血清反应

In peripheral blood,〓increases, so cause 〓 balance is broken, and 〓 which can induce inflammatory reaction increases obviously too; meanwhile, there are immuno- function change in the red cells, the RBC-〓 receptor rosette promoting rate of serum decrease, indicate this uveitic animal model have both cellular immunity dysfunction induced by T cells and the changes of RBC immuno-function. The analysis of the local and systemic immuno-function changes of uveitic animal model offers objective theoretical bases for combining the local treatment and the systemic treatment.

葡萄膜炎兔房水中房水中〓及IL-2均升高，病变葡萄膜组织中T细胞表面分化抗原的免疫组化检查显示大量阳性细胞，表明兔葡萄膜组织本身存在着免疫功能亢进所导致的病理改变，而外周血中〓增高，导致〓平衡失调，介导炎症反应的〓亦明显增高，同时伴有红细胞免疫功能改变，外周血中RBC-〓受体花环率及血清中RBC-〓受体花环促进率均降低，显示葡萄膜炎兔本身既存在T细胞介导的细胞免疫反应功能紊乱，也存在红细胞免疫功能改变，这种局部和全身的免疫功能改变分析为葡萄膜炎的治疗局部和全身相结合提供了客观的理论依据。

The reacting tite with NDPK-A was 1:800 and theantisera was specific.Conclusions: The purified rhNDPK-A protein exists mainly in the form of hexamer;rhNDPK-A protein can promote the survival of spleen cell and may take part in theadjustment of cell growth by the way of changing cell cycle.

ELISA法检测所制备的抗体的反应效价为：1：800，western blotting试验结果证实，该抗血清具备较好的特异性，能与rhNDPK-A蛋白发生反应，结论：rhNDPK-A蛋白可能主要以六聚体形式存在，直接观察的蛋白结构与文献报道的X衍射结果相似；rhNDPK-A蛋白对正常小鼠的脾细胞的存活有促进作用，推测很可能以信号转导的方式调控细胞周期从而达到对细胞生长的调节作用。

The labelled conjugates were stable in aqueous solution and serum solution in vitro, but they were not stable when challenged with cysteine and in vivo.

实验结果表明，标记物在反应溶液和小牛血清中较稳定，但在与半胱氨酸的竞争反应中及在生物体内的稳定性较差。

Antisera from normal mice and mice immunized with pcDNA3 were both negative on Jurkat cells.

免疫组化结果表明，该血清不与表达TCR γδ的人T淋巴瘤细胞发生反应，而与转染该基因的SP2/0细胞产生强阳性反应。

Some exudation and bleedings happen in the pulmonary alveoli, some inflammatory cells infiltrate into lung tissues; in the serum AMY, ALP, TNF-α, IL-6 increases obviously high, COX-2 expression increase; The expression of NF-κB in big rat lung of SAP increase, and the nucleus is thick dyed, it explain that the NF-κ moved into the nucleus; Stimulate the inflamatory reaction.

我们的实验中见到SAP组的大鼠肺脏组织细胞腔扩大、肺泡壁断裂，有的出现肺大泡，肺泡内伴有渗出及少量非出血，肺组织可见咽细胞浸润；血清中AMY、ALP、TNF-α、IL-6明显增高；在SAP组大鼠肝细胞、肾小管上皮细胞、肺脏小气管及肺泡上皮上COX-2表达增加；NF-κB在SAP大鼠肺脏中表达增加，并且出现核浓染，说明NF-κB前移入核；启动炎症反应等一系列反应SAP大鼠受损伤的肺组织中AQP-1无论在mRNA水平上、蛋白水平上还是免疫组化染色病理学检测上都明显下降。

Moreover, in order to make clear whether IV infection touches off the cause and its pathogenesis of artherosclerosis inflammatory reaction. The study also uses IV of type A1 and A3 to infect repeatedly Jimpy mice with E gene defect, to observe AS histology change of Jimpy mice aorta and detect the change of IV antibody and inflammatory reaction medium (eg.TNF-α, IL-1) in Jimpy mice serum and express of monocyte chemotactic factor( MCF-1) and nuclear factor kB in Jimpy mice aorta tissue .

另外，为了弄清流感病毒感染是否是触发动脉粥样硬化炎症反应的发生原因及其机理，本研究还用A1、A3型流感病毒反复感染载脂蛋白E基因缺陷小鼠，观察小鼠主动脉的动脉粥样硬化组织学改变，并检测小鼠血清中流感病毒抗体、炎症反应介质（如TNF-a、IL-1等）变化情况以及小鼠主动脉组织中的单核细胞趋化因子-1（MCP-1）和核转录因子kB表达情况。

Methods The present study has been carried out using NaCMC injected into the mice's abdominal cavity to cause inflammation and induce white blood cell migration,varnishing DNFB to induce mice's DTH,injecting colloid activated carbon suspension into mice's tail vein,with 10% of sheep RBC injected into the abdomen to immunize mice.We observed inhibitory function of Liyankang granule on NaCMC to cause inflammation and induce white blood cell migration,the effect of DNFB on inducing mice's DTH and the effect on clearance speed of carbon particle in mice's blood and serum agglution of mice.

采用大鼠腹腔注射羟甲基纤维素钠致炎诱发白细胞游走，涂抹二硝基氟苯诱导小鼠迟发型超敏反应，小鼠尾静脉注射胶体碳悬液，腹腔注射10%羊红细胞免疫小鼠，观察小儿利咽康颗粒剂对羟甲基纤维素钠致炎诱发白细胞游走的抑制作用，对二硝基氟苯诱导小鼠迟发型超敏反应、对小鼠血中碳粒清除速度、对小鼠血清凝集素的影响。

Methods The present study has been carried out using NaCMC injected into the mice's abdominal cavity to cause inflammation and induce white blood cell migration,varnishing DNFB to induce mice's DTH,injecting colloid activated carbon suspension into mice's tail vein,with 10% of sheep RBC injected into the abdomen to immunize mice.

采用大鼠腹腔注射羟甲基纤维素钠致炎诱发白细胞游走，涂抹二硝基氟苯诱导小鼠迟发型超敏反应，小鼠尾静脉注射胶体碳悬液，腹腔注射10%羊红细胞免疫小鼠，观察小儿利咽康颗粒剂对羟甲基纤维素钠致炎诱发白细胞游走的抑制作用，对二硝基氟苯诱导小鼠迟发型超敏反应、对小鼠血中碳粒清除速度、对小鼠血清凝集素的影响。

Department of Basic Medical Sciences,College of Vocational Technology,Liaoning University of TCM,Shenyang 110101.China Objective To observe immune regulation effect of Liyankang granule on children.Methods The present study has been carried out using NaCMC injected into the mice's abdominal cavity to cause inflammation and induce white blood cell migration,varnishing DNFB to induce mice's DTH,injecting colloid activated carbon suspension into mice's tail vein,with 10% of sheep RBC injected into the abdomen to immunize mice.We observed inhibitory function of Liyankang granule on NaCMC to cause inflammation and induce white blood cell migration,the effect of DNFB on inducing mice's DTH and the effect on clearance speed of carbon particle in mice's blood and serum agglution of mice.

采用大鼠腹腔注射羟甲基纤维素钠致炎诱发白细胞游走，涂抹二硝基氟苯诱导小鼠迟发型超敏反应，小鼠尾静脉注射胶体碳悬液，腹腔注射10%羊红细胞免疫小鼠，观察小儿利咽康颗粒剂对羟甲基纤维素钠致炎诱发白细胞游走的抑制作用，对二硝基氟苯诱导小鼠迟发型超敏反应、对小鼠血中碳粒清除速度、对小鼠血清凝集素的影响。

In the lungs, mainly derived from alveolar macrophages, airway epithelial cells, lymphocytes and neutrophil aggregation, IL-8 may tolerance serum and effusions Endopeptidase degradation, there a long time, which can be accumulated in local, in response to a sustained effect, enlarged inflammatory response .

在肺部，其主要来源于肺泡巨噬细胞，气道上皮细胞，淋巴细胞和聚集的中性粒细胞，IL-8可耐受血清和渗出液中肽酶的降解，存在时间长，因而能在局部累积，在反应中发挥持续效应，放大了炎症反应。

On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面，更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值，分为几个相等的区间作为横轴，并将各区间内所测定值依所出现的次数累积而成的面积，用柱子排起来的图形，也叫做柱状图。