脾的

SAN DIEGO – Untreated patients with type 1 Gaucher disease face a high risk of avascular necrosis during their lifetime, and the risk is greatly increased in untreated splenectomized patients.

圣地亚哥——未经治疗的1型戈谢病患者一生中罹患非血管性坏死的风险增加，在未经治疗的脾切除患者中风险尤其高。

The enzymes from pig allantoic fluid , bovine spleen and red kidney bean were well studied.

从猪的尿囊液、牛脾和红蚕豆中提取出的紫色酸性磷酸酶得到了充分的研究。

In the whole rats, the following tissues or organs were extracted and fixed in the most appropriated fixator (Bouin liquid or Formalin at 10%) then included in paraffin blocks : all the organs showing significant macroscopic lesions, encephalon (brain hemisphere, cerebellum and annular protuberance), spinal cord, sciatic nerve, salivary glands, esophagus, stomach, duodenum, jejunum, ileon, caecum and colon, rectum, liver, spleen, bone marrow, lymph node close to and at distance, thymus, heart, aorta, trachea, lungs, thyroid/parathyroid, surrenals, pancreas, hypophysis, kidney, bladder, gonad, uterus, mammary glands, epididymis, prostate, eyes, lachrymal glands, bones, skin, thigh muscle and implantation sites (2 per animal).

从整体大鼠内，下列组织或器官提取后用最适宜的固化剂（ Bouin 液或10%的甲醛溶液）进行固定，然后包入石蜡块内：所有显示有明显肉眼可见损害的器官、脑髓、脊髓、坐骨神经、唾液腺、食道、胃、十二指肠、空肠、回肠、盲肠、大肠、直肠、肝脏、脾、骨髓、近端和远端淋巴结、胸腺、心脏、主动脉、气管、肺、甲状腺/甲状旁腺、肾上腺、胰腺、垂体、肾、膀胱、性腺、子宫、乳腺、附睾、前列腺、眼睛、泪腺、骨骼、皮肤、股肌以及假体植入部位（每只动物2处）。

In order to evaluate our improved over-fatigue model, we compare it with rhubarb laxation SAS model and over-swimming SAS model, and proved by Sijunzi decoction. Moreover, we elementarily studied the mechanism of the changes in digestion and absorption function by improve over-fatigue method.

通过与大黄泻下法、力竭游泳法塑造的脾虚证动物模型比较、药物反证等几方面来评估和验证此方法的成立，并对其影响动物消化系统功能的机制作了初步探讨。

Common clinical characteristics included marked neoplastic lymphocytosis, systemic lymphadenopathy, splenomegaly, and bone marrow involvement.

这5例共同的临床特征包括显著的肿瘤性淋巴细胞增生，系统性的淋巴结病、脾肿大及骨髓受累。

The experimental study of HSC research has been accelerated by Till and McCulloch who founded the technique of spleen colony formation from HSC.

在造血干细胞的实验研究中，Till和McCulloch建立的脾结节实验技术起了重要的推动作用。

Hemolytic anemia, enlargement of the spleen, and pathologic changes in the liver, kidneys , and heart muscle may occur as secondary effects of methemoglobinemia, which is reversible.

作为高铁血红蛋白症的次要影响，会发生溶血性贫血、脾扩大和肝、肾、心肌的病变，这些影响是不可逆转的。

The diagnosis of ITP is made clinically by exclusion of other causes of thrombocytopenia, the absence of splenomegaly, and the finding of normal or increased megakaryopoiesis on bone marrow examination. The nonimmune thrombocytopenia is caused by some nonimmune factors, such as acute leukemia, hypersplenism and post-chemotherapic bone marrow suppression.

特发性血小板减少性紫癜的诊断仍只是一种排除性的临床诊断，其国内诊断标准为多次化验检查血小板计数减少，脾脏不增大或仅轻度增大，骨髓检查巨核细胞数增多或正常，有成熟障碍，并且具备以下五点中的任何一点：泼尼松治疗有效，切脾治疗有效，血小板相关抗体增多，血小板相关补体增多，血小板寿命测定缩短，同时应排除继发性血小板减少症。

This paper presented histopathology of the syndromes between parotitis and putrid skin disease of Soft shelled Turtle in comparison with health individuals.

对鳃腺炎与腐皮病并发症的鳖的肝、脾、肾、肺、肠和精巢等 6种器官进行病理组织学切片，并与健康鳖的相应器官组织进行比较。

Results The change on morphous,size,liver lobe proportion,parenchyma density and portal vein in decompensation phase of liver cirrhosis was often observed,while splenectasis,collateral circulation construction,abnormality perfuse,ascitic fluid and pleural fluid also found in spiral CT scanning of liver cirrhosis.

结果 肝硬化在失代偿期CT表现有明显异常，肝脏有不同程度的形态、大小及肝叶比例改变，肝脏表面的实质密度、门静脉也有改变，并且常伴有侧支循环的建立、肝脏异常灌注、脾增大、胸腹水等。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。