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蛋白尿

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The patients with or without MAU were divided into two groups:MAU group and NMAU group.

原发性高血压患者存在胰岛素抵抗,微量白蛋白尿和胰岛素抵抗均为心血管疾病的独立危险因素,EH患者出现 MAU是肾脏受损的早期指标。

Also, myotonia, myokymia, and myoglobinuria have been described with systemic illnesses such as malignant hyperthermia, acute renal failure, and compartment syndrome.

另外,诸如恶性高热症、急性肾功能衰竭、筋膜室综合征/室隔综合征等全身性疾病也可导致肌强直、肌纤维颤搐、肌红蛋白尿的发生。

Also, the combination therapy was associated with a significant further decrease in proteinuria in diabetic as well as nondiabetic patients.

并且,两药的联合使用显著降低了病人的蛋白尿,无论是糖尿病病人还是非糖尿病病人。

DM is the most common cause of end stage renal disease, and microalbuminurea incidence increases in DM patients, compared with nondiabetic patients.

糖尿病是导致终末期肾病的最常见原因,且在糖尿病患者中,微量蛋白尿的发病率比非糖尿病患者高。

Results: No protein peak was detected in thirty urine samples of the control group, hut protein peaks were detected in seventy urine samples of the case group with the exception of two and among them 16 were selective proteinuria, 50 were nonselective proteinuria and 2 were overflow proteinuria.

用微流控芯片电泳对30例尿蛋白定性试验为阴性的对照组和70例尿蛋白定性在++以上的病例组进行检测,以白球蛋白的峰面积比值判断蛋白尿的选择性,并与美国Helena琼脂糖凝胶电泳结果比较。

Results:No protein peak was detected in thirty urine samples of the control gruop, but protein peaks were detected in seventy urine samples of the case group with the exception of two and among them 16 were selective proteinuria, 50 were nonselective proteinuria and 2 were overflow proteinuria.

用微流控芯片电泳对30例尿蛋白定性试验为阴性的对照组和70例尿蛋白定性在++以上的病例组进行检测,以白球蛋白的峰面积比值判断蛋白尿的选择性,并与美国Helena琼脂糖凝胶电泳结果比较。

Benazepril is effective in decreasing AER in normotensive and early diabetic nephropathy patients in protecting the renal function.

贝那普利对血压正常伴有微量白蛋白尿的早期糖尿病患者减少尿蛋白和保护肾功能有良好的效果。

Result There were 70 of paraplegia patients, including 39 of hypertension and 31 of non-hypertension.

1.2.2肾脏损害的诊断标准[3]:肾脏结构和功能的异常,伴或不伴肾小球滤过率的降低,观察指标包括尿或血液组分的异常,或影像学的检查的异常,蛋白尿是肾脏损害的独立观测指标。

The clinical and pathologic data of 20 children with simple proteinuria were analyzed.

对20例单纯性蛋白尿的临床和病理资料进行分析。

In the study, Pristane was been used with recombinant BLyS and BLC vector those can expressed in vivo in mice, the influence to SLE-like disease was investigate. Pristane associated with BLyS or BLC aggravates proteinuria. This fact indicates that the abnormal expression of the two genes has definite effect on mice lupus-like disease process.

本研究中,当Pristane与可在体内表达的重组BLyS或BLC基因质粒合用时,小鼠的蛋白尿明显加重,表明这两个基因的异常表达对小鼠的狼疮样病变过程有明显的影响。

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