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Humanm Seroalbumin ,Albumin,Human Serum Albumin

别名: 血清白蛋白,人血白蛋白,白蛋白

The apolipoproteins on the surface of the lipoproteins help to solubilize the lipids and target the lipoproteins to the correct tissues .

在脂蛋白表面的载脂蛋白有助于溶解脂质,并把脂蛋白导向适当的靶组织。

The rule of growth and decline about main biochemical material (solubility protein, solubility sugar, SOD and gossypol IAA) contacting with stomatic embryogenesis showed that content of solubility protein and SOD activity was differed in the different period, being lower in earlier callus and reaching maximum in 120 day.

对陆地棉愈伤组织诱导及体细胞胚发生过程中的主要生化物质(可溶性蛋白含量、可溶性糖含量、SOD活性和IAA氧化酶活性)的测定表明,各个时期的可溶性蛋白含量、SOD活性是不同的,在愈伤组织形成初期较低,随着愈伤组织的生长及胚状体的形成,可溶性蛋白含量、SOD活性逐渐上升,在120d时达最大值;而可溶性糖含量在60d时有一个高峰值,在120d时达到最低值;IAA氧化酶活性在60d、120d时酶活较高,90d的酶活较低。

In this study, the deletion mutants of SulA with regard to C-terminus were constructed and the interaction with ClpY was analyzed in yeast two-hybrid system.

本实验中,为确认ClpYQ蛋白酶辨认SulA蛋白的区域,建构SulA之C-端不同大小片段缺失的突变蛋白,以酵母菌双杂交系统测试,各个SulA突变蛋白与ClpY之间交互作用的情形,发现ClpY辨识的区域可能位於C-端的高疏水性片段,C-端第20 - 30个胺基酸。

The results showed that Tang-an-kang could respond symptomatically, lower blood-sugar, diminish urinary protein, regulate endogenous creatinine clearance rate and lipid metabolism etc. It was suggested that Tang-an-kang had a good action on improvement lipid metabolism, diminishing urinary protein and renal function protection.

结果表明:糖安康治疗组在改善DN患者临床症状,降低血糖、糖基化血红蛋白及血肌酐,减少尿蛋白及尿白蛋白,调节内生肌酐清除率,降低血与尿β2微球蛋白,调节脂代谢,改善血液流变性等方面,疗效均优于单用西药的对照组,提示糖安康有良好改善糖脂代谢、减少尿蛋白、保护肾功能的作用。

The protein had the same antigenicity as IgG, and the change of the protein had the same tendency as SHP-2, which was higher in canceration groups than in uncanceration groups and control groups in thymus cells, but there weren't significant change in bone marrow cells;⑧ The change law of the expression of SHP-1 and SHP-2 during γ-ray induced malignant transformation in BEAS-2B cell is that the expression of SHP-1 was increased synchronistically with malignant transformation of cell, and the expression of SHP-2 didn't have significant correlation with malignant transformation of cell.

癌变组胸腺细胞中SHP-2 mRNA编码PTPase催化区的700~1096bp间可能出现甲基化;⑦癌变组的胸腺细胞中存在一相对分子质量约为55×10〓的蛋白质,它与IgG具有相同的抗原性,其表达量的变化趋势与SHP-2的蛋白表达变化趋势基本一致,即在胸腺细胞中明显高于对照组及未癌变组,但在骨髓细胞中对照组高于癌变组;⑧SHP-1、SHP-2在γ射线诱发的BEAS-2B细胞恶性转化过程的变化规律:SHP-1蛋白表达量的升高与细胞的恶性转化在时间上基本一致,而SHP-2蛋白表达量与恶性转化无明显关系。

The binding behavior and kinetics of uncoupling protein with its ligand, the Fourier-transformation infrared spectra of uncoupling protein and its changes during ligand-binding process, the inhibition of Guanosine 5'-Tetraphosphate (G-4-P) and Adenine-β-Darabinofuranoside 5'-triphosphate on the binding of uncoupling protein with GTP were studied.

本文对解偶联蛋白与配基的结合及其结合动力学,解偶联蛋白及其与配基结合过程中的傅里叶变换红外光谱及其光谱变化,鸟嘌呤核苷四磷酸(G-4-P)和9-β-D-阿拉伯糖基呋喃5'-三磷酸对解偶联蛋白与GTP结合的影响进行了研究。

During the past 3 years, 2 independent experiments of sperm-mediated gene transfer in domestic silkworms were carried out respectively in 2000 and 2002 with the method direct injection of copulation thecae.

在同源重组绿色荧光蛋白表达载体pG350的基础上,以pMD18-T为质粒载体,用egfp基因替换gfp基因,构建了家蚕核型多角体病毒早期即刻蛋白基因启动子BmNPV-IE控制下的增强型绿色荧光蛋白表达载体pMD-Fib-IE-EGFP。

Reactive oxygen species causing DNA oxidative damage comes from two kinds of ways:one is from cellular normal physiological metabolism;the other is from outer environment.Redox-sensitive green fluorescent protein was expressed in Saccharomyces cerevisiae.Recombinant cells were evaluated in monitoring the changes in the redox state of living cells when challenged with toxicologically relevant metal ions NaAsO_2 or Pb(NO_3)_2 by measuring emission intensity at 510 nm with a Hitachi F6500 fluorescence spectrophotometer,roGFP expressed in yeast responded not only to typical membrane-permeant oxidants H_2O_2 and reductants DTT,but also to toxicological metal ion-induced intracellular redox changes in a dose-dependent manner.Moreover,exposure of yeast cells to NaAsO_2 or Pb(NO_3)_2 at concentrations that induced redox changes reported by roGFP caused up to 2~3 fold increases in DNA mutation frequency.This mutagenic effect was largely caused by oxidative stress since blocking the production of hydryl radicals with thiourea significantly reduced the mutation rate as well as delayed the cell death.

本文将对氧化还原状态变化敏感的绿色荧光蛋白roGFP1-R12,在酵母细胞中实现了多拷贝强表达;荧光扫描经强氧化剂H_2O_2和还原剂DTT以及环境中重金属NaAsO_2或Pb(NO_3)_2处理后的酵母细胞悬液,测定510 nm处的荧光发射强度结果显示,表达的绿色荧光蛋白对氧化还原水平敏感,且在510 nm处的荧光强度与一定的重金属浓度呈正相关,即roGFP1-R12在510nm处的荧光发射值随重金属浓度的增高而增强,从而说明重金属对细胞的毒性在一定程度上很可能是通过破坏细胞内的氧化还原平衡发生作用;同时通过该绿色荧光蛋白对胞内氧化还原状态变化的响应情况可以来实时检测环境中的重金属;遗传学的点突变频率及致死率实验数据表明,重金属能导致菌体的点突变频率和致死率升高,且活性氧的清除剂巯基脲能明显降低这种点突变和致死率,说明由重金属引发的这种点突变和致死效应在很大程度上是依赖于重金属对细胞诱导产生的氧化胁迫。

Differentially expressed protein spots were identified by Tandem MS as RNA binding protein regulatory subunit, Proteasome subunit beta type 7 precursor, and Translationally controlled tumor protein.

进一步选择了3个组间差异表达蛋白点进行串联质谱分析,分别鉴定为RNA结合蛋白调节亚基、蛋白酶体β亚基7型前体和翻译调控肿瘤蛋白

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I can not make it blossom and suits me

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When temperatures are above approximately 80 °C discolouration of the raceways or rolling elements is a frequent feature.

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The lawyer's case blew up because he had no proof.

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