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Aliquots were subjected to impedance aggregometryin response to collagen, adenosine diphosphate, ristocetin,and arachidonic acid performed at 22°C, 34°C, 37°C,and 40°C.

一部分用作在22°C 、34°C 、37°C 和40°C 下测定对胶原、二磷酸腺苷、瑞斯西丁素和花生四烯酸反应中的阻抗集合度测试。

There is an apparent dissociation of β 2 AR induced augmentations of the intracellular Ca 2+ transient and contractility from cAMP production and PKA dependent cytoplasmic protein phosphorylation.

受体激活所引起的细胞内钙和收缩力的增加与细胞内环磷酸腺苷的增加和胞浆蛋白磷酸化有明显的脱节。

Adenosine of annulus phosphoric acid and phosphorous ester inositol serves as the 2nd messenger, participate in nerve to give qualitative signal to conduct.

环磷酸腺苷和磷酯肌醇作为第二信使,参与神经递质的信号传导。

Results 5'-nucleotidase significantly inhibited platelet aggregations induced by ADP, AA and PAF in a dose-dependent manner.

结果 白唇竹叶青蛇毒5'-核苷酸酶能抑制ADP、AA、PAF诱导的血小板聚集,且其抑制率与剂量增加成正比;抑制血小板聚集作用与ADP的水解和腺苷的积累有关。

The coronary flow,troponin-T and adenosine of effuent,ecto-5′-nucleotidase activity and MDA in myocardial tissue were measured.

应用兔离体心肌缺血再灌注损伤模型,在Thomas 液中加入黄芪注射液,检测冠脉流量、冠脉回流液中肌钙蛋白-T和腺苷释放、心肌组织中的MDA及外部-5′-核苷酸酶的活性。

Methods Platelet aggregations induced by ADP, AA and PAF, respectively, were measured by turbidimetric method after platelet incubated with 5'-nucleotidase.

采用比浊法测定白唇竹叶青蛇毒5'-核苷酸酶对二磷酸腺苷、花生四烯酸、血小板活化因子诱导血小板聚集的影响。

Washed platelet inhibition experiment and the effect of Adenosine, CP/CPK on platelet aggregations showed that platelet aggregations inhibited by 5'-nucleotidase were associated with ADP hydrolysis and adenosine accumulation.

白唇竹叶青蛇毒5'-核苷酸酶能抑制血小板聚集,抑制作用主要通过水解ADP和积累腺苷,可能与阻断TXA2的生成有关。

ATP is the primary source of energy for all cellular processes.

三磷酸腺苷是所有细胞活动的主要能量来源。

Moreover, Fyn kinase directly phosphorylated LKB1 on tyrosine 261 and 365 residues, and mutations of these sites resulted in LKB1 export into the cytoplasm and increased AMPK phosphorylation.

这是&能量主开关&磷酸化腺苷酸活化蛋白激酶在它们脂肪和肌肉组织中含量较少的结果。这些发现显示,这种酶有可能帮助开发一种减肥新药。

Studies in neuropsychopharmacology and molecular neurobiology indicate that neurotransmitters and its receptors play important roles in alcohol abuse and addiction, and post-receptor signal transduction pathways, including cyclic adenosine 3', 5'-monophosphate-protein kinase A, phosphoinositide, Ca(superscript 2+)-calmodulin, phospholipase D and tyrosine kinase Fyn signaling cascade.

神经精神药理学和神经分子生物学研究表明,酒精滥用和成瘾不仅与中枢神经系统的神经递质及其相关的受体有关,而且,受体后的信号转导通路参与了酒精依赖的形成过程。其中包括环磷酸腺苷-蛋白激酶A、磷脂酰肌醇、Ca(上标 2+)-钙调蛋白、磷脂酶D以及酪氨酸激酶Fyn等信号途径。

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你知道吗,我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

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Chapter Three: Type classification of DE structure in Sino-Tibetan languages.

第三章汉藏语&的&字结构的类型划分。