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MethodsThe model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed.

方法采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响;TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

Methods The model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed.

采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的

Methods The model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed. The content of maiondialdehyde,activity of superoxide dismutasein the brain tissues of cerebral ischemia and anoxia mice were measured by TBA,NBT technology respectively.

采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响;TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

Relative content of-Anethole was increased accompanying increased duration, while Fenchone was decreased.

为 0.92 ml·(100g)-1。精油主要成分反式茴香脑的相对含量随着蒸馏时间的延长而不断上升,小茴香酮的相对含量都随着蒸馏时间的延长而不断下降。

MethodsThe model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed.

方法采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响

Methods The model of cerebral ischemia and anoxia in mice was established by decollation and ligating common bicarotid artery the effect of BVF on the survival time of the mice,and the time of catching their breath after mice decollated,and mouse brain capillary vessel permeability were observed.

采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响;TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

ResultsBVF could remarkably lengthen the survival time of cerebral ischemia and anoxia mice, and the time of catching their breath after mice decollated,could evidently decrease mouse brain capillary vessel permeability and the content of MDA, increase activity of SOD in the brain tissues of cerebral ischemia and anoxia mice.

方法采用小鼠断头、结扎双侧颈总动脉等脑缺血缺氧模型,观察苦菜总黄酮对脑缺血缺氧小鼠存活时间、断头小鼠喘息时间及脑毛细血管通透的影响;TBA法、NBT法测定小鼠脑缺血缺氧后脑组织MDA含量和SOD活性。

Objective:To observe the effects of Naloxone and Nikethamide on chronic respiratory failure with pulmonary encephalopathy.

目的 :观察常规治疗的同时加用纳络酮(Naloxone,NLX)治疗慢性Ⅱ型呼吸衰竭肺性脑病的疗效。

Result:The sterandryl substitution group and the vacation surgery group comparatively castrates the group brain stem dead volume obvious increase; The Bax protein expression masculine cell number expression also increases.

线栓法建立大鼠局灶性脑缺血再灌注损伤模型,TTC染色测脑梗死体积及免疫组化染色检测脑组织Bax的表达。结果:丙酸睾酮替代组和假手术组较去势组脑梗死体积明显增加;Bax蛋白表达阳性细胞数表达也增加。

Naloxone can decrease the brain edemas of rats with traumatic brain injury, promote the nervous function recovery, and the treatment effect changes with the dosage during some range.

盐酸纳洛酮治疗组大鼠的脑含水量明显低于对照组(P<0.05);盐酸纳洛酮内部各实验组之间,0.3mg/kg组的脑含水量高于其他三组(P<0.05),其他三组之间无显著性差异(P>0.05)。

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