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The brain capillary and star glue quality cell which enwrap around it under thenormal condition form the permeation extremely low blood-brain barrier, onlypermit some little molecule to pass; When cerebral isc.

正常状态下的脑毛细血管及包裹在其周围的星形胶质细胞构成了通透性极低的血脑屏障，只允许一些小分子物质通过；当缺血性中风发生时，微血管通透性发生显著改变，这种病理变化与脑内皮细胞损伤导致的功能变化有直接的关联。

Resluts:It has a infection of delayed intracranical heamatoma such as contusion and laceration of brain or contusion and laceration of brain is on the offside of haematoma;a great deal of haematoma in the brain;fracture of skull,especially the fracture of sinus or medium artery of span meningeal,it is the high risk factors of DTIH,and it get high risk of delayde intracranical hematoma;abnormity of clotting time;falling sickness;a grave scalp injury etc.

结果：脑挫裂伤或血肿对侧有脑挫裂伤；脑内血肿量大；颅骨骨折，尤其是跨脑膜中动脉或静脉窦的颅骨骨折是导致外伤性迟发性颅内血肿的高危因素，发生迟发性颅内血肿的危险性大。出凝血时间异常、癫痫发作、重症头皮损伤等对迟发性血肿的发生也起着一定的影响。

Results (1) 6 cases of Tuberous Sclerosis. Diffused subependymal nodular calcification lesions were found in all cases on unenhenced CT. 4 patients are 2 pairs of mother/child relationship. Both of the two mothers are found to suffer from renal angiomyolipoma.(2)1 case of neurofibromatosis showed abnormal spinal canal: scoliosis of thoracic and lumbar spine, concave change of vertebral posterior border. Bilateral renal hypogenesis was found in this patient. Diffused hyper-density lesions were found in kidney and fatty accumulation was found in back skin.(3)6 cases of Sturge-Weber syndrome. On unenhenced CT, curving and strip-shaped calcifications were found along the parietal and occipital gyrus.

结果 ①结节性硬化6例，所有病例CT平扫见两侧脑室室管膜下多发小结节状高密度钙化灶，其中4例为两对母子关系，并见两位母亲合并有肾脏错构瘤，；②神经纤维瘤病1例，MRI表现为椎管异常，胸腰段脊柱侧弯，椎体后缘呈明显的切凹改变；伴有双肾发育不良，CT示肾内多个高密度影，背部皮肤多量脂肪堆积，③脑颜面血管瘤综合征6例， CT可见顶枕部沿脑回分布的弯曲的条状高密度钙化，部分延伸致侧脑室内，增强后见病灶内有扭曲的条状和结节状明显强化的血管影；④小脑血管瘤病4例，影像学表现为小脑内大囊、小结节样占位性病变。

The effects of QDPTP on cerebral blood flow and artery blood pressure in anesthetized rats The regional cerebral blood flow in cerebral pia mater was examined by Laser Doppler Flowmeter and the artery blood pressure (including mean systolic pressure, mean diastolic pressure and mean blood pressure) were monitored by the electrophysiological polygraph before and after drug administration.

结果表明：模型组脑组织及血清IL1-β含量分别为1006.4±247.1 pg/ml和370.8±76.6 pg/ml，与之相比，QDPTP中剂量(550.7±99.8，268±64.6)组中脑组织及血清中IL1-β的含量显著降低(P.01)，川芎嗪脑也明显减少组织IL1-β的含量(627.8±144.7，P.05)。 QDPTP对麻醉大鼠局部脑血流量和动脉血压的影响采用激光多普勒血流仪测量大鼠软脑膜局部的血灌注量，其被视为局部的脑血流量。

RCP is regarded as an assistant method in deep hypothermic circulatory arrest in that it provides partial brain blood flow,maintains a low brain temperature,optimizes cerebral metabolic function during DHCA by supplying oxygen and some nutrient and removal of catabolic products;it also reduces the incidence of cerebral embolization by flushing out air and particulate microemboli from the arterial tree before the reinstitution of antegrade perfusion,thus prolonging the limitation of safety of DHCA.But brain edema probably happens to restrict the clinical use of this method.

上腔静脉逆行性灌注是深低温停循环脑保护的辅助手段，已证明在低温状态下，它为脑部提供低流量血流，维持脑部低温状态；提供部分氧和营养物质，运走代谢产物；减少气栓及栓塞的发生，从而延长了深低温停循环脑保护的安全时限，而脑水肿的危险性限制了该方法在临床的应用。

The results demonstrate that hypertensive cerebral arteriolosclerosis is the pathologic base which cerebral infarction is easy induced by hypotension, and thereis a close relationship between the occur of cerebral infarction and hypertensive degrees and duration in hypotension condition.

表明高血压性脑内小血管病变是低血压容易诱发脑梗塞的病理基础，说明低血压时，脑梗塞的产生与患者高血压程度的高低及持续时间的长短有密切的关系。

It has been found that ectogenic GDNF has nervous protective effec,but ectogenic GDNF cannot enter blood brain barrier.

传统观点认为，星形胶质细胞是神经系统的支持细胞，构成神经组织的网架而起支持作用，与神经元的解剖位置关系紧密，参与中枢神经系统的免疫反应，参与构成血脑屏障，目前研究发现，脑缺血时星形胶质细胞异常活跃，通过合成多种蛋白质、细胞因子和神经营养因子等对神经元起保护作用，对缺血性脑损伤的发展和转归具有重要意义。

The contrast of lesion-to-normal and lesion-to-CSF were higher in FLAIR, was particularly superior in demonstrating lesions in the hypophloeodal or paraventricular area.Small hemorrhagic foci in Sylvian cistern and the cerebral sulci can be easily shown in FLAIR and can?t be done in conventional T2WI.FLAIR was particular of superiority to demonstrate trauma and subarachnoid haemorrhage.

结果 FLAIR共检出病灶1889个，常规T2加权像检出1343个，FLAIR显示病灶的轮廓更为清晰，病灶与正常脑组织的对比度更高，在显示脑皮层下、脑室旁病灶方面更有优势，可显示常规MR T2加权像未能显示的侧裂池及脑表面脑沟的小出血病灶，在诊断颅脑外伤及蛛网膜下腔出血方面具有明显的优势。

The contrast of lesion-to-normal and lesion-to-CSF were higher in FLAIR,which was particularly superior in demonstrating lesions in the hypophloeodal or paraventricular area.Small hemorrhagic foci in Sylvian cistern and the cerebral sulci can be easily shown in FLAIR and can?t be done in conventional T2WI.FLAIR was particular of superiority to demonstrate trauma and subarachnoid haemorrhage.

结果 FLAIR共检出病灶1889个，常规T2加权像检出1343个，FLAIR显示病灶的轮廓更为清晰，病灶与正常脑组织的对比度更高，在显示脑皮层下、脑室旁病灶方面更有优势，可显示常规MR T2加权像未能显示的侧裂池及脑表面脑沟的小出血病灶，在诊断颅脑外伤及蛛网膜下腔出血方面具有明显的优势。

Results Serum EDLS increased within 24 h after brain trauma of different degree in all patients with brain injuries of various intensities.The group of slight injury was the most typal,EDLS demonstrated no change or decrease in a few patients with the injury intensity growing;In most cases EDLS decreased after brain operation or accompaning the wounded recovery; Contents of EDLS at 24 h among different pathological types of brain injuries showed no difference, but in the successful recovery patients with severe or ultra-severe brain injuries, EDLS contents at 72 h posttrauma were lower than in the patients of death or disability. The above change characteristics of EDLS related to injury locations and the degree of brain injury.

结果 不同程度脑外伤后24小时内血清EDLS均升高，以轻伤组最为典型；随着损伤程度的加剧，少数病例EDLS不变甚至下降；术后或随伤情的恢复，EDLS多同步下降；不同病理类型的脑损伤24小时内EDLS无明显差异，在成功康复的重型、特重型脑损伤病人，伤后72小时EDLS含量低于死亡或伤残组；EDLS 的上述变化特点与损伤部位和程度不同有关。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。