蛋白尿

Using bubble oxygenator, ECC time more than 180minutes , pressure before the artery filter higher than 240 mmHg and haematocrit in the first blood gas analysis more than 27% were significantly different.

ECC血红蛋白尿的产生是一种较为常见的并发症，它与ECC用品，ECC时间，患者自身状态等多种因素有关。

The combination of ARBs and ACE inhibitors further reduced proteinuria more than either agent alone.

ARB和ACEI的联合使用进一步降低了蛋白尿，超过了单一使用他们。

The next question is according to your study, are there any differences in the effects on albuminuria in response to ARBs in type 1 diabetes and type 2 diabetes?

国际糖尿病》：下一个问题是根据您的研究，1型和2型糖尿病中白蛋白尿对ARB治疗的反应有什么不同吗？

The pathogen of gas gangrene mainly exist and live in local place, and seldom enter blood system to cause blood poisoning, but than can form lots of toxins, this toxins can harm blood system, kidney system, and also the tissue can putrescence and the wound will enlarge step by step, all tissue putrescence and toxin can make these bacteria easier to live and reproduce, all these situation if not being control in time will lead a serious situation that multiple organs dysfunction and then died

气性坏疽的病原菌主要在伤口内生长繁殖很少侵入血液循环引起败血症。产气夹膜杆菌产生α毒素、胶原酶透明质酸酶、溶纤维酶和脱氧核糖核酸酶等，红细胞破坏引起溶血血红蛋白尿、尿少、肾组织坏死水肿、液化，肌肉大片坏死使病变迅速扩散、恶化。糖类分解产生大量气体使组织膨胀；蛋白质的分解和明胶的液化，产生硫化氢，使伤口发生恶臭由于局部缺血，血浆渗出，及各种毒素的作用伤口内的组织和肌肉，进一步坏死和腐化，更利于细菌的繁殖使病变更为恶化。

There was increased expression of VEGF in the glomerules,that glomerular immunologic peroxidase anti-Peroxidase staining of VEGF in patients with IgAN.

在轻、中度病变中肾小球内VEGF表达增强，且与24h尿蛋白定量呈正相关，与血压、血肌酐水平无明显相关性，提示VEGF在IgA肾病患者肾组织内表达异常增加，促进蛋白尿形成。

There was decreased expression of VEGF in the glomerules,that glomerular immunologic peroxidase anti-Peroxidase staining of VEGF in patients with IgAN（sclero-sis of IgAN）.

在轻、中度病变中肾小球内VEGF表达增强，且与24h尿蛋白定量呈正相关，与血压、血肌酐水平无明显相关性，提示VEGF在IgA肾病患者肾组织内表达异常增加，促进蛋白尿形成。

Vegf in glomeruli might be involved in the mechanism of proteinuria in preeclampsia.

尿液vegf可能来源于肾脏局部的分泌，肾小球vegf可能参与了先兆子痫中大量蛋白尿的产生。

There is a hypothesis that paroxysmal nocturnal hemoglobinuria hematopoitic stem cells are resistant to the cytotoxic effect of T cells because they lack glycosyl phosphatidylinositol2linked proteins.

核心提示：摘要阵发性睡眠性血红蛋白尿症克隆可能由于缺乏GPI 锚连蛋白而逃脱免疫攻击，形成生长优势。

The effect of 1,25-2D3 is in relieving haematuria and proteinuria and up-regulate the expression of nephrin mRNA and protein to protein kidney.

1，25-2D3可减轻血尿、蛋白尿，上调Nephrin mRNA及蛋白质的表达而有肾保护作用。

Groups were divided as follows: diabetic group, 1,25-2D3 treated group [1,25-2D3 group] and control group. At the 12 week, haematuria and proteinuria were determined and the kidneys were removed by histopathology, immunostaining, and the expression of Nephrin mRNA and protein in renal cortex were detected by reversed transcription-polymerase chain reaction.

建立糖尿病大鼠模型，实验12周末观察各组血尿、蛋白尿、肾组织光镜电镜、免疫组织化学，采用反转录聚合酶链反应检测其肾皮质Nephrin mRNA及蛋白的表达。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。