脑的

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中，脑缺血能够诱导P53蛋白表达，P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程，药物通过抑制细胞凋亡，从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高（p＜0.05）。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。

Using the middle cerebral artery occlusion modelin rats and cerebral ischemia-reperfusion models in gerbils and mice, we investigated the influence of pueraria compound on the brain water content and the infarct size, the cerebral apoplexy exponent, the contents of lactic acidand lipid peroxide, the activities of lactic dehydrogenase, glutathione peroxidaseand Na+-K+-ATPase.

采用大鼠大脑中动脉栓塞模型、沙土鼠全脑缺血再灌模型和小鼠脑缺血再灌3种动物模型，研究复方葛根素对脑缺血再灌注脑含水量和梗塞范围、脑卒中指数和脑组织中乳酸、乳酸脱氢酶、过氧化脂质、谷胱甘肽过氧化物酶、Na+-K+-ATP酶的影响。

Yangxue Qingnao granules have definite effect on chronic cerebral circulation insufficiency, and decrease and hemorrheology in patients with CCCI, relieve angiospasm and improve clinical symptoms.

养血清脑颗粒治疗慢性脑供血不足疗效确切，能降低CCCI患者的血液黏稠度及血液流变学指标，增加脑供血，缓解血管痉挛，改善临床症状，改善脑供血不足。

In clinical and experimental studies, it has been found that cerebral ischemia and cerebral infarction were easy induced by hypotension in elder, hypertensive cerebral arteriolosclerosis, and stenosis or occlusion in carotid artery or in major cerbral arteries, butnow the effect machanisms still remain unclear.

从临床研究和动物实验也发现，在年老、高血压脑动脉硬化和颈动脉或大的脑动脉狭窄或闭塞时，低血压易诱发脑缺血和脑梗塞，但是目前其作用机制还未完全阐明。

Of the follow?up studies on 30 mixed bleeding cases,17 was normal,2 was dead,and the others were cerebromalacia,cerebral atrophy,porencephaty and calcium.

在30例合并颅内出血中，随访复查发现17例恢复正常；其他13例中，2例死亡，11例均有脑软化、脑萎缩、脑穿通畸形、脑钙化灶等不可逆性后遗症的表现。

Of the followup studies on 30 mixed bleeding cases,17 was normal,2 was dead,and the others were cerebromalacia,cerebral atrophy,porencephaty and calcium.

在30例合并颅内出血中，随访复查发现17例恢复正常；其他13例中，2例死亡，11例均有脑软化、脑萎缩、脑穿通畸形、脑钙化灶等不可逆性后遗症的表现。

Results: The brain fabric was normal without edema, hemorrhage and necrosis in groupⅠ. The brain parenchyma were loose, perivascular canal became widen, some neurocytes bulk and degenerate in groupⅡ. In groupⅢ, the brain injury aggravateed accompanying the lapse of survival time with extreme cerebral edema at 24 h, and colliquative necrosis in the brain parenchyma aparted at 40 h.

结果：Ⅰ组脑组织结构清晰，无水肿、出血、坏死病灶；Ⅱ组脑实质疏松，小血管周隙增宽，部分神经细胞体积增大变性；Ⅲ组随溺水后存活时间的推移，脑损伤程度逐渐加重，其中24h脑水肿最重，40h个别脑实质小灶性液化性坏死。

Part 1: Permeability of Exogenetic BDNF through placental barrier of pregnant rats and blood-brain barrier of fetus with transient uteroplacental ischemiaObjective To investigate the potential of BDNF to penetrate the placental barrier and get into the fetus and pass through the blood-brain barrier of fetus to play neuroprotective function in the condition of transient ischemia.

第一部分：外源性脑源性神经营养因子在宫内缺氧环境中对胎盘及胚鼠血脑屏障通透性的实验研究目的：探讨外源性脑源性神经营养因子在宫内缺氧环境下能否通过胎盘屏障进入胎鼠体内，再通过胎鼠血脑屏障进入胎鼠脑组织而发挥其生物功能。

Fant by use of serial CK-BB isoenzyme measurement.

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The risk factors for hematencephalon were analysed inchided CpIgG, aging, male, smoking, diabetes, hypertension, hyperlipidemia, mental labor and a high body mass index.

所有个体分为有、无脑出血，有、无脑出血危险因素（CpIgG抗体、年老、男性、吸烟、糖尿病、血脂紊乱、高血压、较少体力劳动和体重指数），比较有、无脑出血患者各种危险因素差别；并应用logistic回归分析，得出调整比值比值，用于评估Cp对脑出血的危险性。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。