤明显减轻，I/R+PMN组与I/R+PMN+SIR组比较有统计学差异(p.05)，并且NE测定以及MTT、LDH实验提示在西维来司钠三个剂量组之间存在明显的量-效关系，其对BMEC的保护作用有剂量依赖性。

Objective To use an ischemical reperfusion injury rat model by pulsinelli-4-VO andobserve the changes of the expression of Glu, N-methy-D-asparagic acidreceptor subunits NR_1 and NR_(2B) and ability of learning and memory after procerebrum ischemic reperfusion injury, and approach the pathogenesy of learning andmemory disorder induced by ischemic reperfusion injury, and the intervention ofginsenoside Rg_2 on the learning and memory disorder after procerebrumischemic reperfusion injury.

目的利用pulsinelli四血管法体建立脑缺血／再灌注损伤模型，观察前脑缺血损伤后谷氨酸、N-甲基-D-天门冬氨酸受体亚单位NR_1、NR_(2B)表达的变化及学习记忆的改变，从兴奋性氨基酸的兴奋毒作用尤其是其受体的表达方面探讨脑缺血／再灌注损伤引起的学习记忆功能障碍的发生机制，并观察人参皂苷Rg_2对脑缺血缺氧损伤引起的学习记忆障碍的干预机制。

Flap dissection studies confirmed that this is because of the poorly developed subfascial plexus in the forearm, with preservation of the deep fascia not contributing to the mechanism of flap perfusion.

用筋膜上和筋膜下技术制取的皮瓣，测量其血管分布区没有显著的差别，皮瓣的解剖研究表明，因为前臂的筋膜下网状组织发育不良，在深筋膜的保护下，并不能对皮瓣的灌注起作用。

Observe the formula's effects to the diabetic nephropathy rat model cause by small dose STZ injured, high lipin feed and hemiresection, the results showed that can decrease significantly blood sugar after diet 2h, control availably hyperplasia of glomerulus mesenteric cell, inhibite tumefaction of glomerulus capillary endothelial cell, keep integrality of podocytic process and basement membrane, relieve vasal cavum stricture, reduce the GFR and excretory rate of 〓-MG, improve the status of high blood lipin, specially decrease significantly TC, reduce the 〓 and T/K, improve the status of microcirculation obstacle, elevate the activity of 〓-〓-ATP enzyme.

观察降糖益肾方对小剂量STZ诱导并单侧肾切除，高脂饲料喂养的糖尿病肾病大鼠模型的影响实验结果：对2h后血糖有显著降低作用（p.05），能有效抑制肾小球系膜基质的增生，减轻肾小球毛细血管内皮细胞肿胀，保持足突与基底膜的完整性，减轻血管腔狭窄，降低GFR、尿微量白蛋白以及〓-微球蛋白的排泄率（p.05），能改善高血脂状态，降低血TG、TC含量（p.05），降低〓与T／K比值（p.05），改善微血管循环障碍的状态；能升高肾组织中〓-〓-ATP酶的活性（p.05）。

The information can also transfer via a signal transduction pathway, therefore ET-1 can cooperate with some growth factors such as bFGF, TGF-β and PDGF. ET-1 is not only a potent vasoconstrictive peptide, but also a strong cellular division factor. It is an important regulator to intimal hyperplasia.

因为ET-1不仅是最强的血管收缩物质，而且还具有强烈的细胞分裂作用，所以，ET-1是内膜增生的重要调节因子，但目前对于静脉移植术后不同时期ET-1的动态变化及其与内膜增生相互关系的研究尚不多见。

Reduce the hoist of leucocytes of sinus coronaries blood of myocardial ischemia and reperfusion injury dogs;2. decrease the leakage of plasma CK, CK-MB and LDH;3. regulate the vaso-active substances such as ET, TXB〓 and PGI〓;4. decreased the inflammatory cell factors such as IL-1, IL-6 and TNF

综上，奥沙恩注射液可降低心肌缺血再灌注损伤模型犬冠状窦血白细胞总数，减少I／R后CK、CK-MB和LDH的释放，调节血管活性物质ET、〓和〓的产生，降低血清炎性细胞因子TNF-α、IL-1和IL-6的含量，抑制PMN的激活及对心肌的浸润，减轻心肌组织的病理改变，以及减轻心肌缺血及坏死程度、降低心肌耗氧量，从而显示出较强的对犬急性心肌缺血再灌注损伤的保护作用。

With Death guitarist Schuldiner adopting vocal duties, the band made a major impact on the scene.

随着死亡的吉他手Schuldiner接受主唱的职务，乐队在现实中树立了重要的影响。

But he could still end up breakfasting on Swiss-government issue muesli because all six are accused of nicking around 45 million pounds they should have paid to FIFA.

不过他最后仍有可能沦为瑞士政府&议事餐桌&上的一道早餐，因为这所有六个人都被指控把本应支付给国际足联的大约4500万英镑骗了个精光。