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Methods Plasma IL-8 levels in 30 ACD patients(patients with acute 40 cerebral hemorrhage, 32 patients with acute cerebral infarction)and in 30 healthy subjects were measured by ELISA.

采用ELISA方法测定了74例急性脑血管病患者(40例脑出血,34例脑梗死)和30例健康对照者的血清IL-8含量。

Rt-PA(5mg/kg) dissolve bolt gives treatment after the 3h that be short of blood, associated group gives vein injection at the same time Yidala is acted according to (the 24h after 3mg/kg) dissolve bolt undertakes nerve function grading and cerebral stalk die concentration percentage by volume leads an analysis, determine blue content of the thread of ideas in writing of the Yi in cerebral tissue, with immune organization chemical method evaluates dissolve bolt afterbrain to organize the change of hematic head protective screen.

缺血3h后给予rt-PA(5mg/kg)溶栓治疗,联合组同时给予静脉注射依达拉奉(3mg/kg)溶栓后24h进行神经功能评分和脑梗死体积百分率分析,测定脑组织中伊文思蓝含量,用免疫组织化学方法评估溶栓后脑组织血脑屏障的变化。

Objective: To observe the motor function variation of focal cerebral ischemical reperfusion injury rats'limbs at different time. To observe the expression of PCNA and VEGF. To observe the effect to that index when electric acupuncture the"Quchi"and"Housanli". To observe the effect to the focal cerebral ischemical reperfusion injury rats, which are inject eNOS catastaltic, when electric acupuncture that two acupoints. Through observing of above all things , we study acupuncture's blood vessel regenerated effect to cerebral ischemia reperfusion injury region, approach the possibility of brain functional recovery angiogenesis which promoted by acupuncture, provide new consider and experiment accordings for deepening acupuncture therapy of the ischemia stroke.

研究目的:通过观察局灶性脑缺血再灌注损伤大鼠不同时间点肢体运动功能的变化及损伤脑组织核心增长蛋白、血管内皮细胞生长因子的表达情况,电针&曲池&、&后三里&穴对以上各指标的影响、电针&曲池&、&后三里&对给予eNOS抑制剂对局灶性脑缺血再灌注损伤大鼠的影响,研究针刺对大鼠脑缺血再灌注损伤区血管再生的作用,探讨针刺促进脑功能恢复的血管生成可能机制,为进一步深化针灸治疗缺血性中风的认识提供新的思路和实验依据。

Objective To study the protective action of compound agent madeup with effective ingredients of Hirudo and Rhizoma chuanxiong and Folium Ginkgo in animal ischemic cerebral infarction.Methods To mix Hirudin extracts of Folium Ginkgo and Ligustrazine according2:25:5into asepsis injection,rats local cereˉbral ischemia induced by embolism of middle cerebral arterywas used as experimental model to study the efˉfects of the compound agent and positive control drug,Nimodipine and extracts of Folium Ginkgo on cerebral infarct size and neurological deficits.Disposing the experimental data with statisticsand comparing their otherness among difˉferent groups.

目的 探讨用中药水蛭、银杏、川芎的有效成分组成的复方制剂对动物局灶脑缺血的保护作用方法将水蛭素、银杏叶提取物、川芎嗪按2∶25∶5的比例制成无菌注射液,应用鼠大脑中动脉栓塞导致缺血性脑卒中的实验模型,并用生理盐水、尼莫地平和银杏提取物作对照,观察不同药物对脑缺血面积和由缺血造成的大鼠行为障碍的影响,将实验据用统计学处理并进行组间的差异性比较。

The accompanying DVD contains royalty free music, sound effects, and video clips from Artbeats, Digital Juice, TwistedTracks, and the Footage Firm. About the Author Carl Plumer has worked professionally as a writer, Web developer, multimedia designer, Help systems developer, and documentation manager for high tech companies including Oracle?

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Previous studies have identified that understanding other's mind engages a neural circuit consisting of the medial prefrontal cortex, temporoparietal junction, superior temporal sulcus, temporal pole, and precuneus/posterior cingulated cortex. However, due to the limitation of experimental paradigms and neuroimaging data analysis approach, the exact neurocognitive mechanisms that underlie the understanding of others' belief remain unclear.

尽管前人的研究已发现内侧前额叶、颞顶联合皮层、后扣带回/楔前叶以及颞上沟等脑区参与他人信念理解,但是,由于实验范式本身和脑成像数据分析方法的局限性,过去的研究工作没有提出准确的认知神经机制来解释相关脑区组成的神经网络如何协同工作、完成视角转换以保证正确理解他人的信念。

Objective To use an ischemical reperfusion injury rat model by pulsinelli-4-VO andobserve the changes of the expression of Glu, N-methy-D-asparagic acidreceptor subunits NR_1 and NR_(2B) and ability of learning and memory after procerebrum ischemic reperfusion injury, and approach the pathogenesy of learning andmemory disorder induced by ischemic reperfusion injury, and the intervention ofginsenoside Rg_2 on the learning and memory disorder after procerebrumischemic reperfusion injury.

目的利用pulsinelli四血管法体建立脑缺血/再灌注损伤模型,观察前脑缺血损伤后谷氨酸、N-甲基-D-天门冬氨酸受体亚单位NR_1、NR_(2B)表达的变化及学习记忆的改变,从兴奋性氨基酸的兴奋毒作用尤其是其受体的表达方面探讨脑缺血/再灌注损伤引起的学习记忆功能障碍的发生机制,并观察人参皂苷Rg_2对脑缺血缺氧损伤引起的学习记忆障碍的干预机制。

The positive production of ER, NGF and ChAT were detected in cirguli cortex, parieta lobe, frontal lobe, piriformis cortex and nucleus amydgaloideus corticalis, hippocampus gyrus fasciolaris, subiculum, CA2 and CA3, and mainly presented in nucleus medial septal, nucleus lateral septal and nucleus of diagonal band vertical division in septal-diagonal band area.

老年大鼠和去卵巢大鼠间脑内ER、NGF和ChAT的表达强度和阳性细胞数目总趋势是显著降低,而补充外源性雌激素可以阻止其下降,表明雌激素可通过ER促进间脑中NGF和ChAT的表达,雌激素对间脑中神经元结构和功能维持是必须的,而且雌激素对间脑神经元的作用与NGF和ChAT的表达是协同和一致的。

The literature on autopsy studies states no apparent tendency toward these venous channels, and brain stem veins and telencephalic veins are equally affected, although there is a clear-cut predominance of lesions in the brain stem.

尽管脑干部位的小静脉的确最容易受累,但以往的研究文献不但没有发现脑干部位的小静脉管腔特别容易受累,反而认为脑干部位的静脉和端脑部位的静脉受累的几率相同。

The FosB/△FosB positive staining was detected in nucleus accumbens septum, tuberculum olfactorium and dorsal caudate putamen, without little cFos positive staining. The results indicate the expression of cFos and FosB/△FosB display the differences in area and intensity, and the area intensively expressing FosB/△FosB is more nearly coincide with the area related to schizophrenia. So FosB/△FosB is a better mark to research the pathogenesis of schizophrenia.

同时大量研究证明,动物模型的行为及生理变化与中枢神经系统神经元的病理变化有关,因此,利用即早基因反应的灵敏性、功能多样性、检测方便性等,应用免疫组织化学的方法对模型小鼠脑内cFos和FosB/△FosB蛋白的表达进行检测,通过确定不同脑区cFos和FosB/△FosB蛋白的表达,来研究哪些脑区或神经元参与了反应,从而发现精神分裂症在脑区内所涉及的解剖结构,有助于对精神分裂症发病机制的认识。

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Do you know, i need you to come back

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Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

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