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Acellular allogeneic dermal matrix is the dermal replacement of heterogenous human skin in which the cellular elements have been removed by special technique and basement membrane complex have been remained. It becomes a kind of 3D porous acellular dermal scaffold. It supports fibroblast infiltration, neovascularization and epithelialization in the absence of an inflammatory response.

异体脱细胞真皮基质(acellular allogeneic dermal matrix, ADM)来源于正常的异体皮肤组织,经过特殊的理化处理去除真皮中的细胞成分,保留原有的胶原三维结构和完整的基底膜,成为一种多孔的三维膜性支架,其空间网状结构有利于细胞的增殖、毛细血管增生,加速组织的愈合过程,移植后可以迅速被血管化和自体细胞重建。

Methods Rabbit pulmonary alveolar macrophages were cultured in vitro. The cell death rate, the malandialdehyde, lactate dehydrogenate and superoxide dismitase contents in the cultured cells were determined at different incubation time and different dose of chrysotile asbestos for the cytotoxicity of the material.

采用体外细胞培养技术,以兔肺泡巨噬细胞死亡率、乳酸脱氢酶、超氧化物歧化酶活力及丙二醛含量变化来表示不同剂量、不同培养时间的纤状蛇纹石石棉对巨噬细胞的毒性。

Description: The bioactive factors can rapidly eliminate allergy, soothe the skin, improve the immunity of skin, repair damaged skin, promote the collagen synthesis of fibrocyte of skin tissue, improve the blood circulation of skin tissue, and reduce the release of cell histamine of skin. They have the efficacy of desensitization, can replenish water and nutrients, and add a natural barrier to sensitive skin.

产品说明:通过生物活性因子快速消除过敏现象,舒缓镇静皮肤,提升肌肤免疫力、修复受损细胞,促进皮肤组织纤维细胞的胶原合成,改善皮肤组织的血液循环并减少皮肤细胞胺释放,脱敏同时大量补充肌肤水份及营养给敏感性肌肤增加一道天然屏障。

DNA damage did not increase levels of the amiloride-sensitive sodium-hydrogen exchanger isoform 1 (NHE-1), intracellular pH, Bcl-x deamidation, and apoptosis.

DNA损伤并不增加细胞的阿米洛利敏感的钠-氢交换蛋白-1 (NHE-1)、细胞内pH值、Bcl -×脱酰胺作用及细胞凋亡水平。

The place and time of cell proliferation are just in accordance with those of the appearances of stem cells and stem-cell blastemas, which means that cells in those areas mentioned above dedifferentiate forming stem cells which proliferate too.

这些分裂细胞在位置和时间上与干细胞及干细胞原基的出现一一对应,表明上述几处发生了细胞的脱分化,形成了干细胞,进行着细胞增殖。

While the tissue spaces surrounding a few blood vessels wasAl and Fg positive,no Al or Fg positive cells were observed.In antemortem injurygroup,diffuse subarachnoid hemorrhage,cerebral edema,swelling or pyknotic neu-rons could be observed.The axons showed irregular swelling and disconnection at1~3h,marked swelling and disconnection at 6h,and retraction ball at 15h whichwas more remarkable at 24h after injury.The space between myelin sheaths andaxons was increased at 3~6h after injury.Tortuous and wavelike myelin sheathswhich adhered on axons incompletely,or even peeled off could be found from 15hto 24h after injury.Perinuclear lysis of Nissl bodies began at 24h after injury.Thenumber of GFAP positive cells in cerebrum and brain-stem increased significantlyfollowed by decrease,and then increased again,but the time courses of the changesin different areas of brain were not same.Al and Fg positive neural cells,mainlysurrounded blood vessels,with diffuse or peripherally distributed positive matter incytoplasm could be observed at 0.5h after injury.The number of Al or Fg positivecells and the intensity of immunoreaction increased with the time of injury.The areaof SYN positivity in medulla oblongata and pons decreased notably 3~6h afterinjury,then return to normal levels and continued to 24h after injury.

生前损伤组,可见广泛蛛网膜下腔出血,脑组织水肿,神经细胞肿胀,晚期神经元固缩;伤后1~3h见部分神经轴突不规则增粗、断裂,伤后6h断端膨大,伤后15h可见收缩球,至伤后24h更为明显;伤后3~6h可见部分神经髓鞘与轴突之间的间隙增宽,伤后15h髓鞘明显曲折,不完全附着在轴突两侧,甚至剥脱,持续到伤后24h;核周尼氏体减少在伤后24h才开始出现;同一部位的GFAP阳性细胞数目随损伤时间发生改变,先增多(最早在伤后0.5h),达到高峰后减少,其后又有增多趋势,但不同部位的GFAP阳性细胞数目增减的时间过程不尽相同,同时,大脑中的GFAP阳性细胞数目也有改变;伤后0.5h,可在脑干组织中见到Al和Fg阳性神经细胞,主要位于血管周围,阳性物在胞浆中呈弥散性分布,但部分细胞的阳性物仅分布于靠近胞膜的胞浆中而呈环状,随损伤时间延长,阳性细胞数目增多,反应强度增加;伤后3~6h,延髓及桥脑中的SYN阳性物面积减少,其后恢复到正常水平,并持续到伤后24h。

RESULTS: APC promoter 1A was methylated in NCI-H460 cells, and unmethylated in NCI-H446 and SPC-A1 cells. Hypermethylation was detected in all 5 CpG islands (687, 707, 714, 719, 726) of APC promoter 1A in NCI-H460 cells. The expression of APC in NCI-H460 cells was decreased by 26.04% of that in NCI-H446 cells and by 32.36% of that in SPCA1 cells. After treatment of 1, 5, 10, 15μmol/L 5-aza-dC, the expression of APC promoter 1A in NCI-H460 cells was enhanced by 4.59, 5.78, 9.58, 5.98 folds, respectively.

结果:SPC-A1和NCI-H446细胞APC甲基化阴性,NCI-H460细胞APC甲基化阳性;甲基化芯片检测Ncl-H460细胞在APC启动子1A 5个CpG位点均存在甲基化(687、707、714、719、726),SPC-A1和NCI-H446甲基化阴性,荧光定量结果NCI-H460的APC转录较SPC-A1和NCI-H446有明显的下降,仅为二者平均的30.04%;经5-aza-dC脱甲基化作用后,NCI-H460细胞的APC表达增加了约5~10倍,其中10μmol/L浓度作用下,APC表达增加最多。

But the superficial epithelium was partially keratinized. Conclusion Tissue engineered gingiva may be constructed with human ginginval epithelia and ADM in vitro.

利用体外培养的牙龈上皮细胞和脱细胞真皮,可以在体外联合构建具有复层上皮细胞结构的组织工程牙龈。

With the existence of additional carbon sources, sodium lactate, sodium propionate or sodium acetate, the faster pH rised, the faster p, p'-DDT dechlorinated, which indicate that the existence of short chain organic substrate supplies sufficient electron donors for the dechlorination process, and p, p'-DDT can obtain electrons more easily during the stage of pH rising.

并且由于p,p'-DDT的还原脱氯与细胞色素氧化酶及辅基FAD有关,外加碳源下p,p'-DDT的缺氧降解多数发生在体系pH值上升的阶段,且pH值上升较快时对应的p,p'-DDT缺氧降解较快,而在无外加碳源的体系中,由于可供p,p'-DDT还原脱氯的电子量限制了p,p'-DDT还原脱氯的速度,未发现上述现象。

objectiveto investigate the effects of yuegan capsule on ccl4 intoxicated primary cultured rat hepatocytes.methodshepatocytes injury models were induced by ccl4 in rats in vitro, and treated with yuegan capsule.structure of rat hepatocytes and effect of sdh、acpase、g 6 pase activeties and amount of hepatic glycogen were observed.

目的探讨悦肝胶囊对四氯化碳(ccl4)损伤原代培养大鼠肝细胞的保护作用。方法以ccl4诱导大鼠肝细胞损伤模型,观察悦肝胶囊对肝细胞的琥珀酸脱氢酶、酸性磷酸酶、葡萄糖-6-磷酸酶(g 6 pase)活性和糖原含量以及肝细胞形态结构的影响。

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We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称;三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起,称之为挽歌体诗人或者史诗诗人,他们被称为诗人,似乎只是因为遵守格律写作,而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。

This is not paper type of business,it's people business,with such huge money involved.

这不是纸上谈兵式的交易,这是人与人的业务,而且涉及金额巨大。