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血小板

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The rabbit platelet activation induced by platelet activating factor can be inhibite d by safflor yellow.

红花总黄色素可抑制PAF诱导的血小板聚集、5-HT释放及血小板内游离钙增加。

At sites of vascular injury, platelets come into contact with the subendothelial extracellular matrix which triggers their activation and the formation of a hemostatic plug.

摘要翻译:在血管受损部位,血小板与内皮下细胞外基质的接触使得血小板活化并形成血栓。

Exposure of subendothelial matrix proteins further facilitates firm attachment of platelets to the vessel wall by binding of collagen to their GPVI receptor.

内皮下基层的暴露进一步促使血小板通过糖蛋白VI受体与胶原结合使得血小板紧密的黏附在血管壁上。

Exposure of subendothelial matrix proteins further facilitates firm attachment of platelets to the essel wall by binding of collagen to their GPI receptor.

内皮下基层的暴露进一步促使血小板通过糖蛋白I受体与胶原结合使得血小板紧密的黏附在血管壁上。

At sites of vascular injury, platelets come into contact with the subendothelial extracellular matrix which triggers their activation and the formation of a hemostatic plug.

血小板损伤处,血小板聚集与内皮下细胞外基质接触而激活形成止血栓子。

At sites of vascular injury, platelets come into contact with the subendothelial extracellular matrix which triggers their activation and the formation of a hemostatic plug.

摘要翻译:在血小板损伤处,血小板聚集与内皮下细胞外基质接触而激活形成止血栓子。

Meta-analyses showed that the response rate of TP (topotecan + cisplatin) regimen had no significant difference compared with EP regimen (etoposide + cisplatin) with OR 0.83 and 95%CI 0.63 to 1.09, but myelo-suppression such as leucopenia and thrombopenia was more severe with TP regimen; the response rate of monotherapy with topotecan was similar with that of CE (carboplatin + etoposide) regimen with OR 0.59 and 95%CI 0.22 to 1.60; the response rate of TEP (topotecan + etoposide + cisplatin) regimen was comparable with that of EP regimen with OR 1.37 and 95%CI 0.82 to –2.28, but myelosuppression and anemia were more severe with TEP regimen; the response rate with OR 0.97 and 95%CI 0.60 to –1.57, median time to progression with WMD –2.32 and 95%CI –5.72 to 1.09 and median survival time with WMD –1.65 and 95%CI –7.13 to 3.83 of IV topotecan were similar to those of oral topotecan, while neutropenia was more severe with IV topotecan.

Meta分析结果表明,TP 方案与EP方案的反应率相似 [OR 0.83, 95%CI (0.63,1.09)],但具有相对高的致血小板下降的骨髓毒性;单药拓朴替康与CE方案的反应率相似 [OR 0.59, 95%CI (0.22,1.60)];TEP方案(拓扑替康+足叶乙甙+顺铂)与EP方案的反应率相似 [OR 1.37, 95%CI (0.82,2.28)],TEP方案致化疗后重度白细胞下降、重度血小板下降、重度血红蛋白下降均高于EP方案;口服拓扑替康与静脉滴注拓扑替康的化疗后反应率 [OR 0.97, 95%CI (0.60,1.57)]、中位疾病进展期 [WMD –2.32, 95%CI (–5.72, 1.09)]、中位生存期 [WMD –1.65, 95%CI (–7.13,3.83)] 相似,口服拓扑替康化疗后重度中性粒细胞下降明显低于静脉滴注拓扑替康。

Meta-analysis based on included studies showed that response rate of TP regimen has no statistic significance compared with EP regimen[OR0.83,95%CI(0.63-1.090)],but myelo-suppression such as leucopenia and thrombopenia is more severe; response rate of single topotecan has no statistic significance compared with CE regimen[OR0.59,95%CI(0.22-1.60)]; response rate of TEP regimen has no statistic significance compared with EP regimen [OR1.37, 95%CI(0.82-2.28)], but myelo-suppression such as leucopenia, thrombopenia and anemia is more severe; response rate of IV topotecan has no statistic significance compared with Oral topotecan[OR0.97, 95%CI(0.60-1.57)],so as median time to progression[WMD-2.32, 95%CI(-5.72,1.09)] and median survival time[WMD-1.65, 95%CI(-7.13,3.83)],while neutropenia is more sever in IV topotecan than Oral topotecan.

分析表明,TP方案与EP方案的反应率相似[OR0.83,95%CI(0.63-1.090)],但具有相对高的致白细胞和血小板下降的骨髓毒性;单药拓扑替康与CE方案的反应率相似[OR0.59,95%CI(0.22-1.60)];TEP方案与EP方案的反应率相似[OR1.37,95%CI(0.82-2.28)],TEP方案致化疗后重度白细胞下降、重度血小板下降、重度血红蛋白下降均高于EP方案;口服拓扑替康与静脉滴注拓扑替康的化疗后反应率[OR0.97,95%CI(0.60-1.57)]、中位疾病进展期[WMD-2.32,95%CI(-5.72,1.09)]、中位生存期[WMD-1.65,95%CI(-7.13,3.83)]相似,口服拓扑替康化疗后重度中性粒细胞下降明显低于静脉滴注拓扑替康。

Thrombospondin-1 (TSP-1) is a large, trimeric, modular glycoprotein that is a major constituent of platelet a granules.

血小板反应素1(Thrombospondin-1,TSP-1)是同源三聚体糖蛋白和血小板a颗粒的主要组成部分。

Objective:To study the dynamic changes of VWF and GMP-140 blood plasma levels in different phases of disease and their significance of suppressing bleeding and activizing Platelet in patients with Hemorragic Fever with Renal Syndrome.

目的 探讨肾综合征出血热患者抗血管性假血友病因子、血小板α-颗粒膜蛋白(GMP-140)在HFRS各病期中的动态变化以及在止血,血小板活化中的意义。

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