血小板

These results indicated that they may be due to the mechahism of DNBEP to trest ITP by means of selective suppressiong of humoral immune function, two-way regulation of the cell immune function and recovery of the platelet function.

提示养血清癜汤的疗效机制是通过对体液免疫的选择性抑制作用，对细胞免疫的双向调节作用和对血小板功能的恢复作用来实现的。

Purpose To investigate the effect of 5'-nucleotidase from Trimeresurus albolabris venom on platelet aggregations and its mechanism.

目的 研究白唇竹叶青蛇毒5'-核苷酸酶对血小板聚集的影响，探讨其作用机制。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

A turbidimetric method was used to measure human washed platelet aggregations induced by O2 and effects of APNF(0134) on them.

并以比浊法观察了O2诱导的人洗涤血小板的聚集及APNF(0134)对其的影响。

Among the 6 coumarins, the Papp values of umbelliferone, osthole, angelolA and angelolB from AP to BL side were identical with that of propranolol, and columbianadin and columbianetin acetate lied between propranolol and atenolol.

前期研究［2］我们报道了独活中香豆素类化学成分的鉴定，这些成分多具有镇痛、抗炎、抑制血小板聚集和实验性血栓形成、拮抗钙通道阻滞剂受体等生物学活性［3］。

RESULTS: Unmedicated major depressed patients had significantly decreased platelet [3H]-rauwolscine binding Bmax values compared to normal volunteers.

结果：与健康志愿者比，未用药的抑郁症患者血小板[3H]-α-育亨宾和Bmax 都显著降低，而[3H]-α-育亨宾的结合常数没有区别。

RESULTS: Unmedicated major depressed patients had significantly decreased platelet [3H]-rauwolscine binding Bmax alues compared to normal olunteers.

结果：与健康志愿者比，未用药的抑郁症患者血小板[3H]-α-育亨宾和Bmax 都显著降低，而[3H]-α-育亨宾的结合常数没有区别。

But it has been manifested that some persons unsensitive to aspirin, namely so-called aspirin resistance exists for them.

但有研究显示，部分个体对阿司匹林的抗血小板作用不敏感，即存在所谓的阿司匹林抵抗现象(aspirin resistance，AR)。

It also reduced the formation of platelet-leukocyte conjugates,both in unstimulated and adenosine diphosphate-stimulated bloodsamples at 3%–4% end-expiratory sevoflurane concentrationsused to maintain anesthesia.

七氟醚呼末浓度为3％－4％时无论用还是不用二磷酸腺苷刺激的血标本中血小板－白细胞结合物的形成均减少。

Haemolytic uraemic syndrome is characterized by the triad of microangiopathic haemolytic anaemia, thrombocytopenia and acute renal failure.

溶血尿毒综合徵以微血管病性溶血性贫血，血小板减少症及急性肾功能衰竭三联症为特点。

Lugalbanda was a god and shepherd king of Uruk where he was worshipped for over a thousand years.

Lugalbanda 是神和被崇拜了一千年多 Uruk古埃及喜克索王朝国王。

I am coming just now,' and went on perfuming himself with Hunut, then he came and sat.

我来只是现在，'歼灭战perfuming自己与胡努特，那麼，他来到和SAT 。