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血小板

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Platelets: adhere to subendothelial tissue and r se PDGF which can lead to DNA synthesis and proliferation of SMCs; aggregate on the damaged site and give rise to thrombi formation.

2血小板:粘附于暴露的胶原组织,释放PDGF,促进SMC增生;在损伤部位聚集,形成血栓,此后机化造成管腔狭窄。

Superacute including detach of endothelium, aggregation of platelet, injury and activation of SMC in the vessel wall.

超急期可有内皮细胞的剥膜,血小板的聚积,管壁内SMC损伤和激活,原癌基因的表达和生长因子的释放。2。

Methods:Forty patients with supraventricular tachycardia under different treatment were observed and divided into heparin group and control group.

以40例室上性心动过速患者为对象,分为肝素组和空白对照组。在不同时间测定血小板聚集率及血栓素B2(TXB2)。

11 Does synergize with IL-3 to enhance the growth of both murine and human megaaryocyte colonies.

因此 IL上作为目前唯一能运用于临床的特异性升高血小板药物得到广泛关注。

The path-physiological mechanism of ICH is complicated, which includes the high intracranial pressure caused by hematom mechanical pressure, the lesion by the decrease of regional cerebral blood flow surrouding the hematom, even regions adjacent to the hematom, Meanwhile, the materials released from hematom including ferrohemoglobin, thrombase, plasma protein and platelet, leucocyte, glusate not only directly injury neurons but also blood brain barrier.

脑出血的病理生理学机制复杂,其中包括血肿的机械压迫所造成的持续的脑缺血性损害;血肿分解后产生的血红蛋白、凝血酶、血浆蛋白、血小板和白细胞、谷氨酸、自由基等毒性物质不仅可直接损伤神经元,而且可损害血脑屏障加重脑水肿。

BACKGROUND: Platelets from patients with type 2 diabetes mellitus display hyperaggregability and increased thrombogenic potential.

背景:在2行糖尿病患者血小板有较高的凝集能力和致栓潜能。

BACKGROUND: Platelets from patients with type 2 diabetes mellitus display hyperaggregability and increased thrombogenic potential.

摘要翻译:背景:在2行糖尿病患者血小板有较高的凝集能力和致栓潜能。

Pretreatment with antiplatelet therapy should decrease the thrombogenic potential of RF ablation.

不同的抗血小板制剂对射频消融所致的血栓前状态有改善作用。

Upon thrombogenic and inflammatory challenges, P-selectin is rapidly expressed, by exocytosis, on activated platelets and stimulated endothelial cells.

当细胞受到凝血酶原或炎症因子刺激后,P-选凝素会迅速地通过外排表达在活化的血小板和内皮细胞的表面。

Four thrombogenic and thrombolytic markers including endothelin-1(ET-1), platelet granule membrane protein-140 (GMP-140), thrombin active fragment and D-Dimer were determined before and after ILLLI or aspirin treatment.

观察两组治疗前后血浆中致血栓和纤溶指标内皮素-1、血小板α颗粒膜蛋白140(GMP-140)、凝血酶活性片段及D-二聚体的变化。

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